224 Ivabradine, a Safe and Effective Monotherapy in the Treatment of Congenital Junctional Tachycardia in Infancy

1857 Is anticoagulation required in patients with unexplained cerebrovascular accident and paroxysmal junctional tachycardia?

European Heart Journal ◽

10.1016/s0195-668x(03)94966-9 ◽

2003 ◽

Vol 24 (5) ◽

pp. 355 ◽

Cited By ~ 1

Author(s):

B BREMBILLAPERROT

Keyword(s):

Cerebrovascular Accident ◽

Junctional Tachycardia

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P3173 CRAVT. A prospective, randomised study comparing transvenous cryothermy and radiofrequency ablation in atrioventricular junctional tachycardia. First results

European Heart Journal ◽

10.1016/s0195-668x(03)95803-9 ◽

2003 ◽

Vol 24 (5) ◽

pp. 592

Author(s):

G KIMMAN

Keyword(s):

Radiofrequency Ablation ◽

Randomised Study ◽

Junctional Tachycardia ◽

First Results ◽

Prospective Randomised Study

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Junctional tachycardia in infants and children after open heart surgery for congenital heart disease

The American Journal of Cardiology ◽

10.1016/0002-9149(87)90887-3 ◽

1987 ◽

Vol 59 (12) ◽

pp. 1216-1218 ◽

Cited By ~ 29

Author(s):

James W. Grant ◽

Gerald A. Serwer ◽

Brenda E. Armstrong ◽

H.Newland Oldham ◽

Page A.W. Anderson

Keyword(s):

Congenital Heart Disease ◽

Heart Disease ◽

Congenital Heart ◽

Heart Surgery ◽

Open Heart Surgery ◽

Infants And Children ◽

Open Heart ◽

Junctional Tachycardia ◽

In Infants And Children

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The importance of sustained junctional tachycardia following cessation of radiofrequency current delivery in slow pathway ablation

Pacing and Clinical Electrophysiology ◽

10.1111/pace.14376 ◽

2021 ◽

Author(s):

Alireza Malekrah ◽

Nader Asgary ◽

Alireza Fattahian ◽

Bahamin Amirabadi

Keyword(s):

Slow Pathway ◽

Junctional Tachycardia ◽

Slow Pathway Ablation ◽

Radiofrequency Current

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Catheter Ablative Techniques for Treatment of Patients with Automatic Ectopic Atrial or Junctional Tachycardia

Catheter Ablation of Cardiac Arrhythmias - Developments in Cardiovascular Medicine ◽

10.1007/978-1-4613-1765-4_10 ◽

1988 ◽

pp. 175-181

Author(s):

Paul C. Gillette ◽

Bertrand Ross ◽

Vickie Zeigler

Keyword(s):

Junctional Tachycardia ◽

Ablative Techniques

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Adrenergic potentiation on spontaneous activity of canine paranodal fibers

AJP Heart and Circulatory Physiology ◽

10.1152/ajpheart.1984.247.3.h415 ◽

1984 ◽

Vol 247 (3) ◽

pp. H415-H421

Author(s):

W. W. Tse

Keyword(s):

Spontaneous Activity ◽

Tricuspid Valve ◽

Action Potentials ◽

Smooth Transition ◽

Convex Side ◽

Av Node ◽

Junctional Tachycardia ◽

His Bundle ◽

Spontaneous Rate

The present study, using in vitro preparations, was designed to determine the anatomic, histological, and automatic properties of canine paranodal fibers. This tissue, together with the atrioventricular (AV) node and His bundle, constituted the three major tissues in the AV junction. The fascicles of the paranodal fibers ran parallel and adjacent to the base of the septal cusp of the tricuspid valve. The distal end of the paranodal fibers joined the lower half of the compact AV node on its convex side. Paranodal fibers when isolated were able to initiate spontaneous activity. Action potentials of many of these fibers showed primary pacemaker characteristics, i.e., a prominent phase 4 depolarization and smooth transition from phases 4 to 0. In 14 preparations, epinephrine (2.0 micrograms injected into the tissue bath) potentiated spontaneous rates to 144 +/- 6.0 beats/min from 61 +/- 5.0, an increase of 136%. Also, under the influence of epinephrine, paranodal fibers consistently generated a spontaneous rate higher than that of the AV node or His bundle, whether they were functionally connected or separated. These findings provide a basis for explaining the junctional tachycardia that occurs under adrenergic influence and demonstrate the presence of three major automatic tissues: the paranodal fibers, AV node, and His bundle in the canine AV junction.

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