Distribution of Pseudorabies Virus Antigen in Hunting Dogs with Concurrent Paragonimus westermani Infection

2021 ◽  
Vol 188 ◽  
pp. 44-51
Author(s):  
Uda Z. Izzati ◽  
Yasuyuki Kaneko ◽  
Chiho Kaneko ◽  
Ayako Yoshida ◽  
Mathurot Suwanruengsri ◽  
...  
2012 ◽  
Vol 24 (1) ◽  
pp. 246-246

JVDI 23:915–923 (September 2011) In the article entitled “Pseudorabies virus infection in Oklahoma hunting dogs” by Sarah D. Cramer et al., on page 918 in the legend to Figure 2, the legend should refer to an “intranuclear” inclusion body instead of an “intracytoplasmic” inclusion body.


2011 ◽  
Vol 23 (5) ◽  
pp. 915-923 ◽  
Author(s):  
Sarah D. Cramer ◽  
Gregory A. Campbell ◽  
Bradley L. Njaa ◽  
Sandra E. Morgan ◽  
Stephen K. Smith ◽  
...  

2008 ◽  
Vol 158 (4) ◽  
pp. 376-379 ◽  
Author(s):  
Yumi Kirino ◽  
Natsuko Nakano ◽  
Mitsuyoshi Hagio ◽  
Yuichi Hidaka ◽  
Fukumi Nakamura-Uchiyama ◽  
...  

2010 ◽  
Vol 138 (11) ◽  
pp. 1590-1600 ◽  
Author(s):  
T. MÜLLER ◽  
B. G. KLUPP ◽  
C. FREULING ◽  
B. HOFFMANN ◽  
M. MOJCICZ ◽  
...  

SUMMARYPseudorabies virus (PrV) infections appear to be more widely distributed in the European wild boar (Sus scrofa) population than assumed. In Europe, attempts to isolate and characterize the causative agents have been limited so far. We therefore collected and examined a total of 35 PrV isolates obtained from wild boar or hunting dogs in Germany, France, Spain, Italy, Slovakia and Hungary between 1993 and 2008. Restriction enzyme analysis of genomic DNA using BamHI showed that all isolates, except one, belonged to genogroup I but different subtypes were evident. For further investigations of the phylogenetic relationships, a 732-bp fragment of the glycoprotein C (gC) gene was amplified by PCR. Sequence analysis revealed about 40 variant positions within this fragment. Comparison of the nucleotide sequences supported the separation into a clade containing isolates from North-Rhine Westphalia, Rhineland-Palatinate (Germany), France and Spain (clade B) and an apparently more variable clade comprising isolates from Brandenburg, Baden-Wurttemberg, Saxony, Saxony-Anhalt (Germany), Slovakia, Hungary, Italy and France (clade A).


2017 ◽  
Vol 79 (8) ◽  
pp. 1419-1425 ◽  
Author(s):  
Takao IRIE ◽  
Yohei YAMAGUCHI ◽  
Pham Ngoc DOANH ◽  
Zhi Hong GUO ◽  
Shigehisa HABE ◽  
...  

1998 ◽  
Vol 10 (1) ◽  
pp. 36-42 ◽  
Author(s):  
M. Narita ◽  
Y. M. Zhao ◽  
K. Kawashima ◽  
S. Arai ◽  
H. Hirose ◽  
...  

Three different strains of pseudorabies (Aujeszky's disease) virus were inoculated into ligated closed loops in the jejunum and ileum of five specific-pathogen-free pigs. Infected areas were compared with respect to distribution of histologic lesions and pseudorabies virus antigen. Two wild-type strains of pseudorabies virus produced enteric lesions consisting of necrosis of the subepithelial macrophages in the basilar crypt epithelium, necrosis of the lymphoid follicles in the Peyer's patches, degeneration of the epithelial cells in the crypt and villi, degeneration of the neuronal cells in the myenteric plexuses, and formation of intranuclear inclusion bodies on postinoculation days 2–4. Pseudorabies virus antigen was initially detected in subepithelial macrophages of the dome of Peyer's patches on postinoculation day 2 and subsequently extended to superficial epithelium and deeper into the lymphoid follicles and myenteric plexuses on postinoculation days 3–4. Many pseudorabies virus particles were also detected in the center of their necrotic foci. However, 1 mutant strain (ara-T-resistant) of pseudorabies virus did not produce enteric lesions. The results suggest that the primary target of infection by wild-type strains of pseudorabies virus might be the macrophages distributed in the subepithelial area of the dome Peyer's patches.


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