Endovascular Treatment of Traumatic Transection of the Vertebral Artery Caused by Penetrating Injury to the Oropharynx in a Pediatric Patient

2020 ◽  
Vol 137 ◽  
pp. 389-392
Author(s):  
Nelson Sofoluke ◽  
Nir Shimony ◽  
Oded Goren
Neurosurgery ◽  
2002 ◽  
Vol 50 (1) ◽  
pp. 204-208 ◽  
Author(s):  
Salvatore Grosso ◽  
Rosa Mostardini ◽  
Carlo Venturi ◽  
Sandra Bracco ◽  
Alfredo Casasco ◽  
...  

2017 ◽  
Vol 45 ◽  
pp. 267.e1-267.e5 ◽  
Author(s):  
Dafne Braga Diamante Leiderman ◽  
Antonio Eduardo Zerati ◽  
Nelson Wolosker ◽  
Henry Augusto Hoffmann Melo ◽  
Erasmo Simão da Silva ◽  
...  

2020 ◽  
Vol 26 (6) ◽  
pp. 805-813
Author(s):  
Jun-Kyeung Ko ◽  
Chang-Hwa Choi ◽  
Lee Hwangbo ◽  
Hie-Bum Suh ◽  
Tae-Hong Lee ◽  
...  

Background Endovascular treatment has been considered a good alternative to surgery for symptomatic vertebral artery origin stenosis (VAOS) due to the high risk of morbidity associated with surgery. The purpose of this study was to evaluate the feasibility and efficacy of insertion of the closed-cell, self-expandable Carotid Wallstent for the treatment of VAOS. Methods The records of 72 patients with VAOS refractory to adequate medication who were treated by endovascular treatment with the Carotid Wallstent from December 2006 to November 2018 were retrospectively evaluated. Results Of the 72 patients, 43 presented with transient ischemic attacks. Forty-seven patients (65.3%) manifested other brachiocephalic stenoses; of these, 40 patients had occlusion, hypoplasia, or stenosis of the contralateral vertebral artery. Overall technical success (defined as 20% or less residual stenosis) was 100%. Procedure-related complications ( n = 8, 11.1%) included sudden asystole ( n = 1), acute in-stent thrombosis ( n = 3), minor stroke ( n = 3), and stent shortening ( n = 1). All complications were resolved without permanent neurological deficit. Angiographic follow-up (mean, 13.0 months) was achieved in 49 patients and revealed in-stent restenosis in 1 patient (2.0%) and stent malposition by shortening in 2 patients (4.1%). Follow-up records were available in 57 patients (mean 15.6 months). Three of the 57 patients ( n = 3, 5.3%) had recurrent symptoms of vertebrobasilar ischemia and none was retreated. Conclusions Endovascular treatment of symptomatic VAOS using the closed-cell, self-expandable Carotid Wallstent is technically feasible and effective in alleviating patient symptoms and for improving vertebrobasilar blood flow.


2015 ◽  
Vol 29 (5) ◽  
pp. 1018.e5-1018.e8 ◽  
Author(s):  
Gabriel Cristian Inaraja Pérez ◽  
Alejandro Rodríguez Morata ◽  
Juan Pedro Reyes Ortega ◽  
Rafael Gómez Medialdea ◽  
Pablo Cabezudo García

2016 ◽  
Vol 18 (3) ◽  
pp. 201 ◽  
Author(s):  
Jinsol Han ◽  
Dong-Jun Lim ◽  
Sung-Kon Ha ◽  
Jong-Il Choi ◽  
Sung-Won Jin ◽  
...  

2011 ◽  
Vol 70 (suppl_1) ◽  
pp. ons75-ons81 ◽  
Author(s):  
Yong Sam Shin ◽  
Byung Moon Kim ◽  
Se-Hyuk Kim ◽  
Sang Hyun Suh ◽  
Chang Woo Ryu ◽  
...  

Abstract BACKGROUND: Optimal management of bilateral vertebral artery dissecting aneurysms (bi-VDAs) causing subarachnoid hemorrhage (SAH) remains unclear. OBJECTIVE: To investigate the treatment methods and outcomes of bi-VDA causing SAH. METHODS: Seven patients were treated endovascularly for bi-VDA causing SAH. Treatment methods and outcomes were evaluated retrospectively. RESULTS: Two patients were treated with 2 overlapping stents for both ruptured and unruptured VDAs, 2 with 2 overlapping stents and coiling for ruptured VDA and with conservative treatment for unruptured VDA, 1 with internal trapping (IT) for ruptured VDA and stent-assisted coiling for unruptured VDA, 1 with IT for ruptured VDA and 2 overlapping stents for unruptured VDA, and 1 with IT for ruptured VDA and a single stent for unruptured VDA. None had rebleeding during follow-up (range, 15-48 months). All patients had favorable outcomes (modified Rankin Scale score, 0-2). On follow-up angiography at 6 to 36 months, 9 treated and 2 untreated VDAs revealed stable or improved state, whereas 3 VDAs in 2 patients showed regrowth. Of the 3 recurring VDAs, 1 was initially treated with IT but recurred owing to retrograde flow to the ipsilateral posterior inferior cerebellar artery (PICA), the second was treated with single stent but enlarged, and the last was treated with 2 overlapping stents and coiling but recurred from the remnant sac harboring the PICA origin. All 3 recurred VDAs were retreated with coiling with or without stent insertion. CONCLUSION: Bilateral VDAs presenting with SAH were safely treated with endovascular methods. However, endovascular treatment may be limited for VDAs with PICA origin involvement.


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