scholarly journals THERAPEUTIC DILEMMAS IN THE MANAGEMENT OF PATENT FORAMEN OVALE (PFO) IN THE SETTING OF PACER LEADS AND TRICUSPID REGURGITATION

2021 ◽  
Vol 77 (18) ◽  
pp. 2446
Author(s):  
Van La ◽  
Ashis Mukherjee ◽  
Padmini Varadarajan ◽  
Ramdas G. Pai
2016 ◽  
Vol 28 (1) ◽  
pp. 1-5
Author(s):  
Hasan KADI ◽  
Birol ÖZKAN ◽  
Eyüp AVCI ◽  
Taha GÜRBÜZER ◽  
Tarık YILDIRIM

Heart Asia ◽  
2012 ◽  
Vol 4 (1) ◽  
pp. 67-68
Author(s):  
Behdad Bahadorian ◽  
Majid Maleki ◽  
Anoushiravan Vakili-Zarch ◽  
Zahra Alizadeh-Sani

2016 ◽  
Vol 43 (2) ◽  
pp. 171-174
Author(s):  
Evan P. Kransdorf ◽  
Lisa N. Kransdorf ◽  
F. David Fortuin ◽  
John P. Sweeney ◽  
Susan Wilansky

Patent foramen ovale is a common clinical finding that generally becomes a concern in the presence of transient ischemic attack or stroke. Rarely, patent foramen ovale is associated with hypoxemia in the presence of substantial right-to-left atrial shunting. We present the case of an 86-year-old woman with a pacemaker, who was initially asymptomatic notwithstanding a patent foramen ovale. Over 1.5 years, her symptoms progressed in a stepwise fashion, in the setting of progressive pacemaker-associated tricuspid regurgitation. Ultimately, the patient's symptoms and her hypoxemia resolved after percutaneous closure of her patent foramen ovale with use of a 25-mm “Cribriform” occluder device. This case highlights the fact that clinically significant right-to-left shunting requires an anatomic lesion, such as patent foramen ovale, together with elevated right atrial pressure, which in this case was contributed by severe tricuspid regurgitation.


2015 ◽  
Vol 2015 ◽  
pp. 1-3 ◽  
Author(s):  
S. A. Zuberi ◽  
S. Liu ◽  
J. W. Tam ◽  
F. Hussain ◽  
D. Maguire ◽  
...  

Ebstein anomaly is characterized by deformities of the anterior leaflet of the tricuspid valve and atrialization of the right ventricle. Patients with severe tricuspid regurgitation are recommended to have tricuspid valve surgery with concomitant atrial septal defect closure. A 73-year-old female with Ebstein anomaly presented with severe hypoxemia. Transthoracic echocardiography revealed severe tricuspid regurgitation and a patent foramen ovale with right-to-left shunting. Complete percutaneous patent foramen ovale closure led to acute decompensation; however, partial closure led to hemodynamic stability and improved oxygenation. In conclusion, similar patients with “patent foramen ovale dependency” from longstanding shunts may benefit from partial patent foramen ovale closure.


2021 ◽  
Vol 5 (7) ◽  
Author(s):  
Laura Fuertes-Kenneally ◽  
Juan Quiles-Granado ◽  
Jessica Sánchez-Quiñones ◽  
Juan Gabriel Martínez-Martínez

Abstract Background Platypnoea–orthodeoxia syndrome (POS) is a rare condition characterized by hypoxaemia and dyspnoea when changing from a recumbent to an upright position. Diagnosis requires a high clinical suspicion and is often underdiagnosed. Case summary We report a case of POS in a 50-year-old woman with dyspnoea and new-onset atrial fibrillation. Oxygen saturation and dyspnoea worsened as she changed from a supine to a sitting position (96 vs. 86%, respectively). Transoesophageal echocardiography demonstrated enlargement of both atria and right ventricle with reduced systolic function and a large Chiari network (CN). Colour Doppler discovered severe tricuspid regurgitation with tenting and tethering of the valve leaflets. Finally, a bubble test revealed the cause of POS to be a patent foramen ovale along with the severe tricuspid regurgitant jet moving into the left atrium and favoured by the CN. Surgical closure of the foramen ovale resulted in the resolution of symptoms. Discussion Platypnoea–orthodeoxia syndrome is most commonly caused by a right-to-left shunt through an anatomical defect of the interatrial septum, typically a patent foramen ovale, combined with elevated right atrium pressure. This case illustrates an uncommon cause of POS in the absence of elevated atrium pressure due to the interplay of three key elements: a patent foramen ovale, tricuspid regurgitation, and the CN. Our aim is to alert physicians to the possibility of an intracardiac shunt as the cause of unexplained and/or refractory hypoxaemia related to position changes. Early recognition of this syndrome promotes timely treatment, greatly improving patient outcomes.


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