scholarly journals Atherosclerotic renal vascular disease (ASRVD) effect of primary stent placement on blood pressure (BP) and renal function

2002 ◽  
Vol 15 (4) ◽  
pp. A177
Author(s):  
R TAGLE
Keyword(s):  
Blood Pressure ◽  
Renal Function ◽  
Vascular Disease ◽  
Stent Placement ◽  
Renal Vascular Disease ◽  
Renal Vascular

scholarly journals ARTERIAL HYPERTENSION IN RATS

1942 ◽  
Vol 75 (5) ◽  
pp. 527-538 ◽  
Author(s):  
Henry A. Schroeder ◽  
Charles Neumann
Keyword(s):  
Blood Pressure ◽  
Cardiac Hypertrophy ◽  
Arterial Hypertension ◽  
Vascular Disease ◽  
Vascular Lesions ◽  
Elevated Blood ◽  
Renal Lesions ◽  
Renal Vascular Disease ◽  
The One ◽  
Renal Vascular

1. When rats developed cardiac hypertrophy or elevation of blood pressure as a result of one of several methods designed to bring about arterial hypertension, renal vascular disease occurred frequently. 2. When injury to one kidney was followed by cardiac hypertrophy or elevation of blood pressure, vascular lesions were found with considerable regularity in the opposite one, as well as in the one injured. 3. Renal lesions rarely occurred in the absence of cardiac hypertrophy or elevated blood pressure. 4. Renal vascular lesions in rats are occasioned, therefore, by injury to one kidney and are usually associated with, and dependent on, the presence of arterial hypertension.

scholarly journals STUDIES ON EXPERIMENTAL HYPERTENSION

1934 ◽  
Vol 59 (3) ◽  
pp. 347-379 ◽  
Author(s):  
Harry Goldblatt ◽  
James Lynch ◽  
Ramon F. Hanzal ◽  
Ward W. Summerville
Keyword(s):  
Blood Pressure ◽  
Renal Function ◽  
Systolic Blood Pressure ◽  
Renal Arteries ◽  
Experimental Hypertension ◽  
Subsequent Increase ◽  
Elevated Systolic Blood Pressure ◽  
Renal Vascular Disease ◽  
In The Beginning ◽  
Renal Vascular

These experiments indicate that, in dogs at least, ischemia localized to the kidneys is a sufficient condition for the production of persistently elevated systolic blood pressure. When the constriction of both main renal arteries is made only moderately severe in the beginning, the elevation of systolic blood pressure is unaccompanied by signs of materially decreased renal function. In this respect the hypertension in these animals resembles the hypertension which is associated with so called benign nephrosclerosis in man. Subsequent increase of the constriction of the main renal arteries does not materially damage renal function, probably because of adequate development of accessory circulation. More delicate methods for detecting a change may yet prove that some damage does occur. Almost complete constriction of both main renal arteries, from the beginning, results in great elevation of systolic blood pressure which is accompanied by severe disturbance of renal function and uremia. This resembles the type of hypertension which is associated with so called malignant nephrosclerosis, in the sense of Fahr (17). In several of the animals with persistent elevation of systolic blood pressure, anatomical changes were observed in the glomeruli, vessels and parenchyma of the kidneys which are most probably directly referable to the ischemia. It is hoped that these investigations will afford a means of studying the pathogenesis of hypertension that is associated with renal vascular disease.

BLOOD PRESSURE IN CHILDREN WITH DIABETES MELLITUS

PEDIATRICS ◽  
1962 ◽  
Vol 30 (6) ◽  
pp. 932-936
Author(s):  
Arthur J. Moss
Keyword(s):  
Diabetes Mellitus ◽  
Blood Pressure ◽  
Statistical Analysis ◽  
Arterial Pressure ◽  
Vascular Disease ◽  
Systolic Arterial Pressure ◽  
Normal Children ◽  
Diabetic Children ◽  
Renal Vascular Disease ◽  
Renal Vascular

Systolic arterial pressure was measured in 123 children with diabetes mellitus and in 889 normal children. Statistical analysis indicates that, when compared to normals, the pressure tends to increase significantly in diabetic children at about 13 years of age. It is postulated that this increase in pressure represents a prehypertensive state due to subclinical renal vascular disease.

Hypertensive renal vascular disease and cardiovascular endpoints

2006 ◽  
Vol 21 (4) ◽  
pp. 305-309 ◽  
Author(s):  
Luis C Matavelli ◽  
Xiaoyan Zhou ◽  
Edward D Frohlich
Keyword(s):  
Vascular Disease ◽  
Renal Vascular Disease ◽  
Renal Vascular

Surgical treatment of unilateral renal vascular disease

1972 ◽  
Vol 30 (8) ◽  
pp. 827-831 ◽  
Author(s):  
Donald G. Vidt ◽  
Fredrick M. Yutani ◽  
Lawrence J. McCormack ◽  
Ray W. Gifford ◽  
Bruce H. Stewart ◽  
...  
Keyword(s):  
Surgical Treatment ◽  
Vascular Disease ◽  
Renal Vascular Disease ◽  
Renal Vascular

Renal Vascular Disease in the Newborn

2012 ◽  
pp. 1235-1244
Author(s):  
Halima Saadia Janjua ◽  
Donald L. Batisky
Keyword(s):  
Vascular Disease ◽  
Renal Vascular Disease ◽  
Renal Vascular

High-potassium diet attenuates salt-induced acceleration of hypertension in SHR

1991 ◽  
Vol 260 (1) ◽  
pp. R21-R26 ◽  
Author(s):  
Y. Sato ◽  
K. Ando ◽  
E. Ogata ◽  
T. Fujita
Keyword(s):  
Blood Pressure ◽  
Renal Function ◽  
Spontaneously Hypertensive Rats ◽  
Hypertensive Rats ◽  
High Potassium ◽  
Spontaneously Hypertensive ◽  
Antihypertensive Action ◽  
High K ◽  
Wistar Kyoto ◽  
Renal Vascular

We studied the effects of K supplementation (8% KCl) for 4 wk on blood pressure (BP), Na space, and renal hemodynamics in 5-wk-old, spontaneously hypertensive rats (SHR) or age-matched Wistar-Kyoto rats (WKY) eating normal-NaCl (0.66%) or high-NaCl (8%) diet. In WKY, high-Na and/or high-K diets had no effects on BP. In SHR, Na load accelerated the development of hypertension, whereas K supplementation did not affect BP of normal-Na SHR but attenuated the increase in BP with Na load. Correspondingly, Na load in SHR significantly increased renal vascular resistance (RVR), and K supplementation attenuated the increased RVR of Na-loaded SHR. Moreover, Na space of SHR was increased compared with that of WKY, and although Na load did not affect Na space, K supplementation tended to decrease Na space in SHR. These results indicate that 9-wk-old SHR is relatively volume-expanded compared with age-matched WKY, and K supplementation could improve the lowered slope of the pressure-Na excretion relationship in SHR, resulting in maintenance of Na balance. Thus the data suggest that changes in RVR, which might be intimately related to renal function for Na excretion, contribute to both salt sensitivity of SHR and antihypertensive action of K supplementation in Na-loaded SHR.

Sign in / Sign up

Export Citation Format

Share Document