Animal Behavior Processes: Editor.

2003 ◽  
Vol 29 (2) ◽  
pp. C2-C2
Author(s):  
No authorship indicated
Keyword(s):  
Animal Behavior

Facet Theory

1998 ◽  
Vol 3 (1) ◽  
pp. 13-36 ◽  
Author(s):  
Ruth Guttman ◽  
Charles W. Greenbaum
Keyword(s):  
Data Analysis ◽  
Animal Behavior ◽  
Infant Development ◽  
Facet Theory ◽  
Structural Relationships ◽  
Future Challenges ◽  
Spatial Configurations ◽  
Multidimensional Methods ◽  
Definition Of ◽  
Scalogram Analysis

This article gives an overview of Facet Theory, a systematic approach to facilitating theory construction, research design, and data analysis for complex studies, that is particularly appropriate to the behavioral and social sciences. Facet Theory is based on (1) a definitional framework for a universe of observations in the area of study; (2) empirical structures of observations within this framework; (3) a search for correspondence between the definitional system and aspects of the empirical structure for the observations. The development of Facet Theory and Facet Design is reviewed from early scale analysis and the Guttman Scale, leading to the concepts of “mapping sentence,” “universe of content,” “common range,” “content facets,” and nonmetric multidimensional methods of data analysis. In Facet Theory, the definition of the behavioral domain provides a rationale for hypothesizing structural relationships among variables employed in a study. Examples are presented from various areas of research (intelligence, infant development, animal behavior, etc.) to illustrate the methods and results of structural analysis with Smallest Space Analysis (SSA), Multidimensional Scalogram Analysis (MSA), and Partial Order Scalogram Analysis (POSA). The “radex” and “cylindrex” of intelligence tests are shown to be outstanding examples of predicted spatial configurations that have demonstrated the ubiquitous emergence of the same empirical structures in different studies. Further examples are given from studies of spatial abilities, infant development, animal behavior, and others. The use of Facet Theory, with careful construction of theory and design, is shown to provide new insights into existing data; it allows for the diagnosis and discrimination of behavioral traits and makes the generalizability and replication of findings possible, which in turn makes possible the discovery of lawfulness. Achievements, issues, and future challenges of Facet Theory are discussed.

Review of Unravelling animal behavior.

1996 ◽  
Vol 41 (2) ◽  
pp. 187-187
Author(s):  
Donald A. Dewsbury
Keyword(s):  
Animal Behavior

Review of Animal behavior in laboratory and field, 2nd Ed.

1975 ◽  
Vol 20 (11) ◽  
pp. 911-911
Author(s):  
RICHARD F. THOMPSON
Keyword(s):  
Animal Behavior

A Well-Behaved Animal Behavior Text

1983 ◽  
Vol 28 (6) ◽  
pp. 427-428
Author(s):  
Charles T. Snowdon
Keyword(s):  
Animal Behavior

Understanding the Students of Animal Behavior

1987 ◽  
Vol 32 (1) ◽  
pp. 49-50
Author(s):  
Erich Klinghammer
Keyword(s):  
Animal Behavior

Review of Comparative Animal Behavior.

1971 ◽  
Vol 12 (2) ◽  
pp. 320-321
Author(s):  
G. Rowland
Keyword(s):  
Animal Behavior

Bone Manifestation of Faulty Perinatal Hormonal Imprinting: A Review

2019 ◽  
Vol 15 (1) ◽  
pp. 4-9
Author(s):  
G. Csaba
Keyword(s):  
Animal Behavior ◽  
Binding Capacity ◽  
Animal Experiments ◽  
Osteoporotic Fractures ◽  
Postnatal Period ◽  
Hormonal Imprinting ◽  
Adult Age ◽  
Pathological Development ◽  
Synthetic Hormones ◽  
Critical Developmental Period

Hormonal imprinting takes place at the first encounter between the developing receptor and its target hormone and the encounter determines the receptor's binding capacity for life. In the critical period of development, when the window for imprinting is open, the receptor can be misdirected by related hormones, synthetic hormones, and industrial or communal endocrine disruptors which cause faulty hormonal imprinting with life-long consequences. Considering these facts, the hormonal imprinting is a functional teratogen provoking alterations in the perinatal (early postnatal) period. One single encounter with a low dose of the imprinter in the critical developmental period is enough for the formation of faulty imprinting, which is manifested later, in adult age. This has been justified in the immune system, in sexuality, in animal behavior and brain neurotransmitters etc. by animal experiments and human observations. This review points to the faulty hormonal imprinting in the case of bones (skeleton), by single or repeated treatments. The imprinting is an epigenetic alteration which is inherited to the progeny generations. From clinical aspect, the faulty imprinting can have a role in the pathological development of the bones as well, as in the risk of osteoporotic fractures, etc.

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