Surgically Repaired Delayed Mitral Regurgitation After Radiofrequency Catheter Ablation for Wolff-Parkinson-White Syndrome

2002 ◽  
Vol 25 (7) ◽  
pp. 1142-1143 ◽  
Author(s):  
TOSHIYA OHTSUKA ◽  
YUJI MURAKAWA ◽  
NORIYUKI HAYAMI ◽  
YUTAKA KOTSUKA ◽  
SHINICHI TAKAMOTO
2005 ◽  
Vol 15 (3) ◽  
pp. 315-318 ◽  
Author(s):  
Radu Vatasescu ◽  
Laszlo Kornyei ◽  
Tamas Szili-Torok

Radiofrequency lesions can, theoretically, be the substrate for new persistent arrhythmias. As far as we know, this has never previously been encountered after transcatheter ablation of accessory pathways. A child with Wolff–Parkinson–White syndrome was referred for radiofrequency catheter ablation of a left-sided accessory pathway. After successful ablation of the accessory pathway using a retrograde transaortic approach, the child developed an incessant wide QRS complex tachycardia at slow rate that was resistant to pharmacologic interventions. The focus of the tachycardia was identical to the ventricular site of insertion of the eliminated accessory pathway.


2017 ◽  
Vol 28 (1) ◽  
pp. 168-170
Author(s):  
Seigo Okada ◽  
Jun Muneuchi ◽  
Hideki Origuchi

AbstractA 21-year-old man with Wolff–Parkinson–White syndrome and aneurysmal septal dyskinesis underwent radiofrequency catheter ablation of the accessory pathways. Before radiofrequency catheter ablation, the activation wavefront arose from the aneurysmal septum, whereas the propagation of the left ventricle was normalised after radiofrequency catheter ablation. These findings demonstrate the importance of the electro-mechanical interaction in patients with Wolff–Parkinson–White syndrome and ventricular dysfunction.


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