Testing a diathesis-stress model during the transition to university: Associations between self-criticism, stress, and internalizing problems

Author(s):  
Christina L. Robillard ◽  
Brianna J. Turner ◽  
Carolyn E. Helps
1997 ◽  
Vol 106 (1) ◽  
pp. 145-153 ◽  
Author(s):  
Thomas E. Joiner ◽  
Todd F. Heatherton ◽  
M. David Rudd ◽  
Norman B. Schmidt

1997 ◽  
Vol 104 (4) ◽  
pp. 667-685 ◽  
Author(s):  
Elaine F. Walker ◽  
Donald Diforio

2021 ◽  
pp. 103985622110373
Author(s):  
Gordon Parker ◽  
Gabriela Tavella

Objective: As burnout has been neglected in medical and psychiatric education, we seek to provide a summary overview. Methods: We extract salient findings from the published literature and offer some challenges. Results: We critique the current principal model of burnout, argue for broadening the symptom constructs and for a diathesis-stress model where a perfectionistic personality style is a key predisposing factor, and observe that burnout is not limited by those in formal work. We argue that burnout is not synonymous with depression, overview biological underpinnings, and summarise a three-fold management model. Conclusion: As many burnout patients are referred to psychiatrists, awareness of its symptom pattern and management nuances is of key importance.


Author(s):  
Kristen Salomon ◽  
Alvin Jin

2003 ◽  
Vol 7 (3) ◽  
pp. 237-250 ◽  
Author(s):  
Victoria M. McKeever ◽  
Maureen E. Huff

The symptoms captured within the contemporary diagnostic definition of posttraumatic stress disorder (PTSD) have been studied for more than 100 years. Yet, even with increasingly advanced discoveries regarding the etiology of PTSD, a comprehensive and up-to-date etiological model that incorporates both medical and psychological research has not been described and systematically studied. The diathesis-stress model proposed here consolidates existing medical and psychological research data on etiological factors associated with PTSD into 3 causal pathways: residual stress, ecological, and biological. In combination, these pathways illuminate how PTSD might develop and who might be at higher risk for developing the disorder. Research and treatment implications related to the diathesis-stress model are discussed.


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