A Method for Predicting Malignant Tumor Destruction by Laser Radiation

1965 ◽  
Vol 34 (2) ◽  
pp. 291-296 ◽  
Author(s):  
John Peter Minton ◽  
Marvin Zelen
Author(s):  
А.В. Макрушин

В статье подвергается сомнению существующее представление о природе рака. Предлагается другое объяснение природы этой болезни. Механизм рака атавистический. Он возник у докембрийских Metazoa, которые были сидячими и колониальными. У них он обеспечивал приспособление к сезонному ухудшению среды. Готовящиеся к диапаузе растущие почки этих животных стали эволюционными предшественниками злокачественной опухоли. Разрушения в организме, происходящие у этих Metazoa при подготовке к диапаузе, стали эволюционными предшественниками разрушений в организме, происходящих при раке. Рассасывания готовящихся к диапаузе почек у них стало эволюционным предшественником регрессии опухоли. Регрессия опухоли - явление редкое и для изучения поэтому трудное. Исследовать его следует не у высоко организованных животных и не у человека, а у колониальных асцидий. Поняв, как работает у них механизм рассасывания готовящихся к диапаузе почек, легче будет понять механизм регрессии опухоли. У растений готовящиеся к покою почки тоже иногда разрушаются. Исследование этого процесса у растений тоже может помочь пониманию механизма регрессии опухоли. The current understanding of the nature of cancer is being questioned, and another explanation of the nature of this disease has been proposed. The mechanism of cancer is atavistic. It emerged in the Precambrian Metazoa, which were sedentary and colonized. This mechanism provided them with a means to adapt to seasonal changes in the environment. The developing kidneys of these animals preparing for diapause were the evolutionary precursors of a malignant tumor. The destruction that occurred in their kidneys when preparing for diapause was the evolutionary predecessor of the tumor destruction in the human body that occurs during cancer regression. Resorption of diapause-preparing kidneys was an evolutionary precursor to tumor regression. Tumor regression is rare and therefore difficult to study. Thus, this phenomenon should not be investigated in highly organized animals or in humans, but in colonized ascidians. Having understood how the mechanism of resorption of their kidneys functions when preparing for diapause, it will be easier to understand the mechanism of tumor regression. In plants, buds preparing for dormancy are sometimes also destroyed. Studying this process can similarly help decipher the mechanism of tumor regression.


Author(s):  
H. J. Finol ◽  
M. E. Correa ◽  
L.A. Sosa ◽  
A. Márquez ◽  
N.L. Díaz

In classical oncological literature two mechanisms for tissue aggression in patients with cancer have been described. The first is the progressive invasion, infiltration and destruction of tissues surrounding primary malignant tumor or their metastases; the other includes alterations produced in remote sites that are not directly affected by any focus of disease, the so called paraneoplastic phenomenon. The non-invaded tissue which surrounds a primary malignant tumor or its metastases has been usually considered a normal tissue . In this work we describe the ultrastructural changes observed in hepatocytes located next to metastases from diverse malignant tumors.Hepatic biopsies were obtained surgically in patients with different malignant tumors which metatastized in liver. Biopsies included tumor mass, the zone of macroscopic contact between the tumor and the surrounding tissue, and the tissue adjacent to the tumor but outside the macroscopic area of infiltration. The patients (n = 5), 36–75 years old, presented different tumors including rhabdomyosarcoma, leiomyosarcoma, pancreas carcinoma, biliar duct carcinoma and colon carcinoma. Tissue samples were processed with routine techniques for transmission electron microscopy and observed in a Hitachi H-500 electron microscope.


1983 ◽  
Vol 44 (C2) ◽  
pp. C2-19-C2-25
Author(s):  
M. C. Gower ◽  
R. G. Caro

1980 ◽  
Vol 41 (C4) ◽  
pp. C4-31-C4-36
Author(s):  
J. R. Meyer ◽  
F. J. Bartoli ◽  
M. R. Kruer

1979 ◽  
Vol 15 (1) ◽  
pp. 254
Author(s):  
KW Kim ◽  
DH Lee ◽  
CY Park

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