Recovery from Brain-Stem Lesions Involving the Nociceptive Pathways: Comparison of Clinical Findings with Laser-Evoked Potentials

1996 ◽  
Vol 13 (4) ◽  
pp. 330-338 ◽  
Author(s):  
H. C. Hansen ◽  
R. D. Treede ◽  
J. Lorenz ◽  
K. Kunze ◽  
B. Bromm
Keyword(s):  
Evoked Potentials ◽  
Brain Stem ◽  
Clinical Findings ◽  
Laser Evoked Potentials ◽  
Nociceptive Pathways ◽  
Brain Stem Lesions ◽  
Stem Lesions

Equilibrium Disorder in Carbamazepine Toxicity

1977 ◽  
Vol 86 (3) ◽  
pp. 318-322 ◽  
Author(s):  
Yoshio Umeda ◽  
Eiji Sakata
Keyword(s):  
Brain Stem ◽  
Optokinetic Nystagmus ◽  
Speech Disorders ◽  
Clinical Findings ◽  
Cerebellar Lesions ◽  
Brain Stem Lesions ◽  
Stem Lesions ◽  
The Brain ◽  
Cerebellar Disorders ◽  
Space Occupying Lesion

Three cases of acute carbamazepine intoxication were evaluated neurotologically and neurologically. Findings included symptoms of equilibrium, gait and speech disorders, drowsiness, gaze nystagmus, depressed optokinetic nystagmus and disturbances of smooth pursuit eye movement. These findings, suggestive of a space-occupying lesion, disappeared after the cessation of the medication. On the basis of the clinical findings it is felt that carbamazepine affects structures within the brain stem and the cerebellum. It is well known that equilibrium disorders caused by anticonvulsant intoxication are due to cerebellar disorders but from our present study it should be noted that they originate not only from cerebellar lesions but also from brain stem lesions.

Dissociation between median and tibial nerve somatosensory evoked potentials (SEPs) in brain-stem lesions

1993 ◽  
Vol 87 (2) ◽  
pp. S32
Author(s):  
M. Gille ◽  
J.M. Guerit ◽  
M. de Tourtchaninoff ◽  
N. Jacquemotte ◽  
J. Delbecq ◽  
...  
Keyword(s):  
Evoked Potentials ◽  
Brain Stem ◽  
Somatosensory Evoked Potentials ◽  
Tibial Nerve ◽  
Brain Stem Lesions ◽  
Stem Lesions

Traumatic Decerebracy with Preserved Consciousness and Voluntary Movement

Neurosurgery ◽  
1985 ◽  
Vol 16 (1) ◽  
pp. 71-74
Author(s):  
Jogi Pattisapu ◽  
Robert R. Smith ◽  
Jose Bebin ◽  
James H. Wood
Keyword(s):  
Brain Stem ◽  
Craniocerebral Trauma ◽  
Physiological Mechanism ◽  
Clinical Findings ◽  
Cerebral Lesions ◽  
Decerebrate Rigidity ◽  
Anatomical Basis ◽  
Brain Stem Lesions ◽  
Stem Lesions ◽  
Unilateral Brain

Abstract Decerebrate rigidity is a frequent occurrence in cases of craniocerebral trauma. It is almost always accompanied by coma and usually denotes a poor prognosis. Primary focal brain stem lesions due to trauma are uncommon and may be accompanied by other diffuse cerebral lesions. This report reviews the anatomical basis and clinical findings of a unilateral brain stem lesion in a patient with traumatic decerebracy and preserved consciousness. The anatomical pathways and possible physiological mechanism are discussed, and a few comments on the prognosis of such lesions are given. (Neurosurgery 16:71–74, 1984)

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