Influence of size scaling and plasma-wall interaction on features of hall thruster microturbulence

Author(s):  
Sedina Tsikata ◽  
Anne Heron ◽  
Cyrille Honore ◽  
Stephane Mazouffre
2019 ◽  
Vol 52 (47) ◽  
pp. 474003 ◽  
Author(s):  
A Domínguez-Vázquez ◽  
F Taccogna ◽  
P Fajardo ◽  
E Ahedo

2002 ◽  
Vol 68 (4) ◽  
pp. 305-319 ◽  
Author(s):  
SUBRATA ROY ◽  
B. P. PANDEY

The dynamics of a Hall thruster is investigated numerically in the presence of a plasma–wall interaction. The plasma–wall interaction is a function of the wall potential, which in turn is determined by the secondary electron emission and sputtering yield. In the present work, the effect of secondary electron emission and sputter yield have been considered simultaneously. Owing to disparate temporal scales, ions and neutrals have been described by a set of time-dependent equations while electrons are considered in a steady state. Based on the experimental observations, a third-order polynomial in electron temperature is used to calculate the ionization rate. The changes in the plasma density, potential and azimuthal electron velocity due to the sputter yield are significant in the acceleration region. The change in ion and electron velocity and temperature is small. The neutral velocity, which decreases initially, starts increasing towards the exit consistent with the computed neutral density profile. The results are qualitatively compared with the experiments.


2001 ◽  
Author(s):  
E. Ahedo ◽  
P. Martinez-Cerezo ◽  
M. Martinez-Sanchez

2003 ◽  
Vol 10 (8) ◽  
pp. 3397-3409 ◽  
Author(s):  
E. Ahedo ◽  
J. M. Gallardo ◽  
M. Martı́nez-Sánchez

1988 ◽  
Vol 60 (02) ◽  
pp. 205-208 ◽  
Author(s):  
Paul A Kyrle ◽  
Felix Stockenhuber ◽  
Brigitte Brenner ◽  
Heinz Gössinger ◽  
Christian Korninger ◽  
...  

SummaryThe formation of prostacyclin (PGI2) and thromboxane A2 and the release of beta-thromboglobulin (beta-TG) at the site of platelet-vessel wall interaction, i.e. in blood emerging from a standardized injury of the micro vasculature made to determine bleeding time, was studied in patients with end-stage chronic renal failure undergoing regular haemodialysis and in normal subjects. In the uraemic patients, levels of 6-keto-prostaglandin F1α (6-keto-PGF1α) were 1.3-fold to 6.3-fold higher than the corresponding values in the control subjects indicating an increased PGI2 formation in chronic uraemia. Formation of thromboxane B2 (TxB2) at the site of plug formation in vivo and during whole blood clotting in vitro was similar in the uraemic subjects and in the normals excluding a major defect in platelet prostaglandin metabolism in chronic renal failure. Significantly smaller amounts of beta-TG were found in blood obtained from the site of vascular injury as well as after in vitro blood clotting in patients with chronic renal failure indicating an impairment of the a-granule release in chronic uraemia. We therefore conclude that the haemorrhagic diathesis commonly seen in patients with chronic renal failure is - at least partially - due to an acquired defect of the platelet a-granule release and an increased generation of PGI2 in the micro vasculature.


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