scholarly journals Case Report: Pheochromocytoma. Aspects of Management

1976 ◽  
Vol 4 (2) ◽  
pp. 156-158 ◽  
Author(s):  
P. J. Maddern ◽  
N. J. Davis ◽  
I. McGlew ◽  
T. Oh
Keyword(s):  
Blood Pressure ◽  
Case Report ◽  
Pulmonary Artery ◽  
Sodium Nitroprusside ◽  
Pulmonary Artery Wedge Pressure ◽  
Pressure Monitoring ◽  
Arterial Blood ◽  
Beta Blocking ◽  
Alpha Blockade ◽  
Wedge Pressure

The management of a patient with Pheochromocytoma is reported. Consideration of pre-operative preparation, hazards of beta-blocking agents in the absence of alpha blockade, potential hazards of butyrophenones, control of arterial blood pressure with sodium nitroprusside and the usefulness of continuous pulmonary artery wedge pressure monitoring are discussed.

Pulmonary Artery Wedge Pressure Respiratory Variation Increases With Sodium Nitroprusside Vasodilator Challenge

2020 ◽  
Vol 26 (12) ◽  
pp. 1096-1099
Author(s):  
Steven P. Maurides ◽  
Devin Blankinship ◽  
Kavin Panneerselvam ◽  
Gregory R. Jackson ◽  
Stefano Ghio ◽  
...  
Keyword(s):  
Pulmonary Artery ◽  
Sodium Nitroprusside ◽  
Respiratory Variation ◽  
Pulmonary Artery Wedge Pressure ◽  
Wedge Pressure

Regulation of Cardiac Output during 2,4-Dinitrophenol-Induced Tissue Hypermetabolism in the Dog

1977 ◽  
Vol 53 (1) ◽  
pp. 17-25
Author(s):  
C. Liang ◽  
W. B. Hood
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Cardiac Output ◽  
Arterial Blood Pressure ◽  
Venous Blood ◽  
The Other ◽  
Pulmonary Artery Wedge Pressure ◽  
Systemic Arterial Blood Pressure ◽  
Arterial Blood ◽  
Wedge Pressure

1. Cardiac output increased in proportion to oxygen consumption in intact chloralose-anaesthetized dogs after four successive intravenous infusions of 2,4-dinitrophenol (11 μmol/kg; 2 mg/kg). 2. Splenectomy abolished the increase in cardiac output after the first three doses of 2,4-dinitrophenol. β-Adrenoreceptor blockade by practolol, on the other hand, did not prevent the cardiac output rise after the first 2,4-dinitrophenol infusion, but further increases by 2,4-dinitrophenol infusion were abolished. When splenectomy and β-adrenoreceptor blockade were combined, cardiac output did not increase significantly after all four doses of 2,4-dinitrophenol. 3. Cardiac output and mean systemic arterial blood pressure increased when the splenic venous blood collected after 2,4-dinitrophenol infusion was infused intraportally. 4. In a vascularly isolated, but normally innervated, lower half-body cross-perfusion preparation, cardiac output and mean systemic arterial blood pressure increased in the upper half-body when tissue hypermetabolism was produced in the cross-perfused area by 2,4-dinitrophenol. Neither pulmonary artery wedge pressure nor heart rate changed significantly. 5. This circulatory stimulation, after regional 2,4-dinitrophenol infusion, was abolished or was prevented from occurring by splenectomy. 6. It appears that the normal cardiac output response to tissue hypermetabolism requires both an intact spleen and normally functioning β-adrenoreceptors.

Haemodynamic Monitoring in Theatre

2003 ◽  
Vol 4 (1) ◽  
pp. 10-16 ◽  
Author(s):  
Heidi Clinton
Keyword(s):  
Blood Pressure ◽  
Pulmonary Artery ◽  
Central Venous Pressure ◽  
Venous Pressure ◽  
Haemodynamic Monitoring ◽  
Pressure Monitoring ◽  
Central Venous ◽  
Noninvasive Measurements ◽  
Non Invasive ◽  
Arterial Blood

AbstractThe number of devices available to monitor the haemodynamic status of patients is increasing. Practitioners need to be aware of the non-invasive and invasive methods available in order to care for their patients safely and effectively. This article reviews a number of noninvasive measurements of haemodynamic function, in addition to invasive methods such as arterial blood pressure, central venous pressure and pulmonary artery pressure monitoring. It is argued that using these methods in combination provides a comprehensive haemodynamic assessment.

Pulmonary Endarterectomy: Assessment of Operability, Surgical Description, and Post-op Care

2014 ◽  
Vol 12 (4) ◽  
pp. 186-192 ◽  
Author(s):  
David Poch ◽  
Victor Pretorius
Keyword(s):  
Pulmonary Hypertension ◽  
Pulmonary Artery ◽  
Pulmonary Artery Pressure ◽  
Pulmonary Arteries ◽  
Chronic Thromboembolic Pulmonary Hypertension ◽  
Pulmonary Artery Wedge Pressure ◽  
Pulmonary Endarterectomy ◽  
Pulmonary Thromboendarterectomy ◽  
Thromboembolic Pulmonary Hypertension ◽  
Wedge Pressure

Chronic thromboembolic pulmonary hypertension (CTEPH) is defined as a mean pulmonary artery pressure ≥25 mm Hg and pulmonary artery wedge pressure ≤15 mm Hg in the presence of occlusive thrombi within the pulmonary arteries. Surgical pulmonary thromboendarterectomy (PTE) is considered the best treatment option for CTEPH.

A Comparison of the Acute Haemodynamic Effects of Propranolol and Pindolol at Rest and during Supine Exercise in Man

1980 ◽  
Vol 59 (s6) ◽  
pp. 465s-468s ◽  
Author(s):  
T. L. Svendsen ◽  
J. E. Carlsen ◽  
O. Hartling ◽  
A. McNair ◽  
J. Trap-Jensen
Keyword(s):  
Blood Pressure ◽  
Heart Rate ◽  
Cardiac Output ◽  
Pulmonary Artery ◽  
Pulmonary Artery Pressure ◽  
Response Curves ◽  
Receptor Blocking ◽  
Artery Pressure ◽  
Arterial Blood ◽  
Β Receptor

1. Dose-response curves for heart rate, cardiac output, arterial blood pressure and pulmonary artery pressure were obtained in 16 male patients after intravenous administration of three increasing doses of pindolol, propranolol or placebo. All patients had an uncomplicated acute myocardial infarction 6–8 months earlier. 2. The dose-response curves were obtained at rest and during repeated bouts of supine bicycle exercise. The cumulative dose amounted to 0.024 mg/kg body weight for pindolol and to 0.192 mg/kg body weight for propranolol. 3. At rest propranolol significantly reduced heart rate and cardiac output by 12% and 15% respectively. Arterial mean blood pressure was reduced by 9.2 mmHg. Mean pulmonary artery pressure increased significantly by 2 mmHg. Statistically significant changes in these variables were not seen after pindolol or placebo. 4. During exercise pindolol and propranolol both reduced cardiac output, heart rate and arterial blood pressure to the same extent. After propranolol mean pulmonary artery pressure was increased significantly by 3.6 mmHg. Pindolol and placebo did not change pulmonary artery pressure significantly. 5. The study suggests that pindolol may offer haemodynamic advantages over β-receptor-blocking agents without intrinsic sympathomimetic activity during low activity of the sympathetic nervous system, and may be preferable in situations where the β-receptor-blocking effect is required only during physical or psychic stress.

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