scholarly journals Foreign body a fish in orohypopharynx with complication vocal cord paralysis

2018 ◽  
Vol 41 (1) ◽  
pp. 32
Author(s):  
Ade Asyari ◽  
Novialdi Novialdi ◽  
Nur Azizah
Keyword(s):  
Foreign Body ◽  
Vocal Cord ◽  
Endotracheal Intubation ◽  
Rare Case ◽  
Vocal Cord Paralysis ◽  
Rare Complication ◽  
Upper Airway ◽  
Pharyngeal Wall ◽  
Magill Forceps ◽  
Rigid Esophagoscopy

Introduction: Foreign body a fish in oro-hypopharynx is a rare case and require rapid diagnosis and immediate treatment to prevent complication. There are some complications that can occur, such as upper airway obstruction, perforation of the pharyngeal wall, vocal cord paralysis, pneumomediastinum, and emphysema. Vocal cord paralysis is rare complication caused by a foreign body in the pharynx. The management for pharyngeal foreign bodies is the extraction of a foreign body with Magill forceps, direct laryngoscopy, and rigid endoscopy. Tracheostomy should be performed if endotracheal intubation could not be done or failed to be performed. Objective: Understanding diagnosis and management of patient foreign body a fish in an oro-hypopharynx. Case report: Reported a case, male 40 years old, with diagnosis foreign body a fish in oro-hypopharynx with complication unilateral vocal cord paralysis. The Foreign body was extracted using Magill forceps and rigid esophagoscopy with tracheostomy preparation if endotracheal intubation was failed to perform. Conclusion: Foreign body a fish in oro-hypopharynx is a rare case. Precise diagnosis and treatment are very important to prevent complication. Vocal cord paralysis is a rare complication caused by a foreign body in oro-hypopharynx.

scholarly journals A Rare Case of a Hypopharyngeal Fish Bone Foreign Body with Vocal Cord Paralysis.

1994 ◽  
Vol 45 (3) ◽  
pp. 249-252
Author(s):  
Masaharu Sudo ◽  
Masahiro Tanabe ◽  
Manabu Minoyama ◽  
Tadahiko Sugimaru ◽  
Michitaka Iwanaga
Keyword(s):  
Foreign Body ◽  
Vocal Cord ◽  
Rare Case ◽  
Vocal Cord Paralysis ◽  
Fish Bone

Stridor

1981 ◽  
Vol 3 (5) ◽  
pp. 146-165
Keyword(s):  
Foreign Body ◽  
Vocal Cord ◽  
Endotracheal Intubation ◽  
Congenital Anomalies ◽  
Vocal Cord Paralysis ◽  
Subglottic Stenosis ◽  
Presumptive Diagnosis ◽  
Delay In Diagnosis ◽  
Vascular Rings ◽  
Early Endoscopy

There were 219 patients less than 2½ years of age who had stridor and were referred to an otolaryngologist. Eighty-seven percent had stridor due to congenital anomalies; 50% of these were laryngotracheomalacia; less frequent were subglottic stenosis (15%), vocal cord paralysis (9%), and vascular rings (8%). Other causes which were less common (for referral) included acquired conditions such as atypical laryngotracheobronchitis (prolonged symptoms), unrecognized foreign body, and stenosis after prolonged endotracheal intubation. Approximately 25% of patients had an erroneous presumptive diagnosis (asthma, croup, bronchiolitis) which led to a delay in diagnosis of several months. Early endoscopy was helpful in diagnosis.

Unilateral vocal cord paralysis following endotracheal intubation

1998 ◽  
Vol 42 (1) ◽  
pp. 131-132 ◽  
Author(s):  
R. J. LaursenM.D ◽  
K. M. Larsen ◽  
J. Mølgaard ◽  
V. Kolze
Keyword(s):  
Vocal Cord ◽  
Endotracheal Intubation ◽  
Vocal Cord Paralysis ◽  
Unilateral Vocal Cord Paralysis

scholarly journals Vocal Cord Paralysis Following Endotracheal Intubation

2017 ◽  
Vol 45 (5) ◽  
pp. 321-322
Author(s):  
Hamdi Tasli ◽  
Umut Kara ◽  
Mert Cemal Gokgoz ◽  
Umit Aydin
Keyword(s):  
Vocal Cord ◽  
Endotracheal Intubation ◽  
Vocal Cord Paralysis

S183 – Intracordal Fat Assessment after Injection Laryngoplasty

2008 ◽  
Vol 139 (2_suppl) ◽  
pp. P137-P137
Author(s):  
Tuan-Jen Fang ◽  
Chao-Jan Wang ◽  
Hsueh-Yu Li
Keyword(s):  
Vocal Cord ◽  
Vocal Cord Paralysis ◽  
Upper Airway ◽  
Fat Injection ◽  
Long Term Outcomes ◽  
Autologous Fat ◽  
The Mean ◽  
Fat Volume ◽  
Unilateral Vocal Cord Paralysis

Objectives Autologous fat injection for unilateral vocal cord paralysis is a popular procedure for immediate symptoms control, but uneven long-term outcomes were reported. Most authors believed that the continual resorption of injected fat was the cause of voice degradation. A long-term residual fat volume was evaluated. Methods We retrospectively reviewed the patients following autologous fat injection for symptomatic unilateral vocal cord paralysis from 2002 Aug to 2006 July. The patients accepted head and neck computed tomogragphy (CT) evaluation following surgery were included. A three-dimensional reconstruction of the images of larynx and upper airway was performed on the work station (Vitrea® 2, version 3.9). The volume of intracordal fat was then calculated. Results 5 males and 15 females of the mean age 49 were enrolled. The mean duration from lipoinjection surgery to the CT study is 23.0 months (12–50 months). The injection fat was found in all study cases. The estimated fat volume ranged from 0.01 to 0.75 ml with a mean 0.33 ml. Compare with the injected fat volume, a mean 27.5% fat survived. The correlation between residual fat volume rate and duration of follow-up is not significant. Conclusions The intracordal fat volume didn't decline from time. Long-term intracordal injected fat diminished but survived in all cases. The degradation may be caused by absorption or immediate extrusion from injection wound. An over-correction and prevention of extrusion postoperatively would improve the long-term outcomes.

Posterior Glottic Laryngeal Stenosis

1980 ◽  
Vol 88 (6) ◽  
pp. 765-772 ◽  
Author(s):  
Ronald S. Bogdasarian ◽  
Nels R. Olson
Keyword(s):  
Airway Obstruction ◽  
Vocal Cord ◽  
Endotracheal Intubation ◽  
Vocal Cord Paralysis ◽  
Voice Quality ◽  
Posterior Commissure ◽  
Muscle Scar ◽  
Laryngeal Stenosis ◽  
Scar Contracture ◽  
Laryngoscopic Examination

Posterior glottic laryngeal stenosis most commonly results from endotracheal intubation and less commonly from external trauma or from infection. Following extubation, the patient may have immediate or delayed onset of symptoms of airway obstruction. Often, as in bilateral vocal cord paralysis, voice symptoms are minimal. Indirect laryngoscopic examination usually establishes the diagnosis. Movement of the arytenoids is seen but is limited, and is reflected in poor mobility of the vocal cords. Direct laryngoscopic examination and palpation of the arytenoids for passive mobility confirms the diagnosis and rules out vocal cord paralysis from recurrent laryngeal nerve injury. With the establishment of stenosis, scarring and web formation occurs over the posterior cricoid lamina and may extend into one or both cricoarytenoid joints and into the interarytenoid muscle. Scar contracture in the posterior commissure causes medial fixation of the vocal processes of the arytenoid cartilages. When caused by endotracheal intubation, the initial injury is usually to the mucosa of the posterior cricoid lamina, vocal processes of the arytenoids, or both. Perichondritis may ensue, its location and severity determining the ultimate functional extent of scarring. When airway obstruction results, treatment is by laryngofissure, scar resection, grafting, and stenting. If bilateral cricoarytenoid joint fibrosis is discovered, arytenoidectomy, and in some cases laryngeal lumen augmentation, is invariably required to reestablish the airway, with probable detrimental effects on voice quality. Five of ten patients are presented to illustrate the etiology, pathogenesis, symptoms, management, and sequelae of this problem.

scholarly journals A Rare Presentation of Bilateral Vocal Cord Paralysis in Late Stage Parkinson’s Disease

2018 ◽  
Vol 5 ◽  
Author(s):  
Sharir Asrul Bin Asnawi ◽  
Mohd Hazmi Bin Mohamed ◽  
Mohamad Bin Doi
Keyword(s):  
Parkinson’S Disease ◽  
Parkinson's Disease ◽  
Multiple System Atrophy ◽  
Medical Treatment ◽  
Airway Obstruction ◽  
Vocal Cord ◽  
Vocal Cord Paralysis ◽  
Upper Airway ◽  
Upper Airway Obstruction ◽  
Bilateral Vocal Cord Paralysis

Introduction: Vocal cord paralysis often causes mortality by upper airway obstruction in some neurodegenerative diseases such as Parkinson’s disease and multiple system atrophy. Vocal cord paralysis is uncommon in Parkinson’s disease (PD) in contrary it is more common in multiple system atrophy (MSA). The pathogenesis of vocal cord paralysis in Parkinson’s disease is not well understood but may involve degeneration of the nucleus ambiguous. In terms of managing patient with bilateral vocal cord paralysis in PD, it can be either performing tracheostomy to relive the upper airway obstruction or by optimizing the medical treatment. There are very few available reported cases whereby patient are treated with medical treatment alone.Case Report: We report a case of 65 years old lady who presented with stridor resulting from bilateral vocal cord paralysis and she has been diagnosed to have Parkinson’s disease for more than 10 years. She had her antiparkinson medication optimized and requiring no surgical intervention to relieve the upper airway obstruction.Conclusion: In conclusion we would like to emphasize that it is important to recognize bilateral vocal cord paralysis in Parkinson’s disease and early optimization of medical treatment could avoid a need of tracheostomy.

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