scholarly journals Clinical Features of Reiter’s Syndrome in Children

2020 ◽  
Vol 6 (5) ◽  
pp. 137-146
Author(s):  
K. Sayakbekov ◽  
G. Saatova ◽  
G. Maimerova
Keyword(s):  
Lumbar Spine ◽  
Clinical Features ◽  
Lower Extremities ◽  
Reiter's Syndrome ◽  
Intense Pain ◽  
Hip Joints ◽  
Reiter’S Syndrome ◽  
Chronic Course ◽  
Extraarticular Manifestations

The clinical features of articular syndrome and extra-articular manifestations in children with Reiter’s syndrome (illness) were studied. Reiter’s syndrome in childhood has some features of the course and development of the disease. In childhood, asymmetry of lesions of the joints of the lower extremities with early involvement of small feet joints in the process, possible flushing of the skin over the affected joints, intense pain, rare involvement in the process of the hip joints, lumbar spine and unilateral sacroiliitis is characteristic. Extraarticular manifestations proceeded often erased, and sometimes were not determined at all. Relapses and a chronic course are not excluded.

HLA B27 and clinical features in Reiter's syndrome

1992 ◽  
Vol 11 (2) ◽  
pp. 239-242 ◽  
Author(s):  
T. Tuncer ◽  
M. Í. Arman ◽  
A. Akyokus ◽  
B. Bütün ◽  
S. Ünal
Keyword(s):  
Clinical Features ◽  
Hla B27 ◽  
Reiter's Syndrome ◽  
Reiter’S Syndrome

Reiter's syndrome

1966 ◽  
Vol 94 (5) ◽  
pp. 666-667
Author(s):  
J. Levine
Keyword(s):  
Reiter's Syndrome ◽  
Reiter’S Syndrome

An "experimental" epidemic of Reiter's syndrome

JAMA ◽  
1966 ◽  
Vol 198 (7) ◽  
pp. 693-698 ◽  
Author(s):  
H. R. Noer
Keyword(s):  
Reiter's Syndrome ◽  
Reiter’S Syndrome

Arthritis and Foodborne Bacteria

1994 ◽  
Vol 57 (10) ◽  
pp. 935-941 ◽  
Author(s):  
JAMES L. SMITH
Keyword(s):  
T Cells ◽  
Ankylosing Spondylitis ◽  
Reactive Arthritis ◽  
Molecular Mimicry ◽  
Food Poisoning ◽  
Cell Subset ◽  
Bacterial Cells ◽  
Hla B27 ◽  
Reiter's Syndrome ◽  
Reiter’S Syndrome

Diarrheic episodes caused by the foodborne pathogens Campylobacter, Salmonella, Shigella or Yersinia may lead to a sterile arthritis such as reactive arthritis, Reiter's syndrome or ankylosing spondylitis. Reiter's syndrome and reactive arthritis have been shown to be sequelae in a few well-studied bacterial food poisoning outbreaks. Reactive arthritis, Reiter's syndrome and ankylosing spondylitis show strong familial association related to the gene for HLA-B27 (HLA = human leucocyte antigen) antigen. Why HLA-B27-positive individuals are more susceptible to arthritis is not known, but molecular mimicry between the HLA-B27 antigen and antigens of triggering bacteria has been demonstrated and this mimicry has been proposed as a mechanism involved in etiology of the arthritides. Antigens from bacteria that triggered the arthritis are present in arthritic joints but bacterial cells are not found. Antibodies and T-cells specific for the triggering bacteria have been demonstrated in arthritic patients. T-cells present in synovial joints respond specifically to the particular arthritic triggering pathogen. The cells that respond to bacterial antigens belong to the T-cell subset TH1 that secrete a limited number of cytokines but it is not known if cytokines are involved in arthritis. A few studies have demonstrated that T-cells from the joints of arthritic patients respond to both bacterial and human heat shock proteins indicating that autoimmunity may be involved in causation of arthritis. While only about 2% of a population exposed to a triggering infection will acquire arthritis, these individuals undergo pain and suffering as well as economic hardships as a result of their disease.

The radiographic pattern of polyarthritis in Reiter's syndrome

1971 ◽  
Vol 14 (4) ◽  
pp. 551-555 ◽  
Author(s):  
Stephen D. Sholkoff ◽  
Morton G. Glickman ◽  
Howard L. Steinbach
Keyword(s):  
Reiter's Syndrome ◽  
Reiter’S Syndrome
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