Idiopathic trigeminal sensory neuropathy. A case report

2013 ◽  
Vol 47 (5) ◽  
pp. 499-501 ◽  
Author(s):  
Piotr Szczudlik ◽  
Biruta Kierdaszuk ◽  
Leopold Bakoń ◽  
Edyta Maj ◽  
Anna Kamińska
2015 ◽  
Vol 126 (1) ◽  
pp. e15-e16
Author(s):  
S. Vigneri ◽  
V. Rispoli ◽  
C. Azzini ◽  
M.L. Caniatti ◽  
M.R. Tola ◽  
...  

1990 ◽  
Vol 237 (1) ◽  
pp. 42-44 ◽  
Author(s):  
F. Kanda ◽  
T. Uchida ◽  
K. Jinnai ◽  
K. Tada ◽  
S. Shiozawa ◽  
...  

BMC Neurology ◽  
2019 ◽  
Vol 19 (1) ◽  
Author(s):  
Florian Rimmele ◽  
Henning Maschke ◽  
Annette Großmann ◽  
Tim P. Jürgens

Abstract Background Numb Chin Syndrome (NCS), which is also characterized as sensory neuropathy of the mental nerve, describes a mostly unilateral numbness of the chin and lower lip. Benign and malignant diseases are known to cause this circumscribed symptom, which can easily be overlooked or misdiagnosed. In this article we present the very rare case of a clinical NCS caused by thalamic lacunar infarction. As a pure sensory stroke it is a rare variant of the Cheiro-Oral Syndrome (COS). Case presentation A 63-year-old male patient received an emergency referral to our department after the patient had noticed a feeling of numbness of the left lower lip and chin on the previous day. The neurological examination revealed an approximately 2 × 3 cm area of hypoesthesia in the area of the chin and left lower lip and the cranial MRI an acute ischemia in the right thalamus. Conclusions In this case report we introduce a patient who clinically shows an NCS. Various diseases may be responsible for NCS, including malignancies or even central neurological disorders such as multiple sclerosis. A lacunar thalamic ischemia as a cause of NCS is very rare and to our knowledge described in the literature only in the contex of a COS in three cases. We wish to remind the reader, through this case, of the purely descriptive and syndromal character of the NCS and the importance for detecting underlying diseases. Furthermore we give a brief overview of the NCS and causative disorders.


2022 ◽  
pp. 107815522110735
Author(s):  
B. Zarei ◽  
M. Moeini Nodeh ◽  
O. Arasteh

Introduction Oxaliplatin is a third-generation platinum compound that used extensively for the treatment of various types of cancer especially gastrointestinal neoplasms. The main dose-limiting toxicities of oxaliplatin are hematological toxicity and peripheral sensory neuropathy. Case report A 42-year-old man with refractory peripheral T-cell lymphoma (PTCL) was admitted to receive GEMOX chemotherapy regimen (gemcitabine, oxaliplatin). Three days after receiving his third cycle of chemotherapy regimen, he was re-admitted to the emergency department with complaint of severe generalized weakness, and paraplegia in the lower extremities. According to clinical and para-clinical findings, chronic sensorimotor polyneuropathy with ongoing axonal loss was confirmed. Management & Outcome Intravenous dexamethasone 8 mg three times daily was started at the time of admission for the patient. Muscle weakness and sensory impairment improved dramatically within 10 days and the patient was able to walk with assistance. Discussion Several cases of neuropathy following oxaliplatin and only one case with gemcitabine-based chemotherapy regimen have been previously reported. However, motor symptoms are rare unless in the setting of acute neuropathy due to oxaliplatin. The most striking finding of our study was the incidence of a chronic sensorimotor axonaldemyelinating polyneuropathy in a patient who were subjected to oxaliplatin therapy. In conclusion, we report a case of severe generalized weakness and paraplegia following administration of Oxaliplatin.


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