scholarly journals Neurogenic Pulmonary Edema Caused by Subarachnoid Hemorrhage: A Case Report

2021 ◽  
Vol 1 (2) ◽  
pp. 1-5
Author(s):  
Tao Zhu

Neurogenic pulmonary edema is a serious and life-threatening complication caused by central nervous system diseases, excluding cardiogenic pulmonary edema, other causes of pulmonary edema, lung injury, etc. The lack of specific diagnostic criteria for NPE and the lack of awareness among clinicians often lead to underdiagnosis and misdiagnosis. The disease progresses rapidly with poor prognosis and high mortality [1]. In this paper, we report a patient with a ruptured right middle cerebral artery aneurysm causing subarachnoid hemorrhage, who developed neurogenic pulmonary edema and underwent elective aneurysm clamping under general anesthesia. The relevant perioperative management is reported as follows, and the anesthesia management of patients with neurogenic pulmonary edema is analyzed in the context of the relevant literature.

2021 ◽  
Vol 104 (1) ◽  
pp. 68-72

Background: Subarachnoid hemorrhage has a high mortality and morbidity rates, and the cerebral aneurysm is the most common cause. The location of the ruptured cerebral aneurysm is diagnosed by cerebral angiogram and by computed tomography (CT) pattern of the subarachnoid hemorrhage. Objective: To find the incidence of CT pattern of each cerebral aneurysm and the correlation between CT pattern of subarachnoid hemorrhage and each location of cerebral aneurysm. Materials and Methods: The 126 patients with ruptured cerebral aneurysm were included in the present study. The information of the patients and CT were recorded and analyzed. Results: There were 49 male and 77 female patients. The highest location of cerebral aneurysm is anterior communicating aneurysm (59 patients, 47.6%). There were correlations between A region and anterior cerebral and anterior communicating aneurysm, and left D legion and the other location aneurysm. There were correlations between intracerebral hemorrhage and anterior communication, posterior communication cerebral, and anterior cerebral and middle cerebral artery aneurysm. Conclusion: The common location of cerebral aneurysm is anterior communicating artery aneurysm. There are correlations between the most thickness region of A and D region with anterior communicating aneurysm and the posterior circulation aneurysm, and the correlation between intracerebral hemorrhage and anterior communicating aneurysm, posterior communicating aneurysm, and middle cerebral artery aneurysm. Keywords: Subarachnoid hemorrhage, Cerebral aneurysm, Intracerebral hemorrhage, Thickness


Neurosurgery ◽  
1986 ◽  
Vol 19 (2) ◽  
pp. 300-304 ◽  
Author(s):  
Michio Yabumoto ◽  
Tsuyoshi Kuriyama ◽  
Munehisa Iwamoto ◽  
Tatsuyuki Kinoshita

Abstract A case of ruptured distal anterior cerebral artery aneurysm associated with neurogenic pulmonary edema is presented. It is suggested that this association should not be accepted as a “taboo” for radical intervention followed by a proper management of ventilation with positive end-expiratory pressure to maintain the anesthetic condition. Cardiorespiratory control is essential in cases of pulmonary edema with ruptured aneurysm. Decompression and evacuation of blood clot surrounding the hypothalamus could subdue the hyperadrenosympathetic discharge that may cause neurogenic pulmonary edema.


2019 ◽  
Vol 76 (10) ◽  
pp. 1087-1093
Author(s):  
Branko Milakovic ◽  
Tijana Nastasovic ◽  
Milan Lepic ◽  
Nenad Novakovic ◽  
Sinisa Matic ◽  
...  

Introduction. Subarachnoid haemorrhage (SAH) can be followed by cardiac abnormalities. We describe a patient with Takotsubo cardiomyopathy and neurogenic pulmonary edema (NPE) after aneurysmal SAH. Case report. A previously healthy, postmenopausal woman, suffered from aneurysmal SAH with consequent hydrocephalus. After external ventricular drainage, craniotomy and clipping of the posterior inferior cerebellar artery aneurysm, the patient developed acute heart failure and NPE. Transthoracic echocardiogram showed the left ventricular apical ballooning and hypercontractile basal segments. On chest radiography, bilateral pulmonary infiltrates were seen. Seventeen days after the SAH attack, the patient was discharged from hospital. Postponed coronary angiography revealed no signs of coronary artery disease. Conclusion. This case and review of the relevant literature suggest that Takotsubo cardiomyopathy and neurogenic pulmonary edema are not uncommon after aneurysmal SAH.


2015 ◽  
Vol 39 (videosuppl1) ◽  
pp. V8
Author(s):  
Leopold Arko ◽  
Eric Quach ◽  
Vishad Sukul ◽  
Anuj Desai ◽  
Kelly Gassie ◽  
...  

We present surgical clipping of a giant middle cerebral artery aneurysm. The patient is a 64-year-old woman who suffered subarachnoid hemorrhage in 2005. She was treated with coiling of the aneurysm at an outside institution. She presented to our clinic with headaches and was found on angiography to have giant recurrence of the aneurysm. To allow adequate exposure for clipping, we performed the surgery through a cranio-orbito-zygomatic (COZ) skull base approach, which is demonstrated. The surgery was performed in an operating room/angiography hybrid suite allowing for high quality intraoperative angiography. The technique and room flow are also demonstrated.The video can be found here: http://youtu.be/eePcyOMi85M.


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