The Role of Sensorimotor Rhythmic EEG Activity in the Etiology and Treatment of Generalized Motor Seizures

Author(s):  
M. Barry Sterman
Keyword(s):  
1996 ◽  
Vol 99 (4) ◽  
pp. 320 ◽  
Author(s):  
S. Suljagic ◽  
N. Rajsic ◽  
J. Ivanus ◽  
Z. Bozovic ◽  
A. Kalauzi ◽  
...  

2019 ◽  
Vol 52 (4) ◽  
pp. 472-484
Author(s):  
P Siemiginowska ◽  
I Iskra-Golec

There is growing evidence for monochromatic blue light effects. However, the role of individual differences in it has not yet been explored. The aim of this experiment was to examine whether chronotype could moderate the monochromatic blue light effect on electroencephalographic (EEG) activity with regard to the timing of exposure. The participants were 30 young male volunteers. A within-subjects counterbalanced design was applied. There were two light conditions comparable in luminance: Monochromatic blue light of 460 nm and polychromatic white light of 6.5 lux. EEG measurements were taken after 4 hours of exposure in the morning, afternoon, and evening. EEG spectral power was categorized into five frequency ranges: delta, theta, alpha1, alpha2, and beta. Chronotypes were assessed by the Morningness-Eveningness Questionnaire. A mixed analysis of variance was applied. Significant interactions between chronotype, light conditions, and the time of the day were found in theta and alpha1 bands after exposure to monochromatic blue light. These preliminary results indicated that in morning-oriented types the spectral power of theta and alpha1 EEG bands was higher in monochromatic blue light when compared to polychromatic white light in the afternoon hours than in the morning or the evening hours. These results may indicate a decrease in alertness in monochromatic blue light in the afternoon hours in morning-oriented types. This could point to the moderating role of individual differences in the monochromatic blue light effect.


1970 ◽  
Vol 17 (2) ◽  
pp. 259-275 ◽  
Author(s):  
A. Costin ◽  
D. Hafemann ◽  
Z. Elazar ◽  
W.R. Adey
Keyword(s):  

2019 ◽  
Vol 131 ◽  
pp. 104542 ◽  
Author(s):  
Yu Fu ◽  
Liane Li ◽  
Yumei Wang ◽  
Guanghui Chu ◽  
Xiangyang Kong ◽  
...  

2015 ◽  
Vol 52 (10) ◽  
pp. 1375-1381 ◽  
Author(s):  
Philip Cheng ◽  
Jennifer Goldschmied ◽  
Patricia Deldin ◽  
Robert Hoffmann ◽  
Roseanne Armitage

1993 ◽  
Vol 265 (2) ◽  
pp. R433-R438 ◽  
Author(s):  
P. Y. Cheng ◽  
D. Wu ◽  
Y. Soong ◽  
S. McCabe ◽  
J. A. Decena ◽  
...  

Recent evidence suggests that administration of low doses of morphine causes respiratory stimulation, along with a more active electroencephalogram (EEG) in the fetal lamb. The present study used selective opioid agonists and antagonists to determine the role mu 1- and delta-opioid receptor subtypes play in the response as well as determine if endogenous opioid peptides exert a tonic influence at the mu 1- and delta-opioid receptors to maintain normal EEG and respiratory activity under control, physiological conditions. Both morphine (2.5 mg/h iv) and [D-Pen2,D-Pen5]enkephalin (DPDPE) (46 nmol/h icv) resulted in a significant activation of fetal EEG, which was blocked by naloxonazine (NALZ, mu 1-opioid antagonist) and naltrindole (NTI, delta-opioid antagonist), respectively. Administration of NALZ alone, but not NTI, resulted in a slowing of the EEG. Morphine and [D-Ala2]deltorphin I (0.36 nmol/h icv) significantly increased breath number and were blocked by NALZ and NTI respectively. Both NALZ and NTI alone resulted in a reduction in breath number. These results suggest that the activation of the delta- or mu 1-opioid receptors will stimulate fetal respiratory and EEG activity. Furthermore, the endogenous opioids play a tonic role at both the delta- and mu 1-opioid receptors in the regulation of respiratory timing and EEG activity.


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