Hepatolithiasis, cystic dilatation of bile ducts and the variable risk of cholangiocarcinoma. Re-evaluation of an old problem

1999 ◽  
pp. 55-67
Author(s):  
Francesco Cetta ◽  
Giulia Montalto ◽  
Claudio Baldi ◽  
Michele Zuckermann ◽  
Gennaro Nuzzo
Keyword(s):  
Author(s):  
Olival Cirilo Lucena da FONSECA-NETO ◽  
Moacir Cavalcante de ALBUQUERQUE-NETO ◽  
Antonio Lopes de MIRANDA

BACKGROUND: The cystic dilatation of the biliary tract is a rare disease and uncertain origin. It is recognized more frequently in children; however, its incidence comes increasing in adults, representing 20% of the cases. AIM: To evaluate morbimortality rates, evolution and handing of patients with cystic dilatation bile ducts in adults. METHODS: Were evaluated, retrospectively, five adults who had the diagnosis of choledochal cyst and that had been submitted to some surgical procedure. RESULTS: Abdominal pain was the commonest complain to all patients. Jaundice was present in 80%. Ultrasound scanning was done in all the cases as initial examination. CT scan, magnetic resonance imaging and endoscopic retrograde cholangiopancreatography were also done in some patients; however, the diagnosis was established intra-operatively in all cases. The cyst resection with reconstruction of the biliary tract was done in 60%; the cystojejunostomy in 20%; and in 20% biliary tract drainage. CONCLUSIONS: Biliary tract cystic dilatation is a rare disease. However, its incidence is increasing in the adult population, so, it must be thought as differential diagnosis when facing obstructive jaundice.


1979 ◽  
Vol 40 (4) ◽  
pp. 672-678
Author(s):  
Keishoku CHO ◽  
Teizo TODO ◽  
Mitsugu NITTA ◽  
Myoshin RAI ◽  
Hiloyosi DOHI ◽  
...  

2008 ◽  
Vol 38 (2) ◽  
pp. 140-142
Author(s):  
A. Koulaouzidis ◽  
M. Haris ◽  
S. Bhat ◽  
S. Clark ◽  
M. Kaleem ◽  
...  

1989 ◽  
Vol 34 (3) ◽  
pp. 367-371 ◽  
Author(s):  
Julio Ponce ◽  
Vicente Garrigues ◽  
Teresa Sala ◽  
Virginia Pertejo ◽  
Joaqu�n Berenguer

1990 ◽  
Vol 11 (2) ◽  
pp. 196-199 ◽  
Author(s):  
Jean Genève ◽  
Nathalie Dubuc ◽  
Didier Mathieu ◽  
Elie Serge Zafrani ◽  
Daniel Dhumeaux ◽  
...  

2001 ◽  
Vol 17 (2-3) ◽  
pp. 108-110 ◽  
Author(s):  
H. Komuro ◽  
S. Makino ◽  
T. Momoya ◽  
Y. Uehara ◽  
K. Tahara ◽  
...  

1998 ◽  
Vol 114 ◽  
pp. A1220
Author(s):  
F Cetta ◽  
G Nuzzo ◽  
Y. Nimura
Keyword(s):  

1944 ◽  
Vol 80 (3) ◽  
pp. 231-246 ◽  
Author(s):  
Eugene L. Opie

A conspicuous feature of the cirrhosis that is produced by butter yellow is the new formation of bile ducts that sprout from ducts of the larger portal spaces and penetrate between "portal units" to anastomose with liver cell columns at the periphery of these units. Hyperplasia of the liver may occur with butter yellow administration in the absence of cirrhosis or other change associated with widespread destruction of liver tissue; it occurs as diffuse or focal lesions and affects both parenchymal cells and newly formed ducts. Focal hyperplasia is a precursor of trabecular and adenomatous hepatomas which arise as multiple tumors. Butter yellow causes localized cystic dilatation of newly formed bile ducts unaccompanied by acute or chronic inflammation and liver cells adjacent to them may undergo hyperplasia. Cystic ducts by proliferation of their epithelium may form cyst-adenomas. Butter yellow causes newly formed ducts to undergo dilatation, and with acute inflammation and fibrosis to produce circumscribed macroscopically recognizable lesions (cholangiofibrosis). Cholangiofibrosis is the precursor of tumors that reproduce the histological characters of small bile ducts (cholangiomas), and often proceed to the formation of scirrhous carcinoma. Hepatomas, cyst-adenomas, and cholangiomas may become malignant with formation of metastases in which the characteristics of the parent tumor are reproduced.


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