Cervical exploration for suspected primary hyperparathyroidism in renal stone formers: A challenge to the surgeon

1988 ◽  
Vol 12 (4) ◽  
pp. 534-536
Author(s):  
Lars -Ove Farnebo ◽  
Gunnar Sandersjöö ◽  
Per -Ola Granberg
1988 ◽  
Vol 62 (1) ◽  
pp. 4-6 ◽  
Author(s):  
M. FUSS ◽  
T. PEPERSACK ◽  
J. CORVILAIN ◽  
J. C. VANDEWALLE ◽  
J. GEERTRUYDEN ◽  
...  

2016 ◽  
Vol 195 (4S) ◽  
Author(s):  
Leslee Matheny ◽  
Tracy Marien ◽  
Mustafa Kadihasanoglu ◽  
Nicole L. Miller

Urolithiasis ◽  
1981 ◽  
pp. 89-92 ◽  
Author(s):  
S. Ljunghall ◽  
B. G. Danielson ◽  
G. Johansson ◽  
L. Wibell

1985 ◽  
Vol 29 (5) ◽  
pp. 289-296 ◽  
Author(s):  
Marielle Gascon-Barré ◽  
Pierre D’Amour ◽  
Louis Dufresne ◽  
Jean-Paul Perreault

1994 ◽  
Vol 86 (3) ◽  
pp. 239-243 ◽  
Author(s):  
Bruno Baggio ◽  
Giovanni Gambaro ◽  
Francesco Marchini ◽  
Massimo Vincenti ◽  
Giulio Ceolotto ◽  
...  

1. Anomalous transmembrane anion transport has been observed in erythrocytes of patients with idiopathic calcium nephrolithiasis. 2. To verify whether cation transport is also abnormal, we investigated the frusemide-sensitive Na+ efflux from Na+-loaded erythrocytes and the natriuretic response to acute intravenous frusemide administration in calcium oxalate renal stone formers. 3. Frusemide administration induced a statistically significant smaller increase in the fractional excretion of Na+ in patients than in control subjects. Abnormal kinetic properties of erythrocyte Na+-K+-2Cl− co-transport were observed in approximately 60% of stone formers. The Km for Na+ of Na+-K+-2Cl− co-transport correlated with urinary Ca2+ excretion. 4. The abnormal kinetic properties of Na+-K+-2Cl− co-transport may be relevant for stone formation, hampering renal Ca2+ reabsorption in the distal nephron and determining critical physicochemical conditions for calcium/oxalate crystallization.


2011 ◽  
pp. P1-244-P1-244
Author(s):  
Albert Liu Chu ◽  
Bien J Matawaran ◽  
David Bolong ◽  
Leilani B Mercado-Asis

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