Quercetin enhances water transport in toad bladder

1981 ◽  
Vol 37 (7) ◽  
pp. 742-744 ◽  
Author(s):  
A. Grosso ◽  
R. C. de Sousa
Keyword(s):  
Water Transport ◽  
Toad Bladder

scholarly journals EFFECTS OF FRUSEMIDE ON SODIUM AND WATER TRANSPORT BY THE ISOLATED TOAD BLADDER

1966 ◽  
Vol 27 (3) ◽  
pp. 528-531 ◽  
Author(s):  
D. R. FERGUSON
Keyword(s):  
Water Transport ◽  
Toad Bladder

Water transport response of toad bladder to prolactin

1975 ◽  
Vol 52 (1) ◽  
pp. 75-76 ◽  
Author(s):  
E.S. Debnam ◽  
R.S. Snart
Keyword(s):  
Water Transport ◽  
Toad Bladder ◽  
Transport Response

Dobutamine inhibits vasopressin-mediated water transport across toad bladder epithelium

2008 ◽  
Vol 148 (1) ◽  
pp. 68-71 ◽  
Author(s):  
Wilson Franco ◽  
Renner de Souza Leite
Keyword(s):  
Water Transport ◽  
Toad Bladder ◽  
Bladder Epithelium

THE EFFECT OF CHLORPROPAMIDE AND RELATED COMPOUNDS ON WATER TRANSPORT ACROSS THE TOAD BLADDER

1971 ◽  
Vol 49 (3) ◽  
pp. 551-552 ◽  
Author(s):  
J. K. WALES
Keyword(s):  
Water Transport ◽  
Toad Bladder ◽  
Related Compounds

Effect of vanadate on water transport by the toad bladder

Life Sciences ◽  
1983 ◽  
Vol 32 (16) ◽  
pp. 1879-1884
Author(s):  
Jose A.L. Arruda ◽  
Christof Westenfelder
Keyword(s):  
Water Transport ◽  
Toad Bladder

Glycine-amide effect on the vasopressin stimulated sodium and water transport across toad bladder

1971 ◽  
Vol 39 (2) ◽  
pp. 203-208 ◽  
Author(s):  
D.W. Wright ◽  
R.S. Snart
Keyword(s):  
Water Transport ◽  
Toad Bladder

Cholinergic modulation of water transport in the toad bladder

1980 ◽  
Vol 239 (2) ◽  
pp. F154-F159
Author(s):  
J. A. Arruda ◽  
S. Sabatini
Keyword(s):  
Cyclic Amp ◽  
Muscarinic Receptor ◽  
Water Transport ◽  
Water Flow ◽  
Calcium Uptake ◽  
Toad Bladder ◽  
Base Line ◽  
Cholinergic Modulation ◽  
45Ca Uptake ◽  
A 23187

The effect of carbachol on water transport by the toad bladder was studied. Carbachol caused a small increase in base-line water flow and inhibited, partially, vasopressin- (AVP) or cyclic AMP-stimulated water flow. The effect of carbachol on base-line or AVP-stimulated water flow was totally prevented by atropine, indicating that the effect of cabachol on water transport is mediated through a muscarinic receptor. Carbachol caused a significant increase in 45Ca uptake by toad bladder; this increase in calcium uptake could be prevented by atropine, pentobarbital, or lanthanum. The effect of carbachol on base-line and AVP-stimulated water flow was also prevented by pentobarbital or lanthanum, suggesting that the effect of carbachol is mediated, at least in part, by an increase in calcium uptake. The ionophore A-23187, an agent that increased 45Ca uptake, also enhanced base-line water flow and inhibited AVP-stimulated water flow. The effects of carbachol and the ionophore A-23187 on base-line water flow, AVP-stimulated water flow, and on calcium uptake were not additive, suggesting that both agents alter water transport by a similar mechanism. These data demonstrate that carbachol stimulates base-line water transport and inhibits AVP-stimulated water transport. They suggest that the alteration in water transport induced by carbachol is related to an increase in calcium uptake.

Simultaneous measurement of the effect of vasopressin on sodium and water transport across toad bladder

Life Sciences ◽  
1971 ◽  
Vol 10 (6) ◽  
pp. 301-308 ◽  
Author(s):  
David W. Wright ◽  
Ronald S. Snart
Keyword(s):  
Water Transport ◽  
Simultaneous Measurement ◽  
Toad Bladder

Effects of prostacyclin on short-circuit current and water flow in the toad urinary bladder

1983 ◽  
Vol 244 (3) ◽  
pp. F270-F277
Author(s):  
T. Pohlman ◽  
J. Yates ◽  
P. Needleman ◽  
S. Klahr
Keyword(s):  
Urinary Bladder ◽  
Water Transport ◽  
Water Flow ◽  
Short Circuit ◽  
Short Circuit Current ◽  
Toad Bladder ◽  
Camp Production ◽  
Acid Product ◽  
Osmotic Water ◽  
Osmotic Water Flow

The effects of prostaglandins of the E series on sodium and water transport have been studied extensively. PGE2 has been shown to inhibit the increase in osmotic water flow produced by vasopressin and to stimulate short-circuit current (SCC) in the toad bladder. On the other hand, the effects of prostacyclin (PGI2), an arachidonic acid product, on sodium and water transport have not been extensively evaluated. The present studies describe the effects of PGI2 on basal and vasopressin-stimulated osmotic water flow and on SCC in the urinary bladder of the toad. Studies were performed in the absence or presence of indomethacin. PGI2 in the absence of indomethacin had no effect on basal or vasopressin-stimulated osmotic water flow. When indomethacin was present, thereby eliminating intrinsic prostaglandin biosynthesis, PGI2 inhibited basal but not vasopressin-stimulated osmotic water flow. PGI2 increased SCC in the presence or absence of indomethacin. 6-keto PGF1 alpha, the stable metabolite of PGI2, had no effect on SCC. PGI2 stimulated cAMP production in isolated toad bladder epithelial cells. 2',5'-Dideoxyadenosine, an inhibitor of cAMP production, blocked the increase in SCC produced by PGI2, suggesting that the effects of this compound on SCC are mediated via cAMP.

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