Mechanism of recovery from acute virus infection II. Effect of treatment of mice with cyclosporin A on their ability to eliminate the lymphocytic choriomeningitis virus

1985 ◽  
Vol 174 (4) ◽  
pp. 187-196 ◽  
Author(s):  
C. Löliger ◽  
F. Lehmann-Grube
Keyword(s):  
Virus Infection ◽  
Cyclosporin A ◽  
Lymphocytic Choriomeningitis ◽  
Lymphocytic Choriomeningitis Virus ◽  
Effect Of Treatment

scholarly journals Lymphocytic Choriomeningitis Virus Infection in FVB Mouse Produces Hemorrhagic Disease

2012 ◽  
Vol 8 (12) ◽  
pp. e1003073 ◽  
Author(s):  
Frederick J. Schnell ◽  
Sarah Sundholm ◽  
Stacy Crumley ◽  
Patrick L. Iversen ◽  
Dan V. Mourich
Keyword(s):  
Virus Infection ◽  
Lymphocytic Choriomeningitis ◽  
Lymphocytic Choriomeningitis Virus ◽  
Hemorrhagic Disease ◽  
Lymphocytic Choriomeningitis Virus Infection

Behavioral effects of persistent lymphocytic choriomeningitis virus infection in mice

1994 ◽  
Vol 62 (2) ◽  
pp. 100-109 ◽  
Author(s):  
Lisa H. Gold ◽  
Michelle D. Brot ◽  
Ilham Polis ◽  
Richard Schroeder ◽  
Antoinette Tishon ◽  
...  
Keyword(s):  
Virus Infection ◽  
Lymphocytic Choriomeningitis ◽  
Lymphocytic Choriomeningitis Virus ◽  
Behavioral Effects ◽  
Lymphocytic Choriomeningitis Virus Infection

scholarly journals IMMUNOPATHOLOGY OF LYMPHOCYTIC CHORIOMENINGITIS VIRUS INFECTION OF NEWBORN MICE

1968 ◽  
Vol 127 (4) ◽  
pp. 757-766 ◽  
Author(s):  
Martin S. Hirsch ◽  
Frederick A. Murphy ◽  
Martin D. Hicklin
Keyword(s):  
Virus Infection ◽  
Antibody Formation ◽  
Lymphocytic Choriomeningitis ◽  
Lymphocytic Choriomeningitis Virus ◽  
Reticular Cell ◽  
Wasting Disease ◽  
Cell Hyperplasia ◽  
Newborn Mice ◽  
Lymphocytic Choriomeningitis Virus Infection ◽  
Cellular Tolerance

Antithymocyte serum, when administered neonatally to mice, delayed the maturation of the lymphoid system, permitting development of cellular tolerance to LCM virus at an older age than is ordinarily possible. Humoral antibody formation was not prevented and the animals exhibited the paradox of high titers of both circulating virus and antibody. This, in turn, was followed by a chronic immunopathologic glomerulonephritis in most animals. Some animals developed wasting disease between 1 and 2 months of age, characterized by reticular cell hyperplasia and widespread infiltration into tissues and organs.

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