Open versus minimally invasive small bowel resection for Crohn’s disease: a NSQIP retrospective review and analysis

Author(s):  
Michael L. Horsey ◽  
Debra Lai ◽  
Aalap Herur-Raman ◽  
Richard Amdur ◽  
Matthew Chandler ◽  
...  
1984 ◽  
Vol 19 (5) ◽  
pp. 691-696 ◽  
Author(s):  
I. Hessov ◽  
L. Mosekilde ◽  
F. Melsen ◽  
S. Fasth ◽  
L. Hultén ◽  
...  

2009 ◽  
Vol 69 (5) ◽  
pp. AB199-AB200
Author(s):  
Osamu Watanabe ◽  
Takafumi Ando ◽  
Kazuhiro Ishiguro ◽  
Nobuyuki Miyake ◽  
Motofusa Hasegawa ◽  
...  

2009 ◽  
Vol 207 (1-6) ◽  
pp. 119-122 ◽  
Author(s):  
Andreas Aly ◽  
Franz Bárány ◽  
Bo Kollberg ◽  
Ulla Monsén ◽  
Olof Wisén ◽  
...  

1996 ◽  
Vol 10 (5) ◽  
pp. 317-321
Author(s):  
Neena S Abraham ◽  
Richard N Fedorak

Osteoporosis has long been a recognized complication of Crohn’s disease (CD), with a documented incidence ranging from 31% to 65%. The cause of osteoporosis in Crohn’s patients is likely multifactorial; corticosteroids, inflammatory cytokines, small bowel resection and the resultant calcium and vitamin D deficiencies, hypogonadism, malnutrition and the cachexia of inflammation all play a role. However, the mechanism responsible for osteoporosis associated with CD remains unclear. Treatment of decreased bone density in CD patients has been limited to calcium and vitamin D replacement. The present understanding of the pathophysiology, mechanism and treatment of osteoporosis in CD is reviewed, with the focus on the role of steroid-induced osteoporosis and the use of bisphosphonates.


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