Application of positive end-expiratory pressure in a case with large laceration on the superior vena cava

2010 ◽  
Vol 24 (2) ◽  
pp. 253-255 ◽  
Author(s):  
Chia Sheng Chao ◽  
Chia Ting Chao ◽  
Hsien Kuo Chin ◽  
Yee Phoung Chang
Keyword(s):  
Superior Vena Cava ◽  
Superior Vena ◽  
Vena Cava ◽  
Positive End Expiratory Pressure

Mechanism by which positive end-expiratory pressure increases cerebrospinal fluid pressure in dogs

1982 ◽  
Vol 52 (1) ◽  
pp. 231-235 ◽  
Author(s):  
J. M. Luce ◽  
J. S. Huseby ◽  
W. Kirk ◽  
J. Butler
Keyword(s):  
Cerebrospinal Fluid ◽  
Superior Vena Cava ◽  
Subarachnoid Space ◽  
Superior Vena ◽  
Fluid Pressure ◽  
Vena Cava ◽  
Cerebrospinal Fluid Pressure ◽  
Positive End Expiratory Pressure ◽  
Arterial Blood ◽  
Before And After

We investigated possible mechanisms by which positive end-expiratory pressure (PEEP) increased cerebrospinal fluid pressure (PCSF) in anesthetized mechanically ventilated dogs. In part I of the study, PEEP was applied in 5 cmH2O increments each lasting 1–2 min, before and after a snare separated the spinal from the cerebral subarachnoid space in each animal. Next, with the spinal cord still ligated, the dogs were ventilated without PEEP while superior vena cava pressure (PSVC) was raised in 5 cmH2O increments by means of a fluid reservoir connected with the superior vena cava. Cerebrospinal fluid pressure in the cisterna magna increased immediately and in parallel with PEEP before and after the spinal subarachnoid space was occluded and also increased when PSVC was raised independently; in all circumstances the increase in PCSF correlated closely with PSVC (r = 0.926). In part II of the study, arterial blood gases were drawn before and after PEEP was applied in the same increments and for the same duration as in part I. Cerebrospinal fluid pressure measured with a hollow skull screw again rose in parallel with PEEP, whereas arterial carbon dioxide tension rose only slightly at 60 s. In part III of the study, mean arterial pressure (Pa) was allowed to decrease with PEEP or was held constant by distal aortic obstruction and volume infusion. Cerebrospinal fluid pressure increased regardless of Pa, but the increase was greater when Pa was held constant than when it fell with PEEP. We conclude that PEEP increases PCSF primarily by increasing PSVC and decreasing cerebral venous outflow. This effect is augmented if cerebral arterial inflow is increased as well.

Bleeding Varices in the Upper Esophagus Due to Obstruction of the Superior Vena Cava

1961 ◽  
Vol 41 (5) ◽  
pp. 505-508 ◽  
Author(s):  
Richard W. Snodgrass ◽  
Sherman M. Mellinkoff
Keyword(s):  
Superior Vena Cava ◽  
Superior Vena ◽  
Vena Cava

Significance of collateral vessels on the prediction of superior vena cava syndrome on CT

1993 ◽  
Vol 29 (4) ◽  
pp. 704
Author(s):  
Hyun Sook Kim ◽  
Hyung Jin Kim ◽  
Hyeng Gon Lee ◽  
In Oak Ahn ◽  
Sung Hoon Chung
Keyword(s):  
Superior Vena Cava ◽  
Superior Vena Cava Syndrome ◽  
Superior Vena ◽  
Vena Cava ◽  
Vena Cava Syndrome ◽  
Collateral Vessels

Right to Left Shunt as a Collateral Circulation in a Patient with Superior Vena Cava Syndrome: A Case Report

1999 ◽  
Vol 41 (1) ◽  
pp. 79 ◽  
Author(s):  
Young Sun Kim ◽  
Seok Chol Jeon ◽  
Won Jin Moon ◽  
Yo Won Choi ◽  
Heung Suk Seo ◽  
...  
Keyword(s):  
Case Report ◽  
Superior Vena Cava ◽  
Collateral Circulation ◽  
Superior Vena Cava Syndrome ◽  
Superior Vena ◽  
Vena Cava ◽  
Vena Cava Syndrome

Single-Patch Fold-Back Technique for Augmentation of the Superior Vena Cava in Sinus Venosus Atrial Septal Defect Associated with Partial Anomalous Pulmonary Venous Return

2015 ◽  
Vol 17 (6) ◽  
pp. 282
Author(s):  
Suguru Ohira ◽  
Kiyoshi Doi ◽  
Takeshi Nakamura ◽  
Hitoshi Yaku
Keyword(s):  
Atrial Septal Defect ◽  
Superior Vena Cava ◽  
Septal Defect ◽  
Venous Return ◽  
Superior Vena ◽  
Pulmonary Veins ◽  
Vena Cava ◽  
Sinus Venosus ◽  
Right Atrial ◽  
Anomalous Pulmonary Venous Return

Sinus venosus atrial septal defect (ASD) is usually associated with partial anomalous pulmonary venous return (PAPVR) of the right pulmonary veins to the superior vena cava (SVC), or to the SVC-right atrial junction. Standard procedure for repair of this defect is a patch roofing of the sinus venosus ASD and rerouting of pulmonary veins. However, the presence of SVC stenosis is a complication of this technique, and SVC augmentation is necessary in some cases. We present a simple technique for concomitant closure of sinus venosus ASD associated with PAPVR and augmentation of the SVC with a single autologous pericardial patch.

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