scholarly journals Mechanism of Acid Hypersecretion Post Curative Gastrinoma Resection

2010 ◽  
Vol 56 (1) ◽  
pp. 139-154 ◽  
Author(s):  
Jeremiah V. Ojeaburu ◽  
Tetsuhide Ito ◽  
Pellegrino Crafa ◽  
Cesare Bordi ◽  
Robert T. Jensen
Keyword(s):  
Acid Hypersecretion

Rebound acid hypersecretion after ranitidine is not due to impaired inhibitory control

2001 ◽  
Vol 120 (5) ◽  
pp. A158-A158 ◽  
Author(s):  
D GILLEN ◽  
A WIRZ ◽  
K MCCOLL
Keyword(s):  
Inhibitory Control ◽  
Acid Hypersecretion

Parenteral control of gastric acid hypersecretion in patients with Zollinger-Ellison syndrome

1993 ◽  
Vol 38 (10) ◽  
pp. 1857-1865 ◽  
Author(s):  
Rakesh Vinayek ◽  
William F. Hahne ◽  
Arthur R. Euler ◽  
Jeffrey A. Norton ◽  
Robert T. Jensen
Keyword(s):  
Gastric Acid ◽  
Zollinger Ellison Syndrome ◽  
Gastric Acid Hypersecretion ◽  
Acid Hypersecretion

scholarly journals Surgery for Gastrinoma and Insulinoma in Multiple Endocrine Neoplasia Type 1

2006 ◽  
Vol 4 (2) ◽  
pp. 148-153 ◽  
Author(s):  
Jeffrey A. Norton ◽  
Tony D. Fang ◽  
Robert T. Jensen
Keyword(s):  
Gastric Acid ◽  
Multiple Endocrine Neoplasia ◽  
Multiple Endocrine Neoplasia Type ◽  
Endocrine Tumors ◽  
Endocrine Neoplasia ◽  
Endocrine Neoplasia Type ◽  
Pancreatic Endocrine Tumors ◽  
Gastric Acid Hypersecretion ◽  
Acid Hypersecretion

The surgical management of pancreatic endocrine tumors in patients with multiple endocrine neoplasia type 1 remains controversial. Gastrinoma and insulinoma are the 2 most common functional pancreatic neuroendocrine tumors in patients with multiple endocrine neoplasia type 1. Gastrinomas cause gastric acid hypersecretion and peptic ulcer disease that are best managed using proton pump inhibitors. Surgery to remove the gastrinoma in patients with multiple endocrine neoplasia type 1 is seldom curative unless a more extensive Whipple pancreaticoduodenectomy is performed. Because the prognosis is excellent, aggressive resections such as a Whipple procedure are only indicated for large, locally metastatic, advanced tumors. Furthermore, surgery to remove imageable tumors that are 2 cm in diameter is associated with excellent outcomes and decreased probability of liver metastases. Because gastrinomas are commonly multiple and most originate in the duodenum and develop lymph node metastases, the duodenum should be opened and all tumors and lymph nodes excised. Insulinomas cause hypoglycemia that results in neuroglycopenic symptoms. Medical management of the hypoglycemia is less effective than that of the gastric acid hypersecretion. Fortunately, the insulinoma is usually clearly identified using routine pancreatic imaging studies. There is a high likelihood of cure when the insulinoma is excised surgically. However, recurrent hypoglycemia may occur, and careful follow-up is indicated.

scholarly journals Nutritive support in short Bowel syndrome (sbs)

2003 ◽  
Vol 131 (1-2) ◽  
pp. 77-81 ◽  
Author(s):  
Dusica Simic ◽  
Nebojsa Djurisic
Keyword(s):  
Short Bowel Syndrome ◽  
Age Of Onset ◽  
Intestinal Atresia ◽  
Nutritional Deficiencies ◽  
Current Therapy ◽  
Normal Body Mass Index ◽  
Short Bowel ◽  
Gastric Acid Hypersecretion ◽  
Acid Hypersecretion

Short bowel syndrome most commonly result after bowel resection for necrosis of the bowel. It may be caused by arterial or venous thrombosis, volvolus and in children, necrotizing enterocolitis. The other causes are Crohn,s disease intestinal atresia. The factors influencing the risk on short bowel syndrome are the remaining length of the small bowel, the age of onset, the length of the colon, the presence or absence of the ileo-coecal valve and the time after resection. Besides nutritional deficiencies there some other consequences of extensive resections of the small intestine (gastric acid hypersecretion, d-lactic acidosis, nephrolithiasis, cholelithiasis), which must be diagnosed, treated, and if possible, prevented. With current therapy most patients with short bowel have normal body mass index and good quality of life.

scholarly journals Impairment of CFTR activity in cultured epithelial cells upregulates the expression and activity of LDH resulting in lactic acid hypersecretion

2019 ◽  
Vol 76 (8) ◽  
pp. 1579-1593 ◽  
Author(s):  
Ángel G. Valdivieso ◽  
Mariángeles Clauzure ◽  
María M. Massip-Copiz ◽  
Carla E. Cancio ◽  
Cristian J. A. Asensio ◽  
...  
Keyword(s):  
Lactic Acid ◽  
Epithelial Cells ◽  
Cultured Epithelial Cells ◽  
Acid Hypersecretion ◽  
Expression And Activity

scholarly journals Control of gastric acid hypersecretion in the management of patients with Zollinger-Ellison syndrome

1993 ◽  
Vol 17 (4) ◽  
pp. 468-480 ◽  
Author(s):  
David C. Metz ◽  
Joseph R. Pisegna ◽  
Vitaly A. Fishbeyn ◽  
Richard V. Benya ◽  
Robert T. Jensen
Keyword(s):  
Gastric Acid ◽  
Zollinger Ellison Syndrome ◽  
Gastric Acid Hypersecretion ◽  
Acid Hypersecretion

scholarly journals Rebound Acid Hypersecretion

2004 ◽  
Vol 94 (5) ◽  
pp. 201-201
Author(s):  
Kenneth E. L. McColl
Keyword(s):  
Acid Hypersecretion
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