Can we implant left ventricle pacing lead in a patient with coronary sinus reducer?

2017 ◽  
Vol 51 (1) ◽  
pp. 87-88 ◽  
Author(s):  
Luca Bontempi ◽  
Francesca Vassanelli ◽  
Manuel Cerini ◽  
Lorenza Inama ◽  
Francesca Salghetti ◽  
...  
EP Europace ◽  
2001 ◽  
Vol 3 (4) ◽  
pp. 317-323 ◽  
Author(s):  
S. Sack ◽  
F. Heinzel ◽  
N. Dagres ◽  
S. Enger ◽  
A. Auricchio ◽  
...  

Abstract Aims This report describes the initial clinical results with a newly designed guiding catheter and an ‘over the wire’ pacing lead based on angiolasty technology to stimulate the left ventricle using the transvenous route via the coronary sinus (OTW-CV lead). Methods and Results In 75% of the 15 patients (6 males, 9 females, mean age of 53±9 years) with congestive heart failure, access to coronary sinus required less than 2 min; in one patient, the attempt failed. Mean R wave amplitudes plus or minus the standard deviation, measured at apical, mid-ventricular and basal positions in the anterior (11·4±9·2, 10·8±6·2, 9·3±6·3 mV) and lateral or posterior veins (10·1±10·7, 8·6±6·4, 7·7±4·3 mV) showed a trend favouring the apex without statistical significance. Pacing impedance, measured at the same sites and vein tributaries, ranged from 670±191 to 915±145 ohms. Pacing thresholds measured at apical and mid ventricular sites were significantly lower than at the base in the anterior vein 2·5±2·8 and 2·8±1·8 vs 5·6±2·7 V at 0·5 ms, P< 0·001). Thresholds in the lateral/posterior veins showed a similar trend but did not reach statistical significance (3·0±1·7, 3·6±1·4±1·8 V at 0·5 ms). In patients, in whom thresholds were determined in more than one vein, the ‘best’ mean threshold was 1·6±0·7 V. Conclusion The new ‘over the wire’ lead and guiding catheter system allows uncomplicated access to the coronary sinus and the depth of the coronary vein tributaries. Left ventricular sensing and pacing thresholds are acceptable for chronic use in implanted cardiac rhythm management systems.


Heart ◽  
1981 ◽  
Vol 45 (1) ◽  
pp. 101-104 ◽  
Author(s):  
K M McGarry ◽  
J Stark ◽  
F J Macartney

2014 ◽  
Vol 86 (2) ◽  
pp. E99-E102
Author(s):  
Zachary M. Gertz ◽  
Jose-Luis E. Velazquez-Cecena ◽  
John. V. Ian Nixon

EP Europace ◽  
2017 ◽  
Vol 19 (suppl_3) ◽  
pp. iii211-iii212
Author(s):  
B. Papelbaum ◽  
SS. Galvao Filho ◽  
JT. Medeiros De Vasconcelos ◽  
C. Eduardo Duarte ◽  
R. Castro Galvao ◽  
...  

2012 ◽  
Vol 28 (5) ◽  
pp. 612.e9-612.e10
Author(s):  
Kiyotaka Watanabe ◽  
Kozo Hoshino ◽  
Kaoru Dohi ◽  
Naritatsu Saito ◽  
Takafumi Hashimoto ◽  
...  

1965 ◽  
Vol 208 (5) ◽  
pp. 946-953 ◽  
Author(s):  
Skoda Afonso ◽  
George G. Rowe ◽  
Jorge E. Lugo ◽  
Charles W. Crumpton

Only a part of heat produced by the left ventricle is removed by the coronary blood. During a cold saline infusion into the right ventricle, LV myocardial temperature decreases and the myocardium loses a measurable amount of heat. A part of this heat is also removed by the coronary blood. If simultaneous thermal curves are recorded from the aorta and coronary sinus during the infusion it is possible to calculate left ventricle heat production by the following formula: H = LV weight x ΔT x Δt x K x 60:A, where ΔT = myocardial temperature drop during the infusion; Δt = coronary sinus-aorta blood temperature difference prior to infusion; K = specific heat of myocardium; A = difference of areas of superimposed coronary sinus and aorta's thermal curves. Heat production estimated by the formula in 19 determinations has been compared with the heat production calculated from myocardial oxygen consumption. Measurements obtained by this method seem to be representative of left ventricle heat production.


EP Europace ◽  
2011 ◽  
Vol 13 (8) ◽  
pp. 1207-1208 ◽  
Author(s):  
J. N. Irvine ◽  
D. J. LaPar ◽  
S. Mahapatra ◽  
J. P. DiMarco ◽  
G. Ailawadi

2019 ◽  
Vol 35 (5) ◽  
pp. 748-751
Author(s):  
Sou Takenaka ◽  
Jun Suzuki ◽  
Akihiko Ueno ◽  
Takashi Uchiyama

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