Role of reactive oxygen species and effect of solution matrix in trichloroethylene degradation from aqueous solution by zeolite-supported nano iron as percarbonate activator

2016 ◽  
Vol 42 (9) ◽  
pp. 6959-6973 ◽  
Author(s):  
Muhammad Danish ◽  
Xiaogang Gu ◽  
Shuguang Lu ◽  
Minhui Xu ◽  
Xiang Zhang ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Aqueous Solution ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Trichloroethylene Degradation ◽  
Solution Matrix

Role of in situ electrogenerated reactive oxygen species towards degradation of organic dye in aqueous solution

2020 ◽  
Vol 344 ◽  
pp. 136146 ◽  
Author(s):  
Md. Saddam Hossain ◽  
M. Yousuf A. Mollah ◽  
Md. Abu Bin Hasan Susan ◽  
Md. Mominul Islam
Keyword(s):  
Reactive Oxygen Species ◽  
Aqueous Solution ◽  
Reactive Oxygen ◽  
Organic Dye ◽  
Oxygen Species

Photolysis of atrazine in aqueous solution: role of process variables and reactive oxygen species

2014 ◽  
Vol 21 (21) ◽  
pp. 12135-12142 ◽  
Author(s):  
Marcela Prado Silva ◽  
Ana Paula dos Santos Batista ◽  
Sueli Ivone Borrely ◽  
Vanessa Honda Ogihara Silva ◽  
Antonio Carlos Silva Costa Teixeira
Keyword(s):  
Reactive Oxygen Species ◽  
Aqueous Solution ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Process Variables

scholarly journals Effect of solution matrix and pH in Z-nZVI-catalyzed percarbonate system on the generation of reactive oxygen species and degradation of 1,1,1-trichloroethane

2017 ◽  
Vol 17 (6) ◽  
pp. 1568-1578 ◽  
Author(s):  
Muhammad Danish ◽  
Xiaogang Gu ◽  
Shuguang Lu ◽  
Usman Farooq ◽  
Ayyaz Ahmad ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Hydroxyl Radicals ◽  
Reactive Oxygen ◽  
Reducing Agent ◽  
Oxygen Species ◽  
Sodium Percarbonate ◽  
Ph Values ◽  
Solution Matrix

Abstract This study primarily focuses on evaluating the effects of solution matrix and pH for the generation of reactive oxygen species (ROSs) in a Z-nZVI-catalyzed sodium percarbonate (SPC) system to degrade 1,1,1-trichloroethane (1,1,1-TCA) in the absence and presence of a reducing agent (RA), i.e. hydroxylamine. Degradation of 1,1,1-TCA was 49.5% and 95% in the absence and presence of RA. Probe tests confirmed the generation of major hydroxyl radicals (OH•) and minor superoxide species (O2–•), and scavenger tests verified the key role of OH• and less of (O2–•) radicals. Degradation of 1,1,1-TCA decreased significantly in the presence of Cl− and HCO3–, while NO3– and SO42– had negligible effects in the absence of RA. Addition of RA significantly enhanced 1,1,1-TCA degradation by generating more OH• and (O2–•) radicals in the presence of anions. Degradation of 1,1,1-TCA increased in the acidic range (1–5), while an inhibitive trend from neutral to basic (7–9) was observed. In contrast, a significant increase in 1,1,1-TCA degradation was observed with the addition of RA at all pH values (1–9). In conclusion, the anions and pH significantly influenced the generation and intensity of ROSs and 1,1,1-TCA was effectively degraded in the Z-nZVI-catalyzed SPC system in the presence of RA.

The Role of Reactive Oxygen Species in Tumor Treatment and its Impact on Bone Marrow Hematopoiesis

2020 ◽  
Vol 21 (5) ◽  
pp. 477-498
Author(s):  
Yongfeng Chen ◽  
Xingjing Luo ◽  
Zhenyou Zou ◽  
Yong Liang
Keyword(s):  
Reactive Oxygen Species ◽  
Bone Marrow ◽  
Oxidative Damage ◽  
Tumor Cells ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Tumor Treatment ◽  
Normal Cells ◽  
Treatment And Prevention

Reactive oxygen species (ROS), an important molecule inducing oxidative stress in organisms, play a key role in tumorigenesis, tumor progression and recurrence. Recent findings on ROS have shown that ROS can be used to treat cancer as they accelerate the death of tumor cells. At present, pro-oxidant drugs that are intended to increase ROS levels of the tumor cells have been widely used in the clinic. However, ROS are a double-edged sword in the treatment of tumors. High levels of ROS induce not only the death of tumor cells but also oxidative damage to normal cells, especially bone marrow hemopoietic cells, which leads to bone marrow suppression and (or) other side effects, weak efficacy of tumor treatment and even threatening patients’ life. How to enhance the killing effect of ROS on tumor cells while avoiding oxidative damage to the normal cells has become an urgent issue. This study is a review of the latest progress in the role of ROS-mediated programmed death in tumor treatment and prevention and treatment of oxidative damage in bone marrow induced by ROS.

The Role of Reactive Oxygen Species, Kinases, Hydrogen Sulfide, and Nitric Oxide in the Regulation of Autophagy and Their Impact on Ischemia and Reperfusion Injury in the Heart

2020 ◽  
Vol 16 ◽  
Author(s):  
Andrey Krylatov ◽  
Leonid Maslov ◽  
Sergey Y. Tsibulnikov ◽  
Nikita Voronkov ◽  
Alla Boshchenko ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Reactive Oxygen Species ◽  
Hydrogen Sulfide ◽  
Ischemia Reperfusion ◽  
Reactive Oxygen ◽  
Ischemia And Reperfusion ◽  
Oxygen Species ◽  
Ischemia And Reperfusion Injury ◽  
Adaptive Process

: There is considerable evidence in the heart that autophagy in cardiomyocytes is activated by hypoxia/reoxygenation (H/R) or in hearts by ischemia/reperfusion (I/R). Depending upon the experimental model and duration of ischemia, increases in autophagy in this setting maybe beneficial (cardioprotective) or deleterious (exacerbate I/R injury). Aside from the conundrum as to whether or not autophagy is an adaptive process, it is clearly regulated by a number of diverse molecules including reactive oxygen species (ROS), various kinases, hydrogen sulfide (H2S) and nitric oxide (NO). The purpose this review is to address briefly the controversy regarding the role of autophagy in this setting and to examine a variety of disparate molecules that are involved in its regulation.

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