scholarly journals How I do it a microsurgical neurovascular decompression for hemifacial spasm

2021 ◽  
Author(s):  
X. Dubernard ◽  
A. Bazin ◽  
J.-C. Kleiber
Keyword(s):  
Hemifacial Spasm ◽  
Neurovascular Decompression

scholarly journals Neurovascular decompression for hemifacial spasm - Transmastoid approach.

1987 ◽  
Vol 80 (2) ◽  
pp. 229-234
Author(s):  
Haruo Saito ◽  
Taizo Takeda ◽  
Kazuyuki Tashima
Keyword(s):  
Hemifacial Spasm ◽  
Neurovascular Decompression ◽  
Transmastoid Approach

Hemifacial spasm caused by vascular compression of the distal portion of the facial nerve

1998 ◽  
Vol 88 (3) ◽  
pp. 605-609 ◽  
Author(s):  
Hiroshi Ryu ◽  
Seiji Yamamoto ◽  
Kenji Sugiyama ◽  
Kenichi Uemura ◽  
Tsunehiko Miyamoto
Keyword(s):  
Facial Nerve ◽  
Hemifacial Spasm ◽  
Cranial Nerve ◽  
Distal Portion ◽  
Vascular Compression ◽  
Neurovascular Compression ◽  
Content Type ◽  
Seventh Cranial Nerve ◽  
Neurovascular Decompression ◽  
Fine Print

✓ It is generally accepted that hemifacial spasm (HFS) and trigeminal neuralgia are caused by compression of the facial nerve (seventh cranial nerve) or the trigeminal nerve (fifth cranial nerve) at the nerve's root exit (or entry) zone (REZ); thus, neurosurgeons generally perform neurovascular decompression at the REZ. Neurosurgeons tend to ignore vascular compression at distal portions of the seventh cranial nerve, even when found incidentally while performing neurovascular decompression at the REZ of that nerve, because compression of distal portions of the seventh cranial nerve has not been regarded as a cause of HFS. Recently the authors treated seven cases of HFS in which compression of the distal portion of the seventh cranial nerve produced symptoms. The anterior inferior cerebellar artery (AICA) was the offending vessel in five of these cases. Great care must be taken not to stretch the internal auditory arteries during manipulation of the AICA because these small arteries are quite vulnerable to surgical manipulation and the patient may experience hearing loss postoperatively. It must be kept in mind that compression of distal portions of the seventh cranial nerve may be responsible for HFS in cases in which neurovascular compression at the REZ is not confirmed intraoperatively and in cases in which neurovascular decompression at the nerve's REZ does not cure HFS. Surgical procedures for decompression of the distal portion of the seventh cranial nerve as well as decompression at the REZ should be performed when a deep vascular groove is noticed at the distal site of compression of the nerve.

Delayed Hearing Loss after Neurovascular Decompression

Neurosurgery ◽  
1990 ◽  
Vol 27 (6) ◽  
pp. 997-1003 ◽  
Author(s):  
Dennis E. McDonnell ◽  
Bahman Jabbari ◽  
Giovana Spinella ◽  
Gustave H. Mueller ◽  
Peter M. Klara
Keyword(s):  
Hearing Loss ◽  
Facial Nerve ◽  
Hemifacial Spasm ◽  
Cerebellopontine Angle ◽  
Unilateral Hearing Loss ◽  
Neurovascular Compression ◽  
Bilateral Hearing Loss ◽  
First Case ◽  
Neurovascular Decompression ◽  
Vascular Decompression

Abstract We report two unusual cases of delayed hearing loss after neurovascular decompression of structures within the cerebellopontine angle. In the first case, the patient noted a unilateral hearing loss 3 weeks after undergoing vascular decompression of the trigeminal nerve for tic douloureux. This gradually improved over an 18-month period. In the second case, the patient awoke on the 4th day after vascular decompression of the facial nerve for hemifacial spasm with a bilateral hearing loss that has remained unchanged after the onset. These are examples of delayed acoustic dysfunction occurring with a shift in surgically freed vessels and may have been induced by newly directed neurovascular compression or distortion.

Neurovascular Decompression for Trigeminal Neuralgia associated with Hemifacial Spasm

1986 ◽  
Vol 26 (11) ◽  
pp. 900-903 ◽  
Author(s):  
Jun MIYAGI ◽  
Yuichiro HANABUSA ◽  
Shinken KURAMOTO ◽  
Masao KUBOYAMA ◽  
Kazunori KAJIWARA
Keyword(s):  
Trigeminal Neuralgia ◽  
Hemifacial Spasm ◽  
Neurovascular Decompression

Efficacy and Complications of Microsurgical Neurovascular Decompression in 55 Patients With Hemifacial Spasm

2020 ◽  
pp. 019459982096916
Author(s):  
Chloé Compagnon ◽  
Marc Labrousse ◽  
Esteban Brenet ◽  
André Chays ◽  
Arnaud Bazin ◽  
...  
Keyword(s):  
Quality Of Life ◽  
Hemifacial Spasm ◽  
Microvascular Decompression ◽  
Unilateral Deafness ◽  
Quality Of Life Scale ◽  
Life Scale ◽  
Life Improvement ◽  
Neurovascular Decompression

Objective To analyze the efficacy and complications of microvascular decompression for hemifacial spasm. Study Design Retrospective study. Setting Regional hospital. Methods Fifty-five patients with hemifacial spasm were treated by microvascular decompression. All patients with hemifacial spasm who underwent retrosigmoid microvascular decompression from May 2004 to January 2017 were included. Patients with no conflict on preoperative magnetic resonance imaging or with an alternate diagnosis were excluded. Results The overall cure rate was 83.64%, with an average follow-up of 7.4 years. A left-sided hemifacial spasm was a healing-promoting factor ( P = .01). The median healing was 0.03 months, and the mean was 6 months. The efficacy remained high in the medium term (88% at 3 years), long term (90.24% at 5 years), and very long term (90.48% at 8 years). The recurrence rate was 9.8%. Favorable criteria included a right-sided spasm ( P = .01) and an average age of 62 years ( P = .03). The specific complications were permanent facial palsy (3.63%), unilateral deafness (5.45%), and hearing loss (3.63%). No death was reported. Regarding the quality of life of the patients, 94.7% had a modified HFS-8 postoperative score of 0 (Hemifacial Spasm 8 Quality of Life Scale). Conclusion Microvascular decompression for hemifacial spasm is an effective and lasting technique. Its low rate of complications and the considerable quality-of-life improvement should lead surgeons to propose it to patients as soon as botulinum toxin injections become ineffective or poorly tolerated.

Neurovascular decompression of the eighth cranial nerve in patients with hemifacial spasm and incidental tinnitus: an alternative way to study tinnitus

1998 ◽  
Vol 88 (2) ◽  
pp. 232-236 ◽  
Author(s):  
Hiroshi Ryu ◽  
Seiji Yamamoto ◽  
Kenji Sugiyama ◽  
Kenichi Uemura ◽  
Michihiko Nozue
Keyword(s):  
Hemifacial Spasm ◽  
Cranial Nerve ◽  
Diagnostic Value ◽  
Sensorineural Hearing ◽  
Neurovascular Compression ◽  
Eighth Cranial Nerve ◽  
Chi Square ◽  
Content Type ◽  
Neurovascular Decompression ◽  
Fine Print

Object. The authors sought to clarify the clinical characteristics of tinnitus resulting from neurovascular compression (NVC) of the eighth cranial nerve. Methods. The authors explored the eighth cranial nerve in the cerebellopontine cistern during neurovascular decompression (NVD) of the facial nerve in 10 patients with hemifacial spasm who suffered from incidental tinnitus on the same side. The diagnosis of NVC of the eighth cranial nerve was confirmed in all patients. This condition was found in only seven of 114 patients with hemifacial spasm alone, indicating that NVC of the eighth cranial nerve is one of the causes of tinnitus (p < 0.001, chi-square test). The tinnitus resolved or was markedly improved after NVD of the eighth cranial nerve in eight patients (80%). Both pulsatile and continuous tinnitus responded well to NVD. All patients experienced various degrees of sensorineural hearing disturbance, but other neurotological examinations provided poor diagnostic value. Conclusions. It is the authors' opinion that sensorineural hearing loss and positive findings on magnetic resonance imaging are the most reliable evidence for the presence of tinnitus caused by NVC of the eighth cranial nerve.

Sign in / Sign up

Export Citation Format

Share Document