Plasma Serotonin in Heart Failure: Possible Marker and Potential Treatment Target

2017 ◽  
Vol 26 (5) ◽  
pp. 442-449 ◽  
Author(s):  
Ahmed M. Selim ◽  
Nitasha Sarswat ◽  
Iosif Kelesidis ◽  
Muhammad Iqbal ◽  
Ramesh Chandra ◽  
...  
2020 ◽  
Vol 2 (9) ◽  
pp. 1614-1627
Author(s):  
Joshua Henrina ◽  
Irvan Cahyadi ◽  
Hoo Felicia Hadi Gunawan ◽  
Leonardo Paskah Suciadi

2022 ◽  
pp. 1-5
Author(s):  
Trine Engelbrecht Hybel ◽  
Maja Ølholm Vase ◽  
Kristina Lystlund Lauridsen ◽  
Marie Beck Enemark ◽  
Michael Boe Møller ◽  
...  

2018 ◽  
Vol 7 (4) ◽  
pp. 199-208
Author(s):  
Caroline Meyer Olesen ◽  
Maja-Lisa Clausen ◽  
Paal Skytt Andersen ◽  
Tove Agner

2021 ◽  
Author(s):  
Enyuan Cao ◽  
Matthew J. Watt ◽  
Cameron J. Nowell ◽  
Tim Quach ◽  
Jamie S. Simpson ◽  
...  

Oral Diseases ◽  
2021 ◽  
Author(s):  
Kae Ishii ◽  
Takahito Tamura ◽  
Keisuke Hatori ◽  
Kazuma Himi ◽  
Takeshi Nakamura ◽  
...  

ESC CardioMed ◽  
2018 ◽  
pp. 710-713
Author(s):  
Frank Weidemann

Genetic disease can lead to myocardial infiltration which results in a cardiomyopathy characterized by the deposition of storage products within ventricular walls. The storage is inducing diastolic filling abnormalities which are finally responsible for the symptoms such as dyspnoea. During follow-up most patients die because of heart failure. Transthyretin-related amyloidosis is the most common infiltrative cardiomyopathy. Rarer diseases are storage disorders such as Fabry disease and Pompe disease. The primary neurological disease Friedreich’s ataxia can in addition induce an infiltrative cardiomyopathy. This chapter focuses on the pathophysiology, the symptoms, the typical image findings, and the potential treatment of these different rare genetic infiltrative diseases.


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