Parkinsonism associated with chronic acquired hepatocerebral degeneration and autoimmune manifestation: Case report and review

2017 ◽  
Vol 381 ◽  
pp. 699
Author(s):  
Y.C. Kuo ◽  
C.C. Yang ◽  
J.J. Su
Author(s):  
Bahaar Athavale ◽  
Jaya Pathak ◽  
Vaishali Patel ◽  
Suvorit Subhas Bhowmick

2016 ◽  
Vol 4 (1) ◽  
pp. 54-57
Author(s):  
Abdul Kader Shaikh ◽  
Mahjabin Islam ◽  
Ahijit Chowdhury ◽  
Ashraf Uddin Ahmed ◽  
Utpal Dasgupta

Acquired Hepatocerebral Degeneration (AHD) produces a panel of neurological symptoms occurring in patients with chronic liver disease. These symptoms may mimic different neurological abnormalities e.g. abnormal movements, rigidity, parkinsonism, ataxia, neuropsychiatric and cognitive manifestations.[1] Acquired Hepatocerebral Degeneration is usually encountered in patients with CLD due to accumulation of manganese & other metabolic toxins in basal ganglia, following repeated episodes of liver failure[2] .The aim of this case report is to highlight the MRI finding of Acute Hepatocerebral Degeneration which often can be underappreciated owing to the possibility of diagnosing a relatively common condition that is Bilateral Ischemic stroke.[19].Bangladesh Crit Care J March 2016; 4 (1): 54-57


2002 ◽  
Vol 6 (3) ◽  
pp. 34-35
Author(s):  
B. J. Sher ◽  
Ian C. Duncan

Acquired hepatocerebral degeneration is a subtype of chronic recurrent hepatic encephalopathy and is characterised by movement disorders and increased signal intensity in the basal ganglia on Tl-weighted MRI images.


2009 ◽  
Vol 15 ◽  
pp. S68
Author(s):  
M. Navarro-Fernández-Hidalgo ◽  
N. Rodríguez-Gómez-Hidalgo ◽  
J. Porta-Etessam ◽  
R. Garcia Ramos ◽  
M.J. Catalán

2012 ◽  
Vol 6 (1) ◽  
pp. 59-63 ◽  
Author(s):  
Clarice Listik ◽  
Gislaine Cristina Lopes Machado-Porto ◽  
Maira Okada de Oliveira ◽  
Fábio Henrique de Gobbi Porto

ABSTRACT Acquired hepatocerebral degeneration is an underdiagnosed neurologic syndrome characterized by parkinsonism, ataxia or other movement disorders and by neuropsychiatric and cognitive symptoms. It occurs in patients with chronic liver disease, especially those who develop portosystemic shunting and is often unrecognized as a cause of cognitive decline. Recently, its pathogenesis has been associated with manganese accumulation in basal ganglia and some treatments proposed. The aim of this article was to report a case and discuss some discoveries in connection with the disease.


2015 ◽  
Author(s):  
Ronald F. Pfeiffer

Contrary to what one may assume, neurology and gastroenterology are similar to each other in many ways. A process found in one system can also affect the other, whether it be gastrointestinal (GI) dysfunction present in neurologic diseases or neurologic dysfunction present in GI diseases. Several disorders from both fields of medicines are highlighted in this review, ranging from stroke, Parkinson disease, and multiple sclerosis (MS) as examples of GI dysfunction in neurologic disease, to celiac disease and inflammatory bowel disease as examples of neurologic dysfunction in GI disease. Rare disorders such as Whipple disease (WhD), chronic acquired hepatocerebral degeneration (CAHD), and Wilson disease (WD) should also be considered for their neurologic dysfunctions as manifested extraintestinally. Tables give an overview of clinical features of the various disorders presented in this review.   This module contains ­4 highly rendered figures, 9 tables, 349 references, and 5 MCQs. 


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