Poster 334: Hashimoto Encephalopathy: Cognitive Advancement: A Case Report

PM&R ◽  
2017 ◽  
Vol 9 ◽  
pp. S238-S238
Author(s):  
Ka Hoi Hui ◽  
Mohammad A. Issa ◽  
Craig DiTommaso
2016 ◽  
Vol 59 ◽  
pp. 62-70 ◽  
Author(s):  
Brett R. Graham ◽  
Natalie Shiff ◽  
Munier Nour ◽  
Simona Hasal ◽  
Richard Huntsman ◽  
...  

2006 ◽  
Vol 37 (01) ◽  
Author(s):  
M Lanz ◽  
S Bunten ◽  
O Grothues ◽  
K Wittig ◽  
HU Voelter ◽  
...  

2014 ◽  
Vol 5 (2) ◽  
pp. 70-73 ◽  
Author(s):  
Sarita K. Sapkota ◽  
Biggya L. Sapkota ◽  
Nataria Pitiyanuvath

2016 ◽  
Vol 129 (9) ◽  
pp. e187
Author(s):  
Ryota Sato ◽  
Tadaaki Takada ◽  
Michitaka Nasu

Author(s):  
Diana Simoniene ◽  
Raimonda Klimaite ◽  
Greta Mikelaityte ◽  
Evalda Danyte

2008 ◽  
Vol 7 (S1) ◽  
Author(s):  
Styliani Spyridi ◽  
Marianthi Arnaoutoglou ◽  
Eugenia Avdelidi ◽  
Eirini Tsirka ◽  
Jannis Nimatoudis ◽  
...  

2018 ◽  
Vol 28 (1) ◽  
pp. 91-95 ◽  
Author(s):  
Alixandria A. Fiore ◽  
Wilson B. Pfeiffer ◽  
Syed A.A. Rizvi ◽  
Anais  Cortes ◽  
Conrad  Ziembinski ◽  
...  

Objective: To present a case of Hashimoto encephalopathy as a complication of autoimmune thyroiditis. Clinical Presentation and Intervention: A previously healthy 56-year-old female presented with rapidly progressive cognitive decline and visual hallucinations. Being a diagnosis of exclusion, Hashimoto encephalopathy required an extensive laboratory and diagnostic workup, which was done over the course of a 15-day hospitalization. The patient recovered after initial treatment with intravenous methylprednisolone and was then switched to prednisone p.o. Conclusion: This case report illustrates the importance of awareness for Hashimoto encephalopathy, as it remains one of the few easily treatable and reversible causes of rapid cognitive decline.


2014 ◽  
Vol 8 (2) ◽  
pp. 515-518 ◽  
Author(s):  
XU HUANG ◽  
YANG YU ◽  
HUA ZHANG ◽  
JIE LIU ◽  
YUQIAN SUN ◽  
...  

2007 ◽  
Vol 118 (4) ◽  
pp. e66-e67
Author(s):  
M. Lanz ◽  
S. Bunten ◽  
O. Grothues ◽  
K. Wittig ◽  
H.U. Voelter ◽  
...  

2021 ◽  
Vol 46 (3) ◽  
pp. 137-140
Author(s):  
Vesna Martić ◽  
Aleksandar Stojanov ◽  
Tihomir Ilić

Introduction: Hashimoto encephalopathy (HE) is a rare and often misdiagnosed entity. Except high levels of the thyroid peroxidase (anti-TPO) and antithyroglobulin (anti-TG) antibodies, neurophysiological and psychological tests are beneficial for the diagnosis. The presence of oligoclonal bands in the cerebrospinal fluid (CSF) of these patients is very rare. We present a patient with HE and oligoclonal bands in CSF with good clinical response on corticosteroid therapy. Case report: Male patient, 39 years old, suddenly developed focal neurologic deficit. He had elevated anti-TPO and anti TG antibodies, impaired concentration on psychologist report and oligoclonal bands in CSF. Slowing of electroencephalography activity was normalized with full clinical recovery of the patients, after corticosteroid therapy. The patient is in clinical remission 5 years after establishing the diagnosis. Conclusion: Oligoclonal bands in the CSF may be helpful in the diagnosis of HE considering that it is still poorly understood entity. Also fast diagnosis of HE and treatment with corticosteroids are important for a full recovery of this patients.


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