Palpebral Fissure Height and Downgaze in Patients with Graves Upper Eyelid Retraction and Congenital Blepharoptosis

Ophthalmology ◽  
1995 ◽  
Vol 102 (8) ◽  
pp. 1218-1222 ◽  
Author(s):  
Fernando C. Guimarães ◽  
Antonio A.V. Cruz
2017 ◽  
Vol 2017 ◽  
pp. 1-5 ◽  
Author(s):  
Jurate Jankauskiene ◽  
Dalia Jarusaitiene

Purpose. To investigate juvenile Graves’ ophthalmopathy (GO) signs and compare Graves’ disease (GD) course in patients with or without GO. Patients and Methods. There were analyzed data (visual acuity, proptosis, palpebral fissure measurements, clinical activity score (CAS), and the course of GD) of 67 children who have been newly diagnosed with GD. 26.9% of patients with GD had signs of ophthalmopathy (GO+), and 73.1% were without ophthalmopathy (GO−). Results. Upper eyelid retraction (72.3%), proptosis (66.7%), and soft tissue changes (27.8−38.9%) were in GO+ patients. The palpebral fissure, CAS, and proptosis values were greater in the GO+ group than in the GO− group (p<0.001). GD course in GO+ patients was longer than that in GO− patients (p<0.001). The duration of the first remission was longer in GO− than in GO+ patients (p<0.001). The duration of first remission was longer than one year for 61.2% in GO− and 33.3% in GO+ patients (p<0.02). Conclusion. The common manifestations of juvenile GO patients were upper eyelid retraction, proptosis, and soft tissue involvement. The study demonstrates that pediatric patients with GO are more likely to have a severe course of autoimmune thyroid disease.


2007 ◽  
Vol 15 (2) ◽  
pp. 81-88 ◽  
Author(s):  
Bamini Gopinath ◽  
Cherie-Lee Adams ◽  
Reilly Musselman ◽  
Junichi Tani ◽  
Jack R. Wall

2019 ◽  
pp. 137-140
Author(s):  
Matthew J. Thurtell ◽  
Robert L. Tomsak

Supranuclear ophthalmoplegia results from an interruption of the saccadic, pursuit, optokinetic, or vergence inputs to the ocular motor nuclei. In this chapter, we begin by reviewing potential causes for difficulty reading. We next review the neuro-ophthalmic and neurologic features of progressive supranuclear palsy, which can include a vertical supranuclear ophthalmoplegia, convergence insufficiency, square-wave jerks, upper-eyelid retraction, reduced blink rate, apraxia of eyelid opening, and blepharospasm. We then discuss the differential diagnosis of progressive supranuclear palsy and point out clinical features that help to differentiate these conditions. Lastly, we present a practical approach to the management of the visual symptoms commonly caused by progressive supranuclear palsy.


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