Maternal Life History- Versus Gestation-Focused Assessment of Prenatal Exposure to Substances of Abuse

2000 ◽  
Vol 11 (4) ◽  
pp. 355-368 ◽  
Author(s):  
Risë B Goldstein ◽  
Gail J McAvay ◽  
Edward V Nunes ◽  
Myrna M Weissman
Keyword(s):  
Life History ◽  
Prenatal Exposure ◽  
Substances Of Abuse

scholarly journals Prevalence of prenatal exposure to substances of abuse: questionnaire versus biomarkers

2017 ◽  
Vol 14 (1) ◽  
Author(s):  
Antonella Chiandetti ◽  
Gimena Hernandez ◽  
María Mercadal-Hally ◽  
Airam Alvarez ◽  
Vicente Andreu-Fernandez ◽  
...  
Keyword(s):  
Prenatal Exposure ◽  
Substances Of Abuse

Interrelationships of factors of social development are more complex than Life History Theory predicts

2019 ◽  
Vol 42 ◽  
Author(s):  
Boris Kotchoubey
Keyword(s):  
Life History ◽  
Social Development ◽  
Life History Theory ◽  
Strong Correlations ◽  
Behavioral Features

Abstract Life History Theory (LHT) predicts a monotonous relationship between affluence and the rate of innovations and strong correlations within a cluster of behavioral features. Although both predictions can be true in specific cases, they are incorrect in general. Therefore, the author's explanations may be right, but they do not prove LHT and cannot be generalized to other apparently similar processes.

Reversal of cardiomyopathy resulting from prenatal exposure to ethanol

1984 ◽  
Vol 42 ◽  
pp. 716-717
Author(s):  
C. Uphoff ◽  
C. Nyquist-Battie
Keyword(s):  
Prenatal Exposure ◽  
Heart Development ◽  
Membrane Integrity ◽  
Ethanol Exposure ◽  
Prenatal Ethanol Exposure ◽  
Pathological Changes ◽  
Prenatally Exposed ◽  
Inner Mitochondrial Membrane ◽  
Fetal Alcohol ◽  
Newborn Mice

Fetal Alcohol Syndrone (FAS) is a syndrome with characteristic abnormalities resulting from prenatal exposure to ethanol. In many children with FAS syndrome gross pathological changes in the heart are seen with septal defects the most prevalent abnormality recorded. Few studies in animal models have been performed on the effects of ethanol on heart development. In our laboratory, it has been observed that prenatal ethanol exposure of Swiss albino mice results in abnormal cardiac muscle ultrastructure when mice were examined at birth and compared to pairfed and normal controls. Fig. 1 is an example of the changes that are seen in the ethanol-exposed animals. These changes include enlarged mitochondria with loss of inner mitochondrial membrane integrity and loss of myofibrils. Morphometric analysis substantiated the presence of these alterations from normal cardiac ultrastructure. The present work was undertaken to determine if the pathological changes seen in the newborn mice prenatally exposed to ethanol could be reversed with age and abstinence.

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