Horse Lumbrical Muscle: Possible Structural and Functional Reorganization in Regressive Muscle

2002 ◽  
Vol 31 (2) ◽  
pp. 85-98 ◽  
Author(s):  
W. KLOMKLEAW ◽  
Y. KASASHIMA ◽  
G. A. FULLER ◽  
A. KOBAYASHI ◽  
T. YOSHIHARA ◽  
...  
2021 ◽  
Vol 49 (01) ◽  
pp. 079-087
Author(s):  
Esther Fernández Tormos ◽  
Fernando Corella Montoya ◽  
Blanca Del Campo Cereceda ◽  
Montserrat Ocampos Hernández ◽  
Teresa Vázquez Osorio ◽  
...  

AbstractRecurrence of carpal tunnel syndrome implies the reappearance of symptoms after release surgery. If the cause of recurrence is not an incomplete release, but a traction neuritis, the tendency is to add to the revision surgery of the carpal tunnel the use of flaps to cover the median nerve. These flaps establish a physical barrier between the nerve and the rest of the adjacent structures, preventing adhesions, and providing neovascularization and better nerve sliding.In the present work, we detail a revision surgery in which the first lumbrical muscle is used as a covering flap. This flap has two benefits. Firstly, it acts as a vascularized coverage for the median nerve (avoiding the formation of fibrosis and favoring its sliding); secondly, a structure that takes up space is removed from the carpal tunnel, thus reducing the pressure within it.Along with the explanation of the technique, the present article provides a detailed description of the anatomical variability of the first lumbrical muscle and its vascularization, as well as the results of a cadaveric study on the location of the vascular pedicle of the first lumbrical muscle.


2021 ◽  
Author(s):  
Heather R. McGregor ◽  
Jessica K. Lee ◽  
Edwin R. Mulder ◽  
Yiri E. De Dios ◽  
Nichole E. Beltran ◽  
...  

Neuroreport ◽  
1990 ◽  
Vol 1 (2) ◽  
pp. 161-164 ◽  
Author(s):  
Francisco Gonzalez-Lima ◽  
Javier Agudo

2015 ◽  
Vol 2015 ◽  
pp. 1-8 ◽  
Author(s):  
Amanda G. Jaimes-Bautista ◽  
Mario Rodríguez-Camacho ◽  
Iris E. Martínez-Juárez ◽  
Yaneth Rodríguez-Agudelo

The impairment in episodic memory system is the best-known cognitive deficit in patients with temporal lobe epilepsy (TLE). Recent studies have shown evidence of semantic disorders, but they have been less studied than episodic memory. The semantic dysfunction in TLE has various cognitive manifestations, such as the presence of language disorders characterized by defects in naming, verbal fluency, or remote semantic information retrieval, which affects the ability of patients to interact with their surroundings. This paper is a review of recent research about the consequences of TLE on semantic processing, considering neuropsychological, electrophysiological, and neuroimaging findings, as well as the functional role of the hippocampus in semantic processing. The evidence from these studies shows disturbance of semantic memory in patients with TLE and supports the theory of declarative memory of the hippocampus. Functional neuroimaging studies show an inefficient compensatory functional reorganization of semantic networks and electrophysiological studies show a lack of N400 effect that could indicate that the deficit in semantic processing in patients with TLE could be due to a failure in the mechanisms of automatic access to lexicon.


Author(s):  
Raffaele Nardone ◽  
Luca Sebastianelli ◽  
Davide Ferrazzoli ◽  
Francesco Brigo ◽  
Piergiorgio Lochner ◽  
...  

2012 ◽  
Vol 120 (04) ◽  
pp. 197-198 ◽  
Author(s):  
S. Müller-Krebs ◽  
W. Zhang ◽  
L. Kihm ◽  
J. Reiser ◽  
P. Nawroth ◽  
...  

AbstractDuring long-term peritoneal dialysis (PD) the peritoneal membrane underlies processes of structural and functional reorganization mediated by high glucose and reactive glucose metabolites that are contained in PD solutions; this process is accompanied by increasing fibrosis. Mechanistically, the peritoneal damage is triggered by the interaction of advanced glycation end-products with their receptor; this is true for rodents as well as for humans. With this knowledge interventional strategies can be tested in rodent models, among them are the lipid soluble vitamin B1 analogue benfotiamine (BF) or detoxifying enzymes such as glyoxalase. Of additional interest is the finding that PD fluids do not only cause local but also systemic damage, in particular renal and cardiovascular. In the case of kidney damage, the intervention with BF was also successful. Taken together, PD can be regarded as a local model for long-term diabetes together with systemic aspects of damage.


2008 ◽  
Vol 46 (12) ◽  
pp. 2936-2942 ◽  
Author(s):  
P. Paiement ◽  
F. Champoux ◽  
B.A. Bacon ◽  
M. Lassonde ◽  
J.-P. Gagné ◽  
...  

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