Anatomically Corrected Malposition of the Great Arteries, Inflow Ventricular Septal Defect, and Subaortic Stenosis: Diagnostic and Operative Implications

1989 ◽  
Vol 37 (03) ◽  
pp. 147-150 ◽  
Author(s):  
F. Schmid ◽  
H. Oelert ◽  
H. Jakob ◽  
I. Luhmer ◽  
D. Schranz
1998 ◽  
Vol 8 (2) ◽  
pp. 217-220 ◽  
Author(s):  
Lindsey D Allan ◽  
Howard D Apfel ◽  
Yosef Levenbrown ◽  
Jan M Quaegebeur

AbstractBackgroundInterrupted aortic arch is often associated with subaortic narrowing and hypoplasia of the aortic orifice. The best surgical strategy for the management of these additional lesions is a matter of current debate.Methods and ResultsBetween 1986 and 1996, 19 patients underwent repair of interrupted aortic arch with closure of ventricular septal defect in a single stage, with no attempt at subaortic resection, irrespective of the dimensions of the left ventricular outflow tract. There was no perioperative hospital mortality, and all patients were alive at 1 year. Follow-up ranges from 0.75 −10 years, with a mean 4.2 ± 3.0 years. Seven patients (37%) have required reintervention for relief of subaortic stenosis, 2 of whom have died.ConclusionsOur results suggest that primary one-stage biventricular repair can be accomplished with low perioperative mortality without addressing the subaortic region. Further long-term follow-up will determine whether this is accomplished at the expense of later morbidity and mortality.


1990 ◽  
Vol 49 (3) ◽  
pp. 429-434 ◽  
Author(s):  
Serafin Y. DeLeon ◽  
Michel N. Ilbawi ◽  
Rene A. Arcilla ◽  
Otto G. Thilenius ◽  
Jose A. Quinones ◽  
...  

1998 ◽  
Vol 46 (01) ◽  
pp. 33-36 ◽  
Author(s):  
T. Watanabe ◽  
K. Tajima ◽  
Y. Sakai ◽  
T. Shimomura ◽  
M. Song ◽  
...  

1982 ◽  
Vol 2 (4) ◽  
pp. 265-269 ◽  
Author(s):  
David J. Fisher ◽  
A. Rebecca Snider ◽  
Norman H. Silverman ◽  
Paul Stanger

1995 ◽  
Vol 5 (4) ◽  
pp. 367-369
Author(s):  
Robin Joseph Pinto ◽  
Satyavan Sharma ◽  
Nemish Shah

AbstractWe report a case of interruption of the aortic arch associated with patency of the arterial duct, restrictive ventricular septal defect, obstruction of the left ventricular outflow due to a discrete subaortic shelf and severe pulmonary arterial hypertension. The patient survived to adulthood, when a complete angiographic diagnosis was made. The subaortic obstruction was treated by percutaneous balloon dilation performed through a right brachial arteriotomy. Correction of the interrupted arch was carried out surgically using a woven Dacron graft from the aortic arch to the descending thoracic aorta associated with ligation of the duct. Hemodynamic studies after one year revealed a marked decrease in pulmonary vascular resistance and a sustained reduction in the gradient across the left ventricular outflow tract. The case is of interest in the light of the unusual survival and the success of staged management of this complex situation.


1996 ◽  
Vol 39 ◽  
pp. 39-39
Author(s):  
Michael D VanAuker ◽  
Pedro J del Nido ◽  
Theresa A Tacy ◽  
Gunnlaugur Sigfusson ◽  
Edward G Cape

1993 ◽  
Vol 105 (2) ◽  
pp. 289-296 ◽  
Author(s):  
Edward L. Bove ◽  
L. LuAnn Minich ◽  
Ara K. Pridjian ◽  
Flavian M. Lupinetti ◽  
A. Rebecca Snider ◽  
...  

PEDIATRICS ◽  
1986 ◽  
Vol 77 (5) ◽  
pp. 687-691
Author(s):  
Susie C. Truesdell ◽  
David J. Skorton ◽  
Ronald M. Lauer

To determine the life insurability of young people with cardiovascular disease, we sent questionnaires to 99 life insurance companies concerning 18 congenital defects, rheumatic heart disease, and four dysrhythmias. We received 50 responses (50%) from companies whose sales make up 41% of the life insurance market. The concensus of insurability for the defects listed was: standard rates—mild pulmonic stenosis, rheumatic fever without carditis, mitral valve prolapse without regugitation, and the following postoperative lesions: patent ductus arteriosus, atrial septal defect, pulmonic stenosis, ventricular septal defect; uninsurable—most unoperated lesions, postoperative lesions with complex dysrrhythmias, severe aortic insufficiency, idiopathic hypertrophic subaortic stenosis, Ebstein's anomaly, truncus arteriosus, tricuspid atresia; insurable at increased rates—most other defects, including dextrotransposition of the great vessels, postoperative aortic stenosis, mild aortic insufficiency, postoperative coarctation of aorta, postoperative tetralogy of Fallot, and small ventricular septal defect. We conclude that life insurance is available to many children with cardiovascular disease, including most postoperative patients. Whether the increased rates requested for some defects are prohibitive is a matter to be decided by each family.


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