scholarly journals Effects of magnetic stimulation over supplementary motor area on movement in Parkinson's disease

Brain ◽  
1996 ◽  
Vol 119 (3) ◽  
pp. 815-822 ◽  
Author(s):  
Ross Cunnington ◽  
Robert Iansek ◽  
Gary W. Thickbroom ◽  
Bev A. Laing ◽  
Frank L. Mastaglia ◽  
...  
2019 ◽  
Vol 12 (1) ◽  
pp. 152-160 ◽  
Author(s):  
Silvia Casarotto ◽  
Francesco Turco ◽  
Angela Comanducci ◽  
Alessio Perretti ◽  
Giorgio Marotta ◽  
...  

1997 ◽  
Vol 147 (2) ◽  
pp. 177-183 ◽  
Author(s):  
Saša R. Filipović ◽  
Nadežda Čovičković-Šternić ◽  
Vladan M. Radović ◽  
Nataša Dragašević ◽  
Marina Stojanović-Svetel ◽  
...  

2014 ◽  
Vol 262 (2) ◽  
pp. 357-364 ◽  
Author(s):  
Carsten Eggers ◽  
Miriam Günther ◽  
John Rothwell ◽  
Lars Timmermann ◽  
Diane Ruge

2018 ◽  
Vol 39 (9) ◽  
pp. 3691-3700 ◽  
Author(s):  
Stefanie Kübel ◽  
Katharina Stegmayer ◽  
Tim Vanbellingen ◽  
Sebastian Walther ◽  
Stephan Bohlhalter

2021 ◽  
Vol 12 (1) ◽  
Author(s):  
Ashwini Oswal ◽  
Chunyan Cao ◽  
Chien-Hung Yeh ◽  
Wolf-Julian Neumann ◽  
James Gratwicke ◽  
...  

AbstractParkinson’s disease (PD) is characterised by the emergence of beta frequency oscillatory synchronisation across the cortico-basal-ganglia circuit. The relationship between the anatomy of this circuit and oscillatory synchronisation within it remains unclear. We address this by combining recordings from human subthalamic nucleus (STN) and internal globus pallidus (GPi) with magnetoencephalography, tractography and computational modelling. Coherence between supplementary motor area and STN within the high (21–30 Hz) but not low (13-21 Hz) beta frequency range correlated with ‘hyperdirect pathway’ fibre densities between these structures. Furthermore, supplementary motor area activity drove STN activity selectively at high beta frequencies suggesting that high beta frequencies propagate from the cortex to the basal ganglia via the hyperdirect pathway. Computational modelling revealed that exaggerated high beta hyperdirect pathway activity can provoke the generation of widespread pathological synchrony at lower beta frequencies. These findings suggest a spectral signature and a pathophysiological role for the hyperdirect pathway in PD.


2008 ◽  
Vol 23 (11) ◽  
pp. 1524-1531 ◽  
Author(s):  
Masashi Hamada ◽  
Yoshikazu Ugawa ◽  
Sadatoshi Tsuji ◽  

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