Therapeutically Targeting Microvascular Leakage In Experimental Hemorrhagic Shock

Shock ◽  
2021 ◽  
Vol Publish Ahead of Print ◽  
Author(s):  
Anoek L.I. van Leeuwen ◽  
Marieke P. Borgdorff ◽  
Nicole A.M. Dekker ◽  
Charissa E. van den Brom
1992 ◽  
Vol 48 (7) ◽  
pp. 663-667 ◽  
Author(s):  
S. Guarini ◽  
C. Bazzani ◽  
S. Tagliavini ◽  
A. Bertolini ◽  
W. Ferrari

1994 ◽  
Vol 38 (6) ◽  
pp. 311
Author(s):  
THOMAS H. GOODIN ◽  
ELUOTT B. GROSSBARD ◽  
ROBERT J. KAUFMAN ◽  
THOMAS J. RICHARD ◽  
RONALD J. KOLATA ◽  
...  

1956 ◽  
Vol 188 (1) ◽  
pp. 86-90 ◽  
Author(s):  
Dorothy K. Rosenbaum ◽  
Edward D. Frank ◽  
Alexander M. Rutenburg ◽  
Howard A. Frank

The ‘high-energy phosphate’ content of liver tissue declines during the course of hemorrhagic shock in the dog. The degree of decline does not correlate with the loss of responsiveness to transfusion therapy. Phosphate energy stores depleted during hemorrhagic shock are rapidly rebuilt following replacement transfusion. Prior treatment with aureomycin reduces the depletion of phosphate energy stores during hemorrhagic shock. Aureomycin given in the same manner has been found to preserve the dog's responsiveness to transfusion therapy. The protective action of aureomycin in hemorrhagic shock is not attributable to the preservation of phosphate energy stores, however, because the change in these stores does not correlate with responsiveness to blood replacement.


1984 ◽  
Vol 13 (1) ◽  
pp. 26-29 ◽  
Author(s):  
H.G. Garrison ◽  
A.R. Hansen ◽  
R.E. Cross ◽  
H.J. Proctor

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