scholarly journals Choice of host cell line is essential for the functional glycosylation of the fragment crystallizable (Fc) region of human IgG1 inhibitors of influenza B viruses

2019 ◽  
Author(s):  
Patricia A. Blundell ◽  
Dongli Lu ◽  
Anne Dell ◽  
Stuart M. Haslam ◽  
Richard J. Pleass

AbstractAntibodies are glycoproteins that carry a conserved N-linked carbohydrate attached to the Fc, whose presence and fine structure profoundly impacts on their in vivo immunogenicity, pharmacokinetics and functional attributes. The host cell line used to produce IgG has a major impact on this glycosylation, as different systems express different glycosylation enzymes and transporters that contribute to the specificity and heterogeneity of the final IgG-Fc glycosylation profile. Here we compare two panels of glycan-adapted IgG1-Fc mutants expressed in either the HEK 293-F or CHO-K1 systems. We show that the types of N-linked glycans between matched pairs of Fc mutants vary significantly, and in particular with respect to sialylation. These cell line effects on glycosylation profoundly influence the ability of the engineered Fcs to interact with either human or pathogen receptors. For example, we describe Fc mutants that potently disrupted influenza B-mediated agglutination of human erythrocytes when expressed in CHO-K1 but not in HEK 293-F cells.

2020 ◽  
Vol 204 (4) ◽  
pp. 1022-1034 ◽  
Author(s):  
Patricia A. Blundell ◽  
Dongli Lu ◽  
Anne Dell ◽  
Stuart Haslam ◽  
Richard J. Pleass

2015 ◽  
Vol 9 (S9) ◽  
Author(s):  
Anett Ritter ◽  
Johannes Wienberg ◽  
Bernd Voedisch ◽  
Burkhard Wilms ◽  
Sabine Geisse ◽  
...  
Keyword(s):  

2012 ◽  
Vol 148 (1-2) ◽  
pp. 116-125 ◽  
Author(s):  
Gabriela Calzada-Nova ◽  
Robert J. Husmann ◽  
William M. Schnitzlein ◽  
Federico A. Zuckermann

Author(s):  
Candy Rivas ◽  
Michael Yee ◽  
Kenneth Addison ◽  
Marissa Lovett ◽  
Kasturi Pal ◽  
...  

Background and Purpose: Despite availability of a variety of treatment options, many asthma patients have poorly controlled disease with frequent exacerbations. Proteinase-activated receptor-2 (PAR2) has been identified in pre-clinical animal models as important to asthma initiation and progression following allergen exposure. Proteinase activation of PAR2 induces intracellular Ca2+, mitogen activated protein kinase (MAPK) and -arrestin signaling the airway, leading to both inflammatory and protective effects. We have developed C391, a potent PAR2 antagonist effective in blocking peptidomimetic- and trypsin-induced PAR2 signaling in vitro as well as reducing inflammatory PAR2-associated pain in vivo. We hypothesized that PAR2 reduction with C391 would attenuate allergen-induced asthma indicators in murine models. Experimental Approach: We evaluated the ability for C391 to alter Alternaria alternata-induced PAR2 signaling pathways in vitro using a human airway epithelial cell line that naturally expresses PAR2 (16HBE14o-) and a transfected embryonic cell line (HEK 293). We next evaluated the ability for C391 to reduce A. alternata-induced asthma indicators in vivo in two murine strains. Key Results: C391 blocked A. alternata-induced, PAR2-dependent Ca2+ and MAPK signaling in 16HBE14o- cells, as well as -arrestin recruitment in HEK 293 cells. C391 effectively attenuated A. alternata-induced inflammation, mucus production, mucus cell hyperplasia and airway hyperresponsiveness in acute asthma murine models. Conclusions and Implications: To our knowledge, this is the first demonstration of pharmacological intervention of PAR2 to reduce allergen-induced asthma indicators in vivo. These data support further development of PAR2 antagonists as potential first-in-class allergic asthma drugs.


2013 ◽  
Vol 12 (3) ◽  
pp. 1223-1234 ◽  
Author(s):  
Eden P. Go ◽  
Hua-Xin Liao ◽  
S. Munir Alam ◽  
David Hua ◽  
Barton F. Haynes ◽  
...  

mAbs ◽  
2017 ◽  
Vol 10 (2) ◽  
pp. 290-303 ◽  
Author(s):  
Oliver Popp ◽  
Samuel Moser ◽  
Jörg Zielonka ◽  
Petra Rüger ◽  
Silke Hansen ◽  
...  

2007 ◽  
Vol 130 (3) ◽  
pp. 300-310 ◽  
Author(s):  
Yutaka Kanda ◽  
Harue Imai-Nishiya ◽  
Reiko Kuni-Kamochi ◽  
Katsuhiro Mori ◽  
Miho Inoue ◽  
...  

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