Saddle nose deformity and septal perforation in granulomatosis with polyangiitis

2017 ◽  
Vol 43 (1) ◽  
pp. 291-299
Author(s):  
A. Coordes ◽  
S.M. Loose ◽  
V.M. Hofmann ◽  
G.S. Hamilton ◽  
F. Riedel ◽  
...  

1979 ◽  
Vol 22 (1) ◽  
pp. 101-102 ◽  
Author(s):  
Benjamin K Harris ◽  
Robert Tello


2002 ◽  
Vol 81 (8) ◽  
pp. 562-563 ◽  
Author(s):  
Ronald J. Vilela ◽  
Carol Langford ◽  
Linda McCullagh ◽  
Erik S. Kass

The effects of chronic cocaine abuse have been widely described in the literature. Common complications include nasal septal perforation, saddle-nose deformity, and palatal perforation. Erosion of the external structures of the face has not been as extensively described, nor have oronasal fistulas that involve structures other than the hard or soft palate. In this article, we present the first reported case of cocaine-induced external nasal erosion that included multiple oronasal fistulas in the anterior gingival sulcus but did not involve the hard or soft palate. We stress the importance of a thorough history in such patients and consideration of all possible diagnoses, including drug abuse.



2020 ◽  
pp. 54-55
Author(s):  
Sanjeev Bhagat ◽  
Jasmine Ratti ◽  
Sachiv Garg

Button batteries usage in electrical devices like toys, watches, calculators etc has significantly increased in recent times and due to their easy availability and small size, these batteries can be very easily inserted by small children in the nose. A nasal button battery is an otorhinolaryngological emergency as it can lead to severe damage, necrosis, and perforation of the nasal septum, intranasal synechiae , and nasal deformity [1]. Button batteries should ideally be removed in a controlled setting under general anesthesia if they cannot be removed in the outpatient department. We hereby report this case to advocate that button battery in the nasal cavity is a serious condition and can lead to grave complications like nasal synechiae, granulations , septal perforation further causing saddle nose deformity.



QJM ◽  
2017 ◽  
Vol 111 (1) ◽  
pp. 55-55 ◽  
Author(s):  
Byung Hoon Ban ◽  
Jayne Littlejohn Crowe ◽  
Maria Tudor


2017 ◽  
Vol 143 (5) ◽  
pp. 507 ◽  
Author(s):  
Waleed H. Ezzat ◽  
Rebecca A. Compton ◽  
Krystyne C. Basa ◽  
Jessica Levi




2017 ◽  
Vol 41 (6) ◽  
pp. 1463-1464
Author(s):  
Osamu Ito ◽  
Tomoyuki Yano ◽  
Minako Ito


Author(s):  
Gwan Choi ◽  
Joo Yeon Kim ◽  
Yeong Joon Kim ◽  
Seong Uk Jang ◽  
Joo-Wan Jo ◽  
...  


1987 ◽  
Vol 79 (6) ◽  
pp. 1012
Author(s):  
M. J. Earley ◽  
J. Lendrum ◽  
Colin R. Rayner


2017 ◽  
Author(s):  
Alexandra Villa-Forte ◽  
Brian F Mandell

Vasculitis is defined by histologic evidence of inflammation that involves the blood vessels. The diagnosis of a specific primary vasculitic disorder depends on the pattern of organ involvement, the histopathology, the size of affected blood vessels, and the exclusion of diseases that can cause “secondary” vasculitis. This review presents an approach to the patient suspected of having vasculitis, and goes on to discuss small vessel vasculitis, granulomatosis with polyangiitis, eosinophilic granulomatosis with polyangiitis, microscopic polyangiitis, polyarteritis nodosa, Kawasaki disease, large vessel arteritis, and Behçet disease. Figures show classification of the systemic vasculitis syndromes, the relationships among the causes of small vessel (“hypersensitivity”) vasculitis, palpable purpura of the distal extremities, saddle nose deformity, the nodular infiltrates of the lung in granulomatosis with polyangiitis shown on plain radiograph as well as computed tomography, necrotizing scleritis, livedo reticularis, and angiograms of a patient with Takayasu arteritis. Tables list selected laboratory tests for patients with multisystem disease and possible vasculitis, practical comments on immunosuppressive therapies for vasculitis, features of vasculitis, diagnostic criteria for Kawasaki disease, and giant cell arteritis. This review contains 8 highly rendered figures, 5 tables, and 59 references.



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