Wide QRS Complex Tachycardia in a Patient With Concealed Accessory Pathway: What Is the Mechanism?

2016 ◽  
Vol 27 (9) ◽  
pp. 1121-1123
Author(s):  
MAREK JASTRZEBSKI ◽  
MACIEJ PITAK ◽  
ANDRZEJ RUDZINSKI ◽  
DANUTA CZARNECKA
2013 ◽  
Vol 163 (3) ◽  
pp. S198
Author(s):  
H. Sunman ◽  
K. Aytemir ◽  
Ş.G. Fatihoglu ◽  
U. Canpolat ◽  
N. Maharjan ◽  
...  

Author(s):  
Mustafa A Tatlisu ◽  
Oguz Konal ◽  
Adem Atici ◽  
Omer Faruk Baycan ◽  
Yusuf Yilmaz

2018 ◽  
Vol 59 (4) ◽  
pp. 857-861 ◽  
Author(s):  
Kyoichiro Yazaki ◽  
Kenji Enta ◽  
Masahiro Watarai ◽  
Mitsuru Kahata ◽  
Asako Kumagai ◽  
...  

2005 ◽  
Vol 15 (3) ◽  
pp. 315-318 ◽  
Author(s):  
Radu Vatasescu ◽  
Laszlo Kornyei ◽  
Tamas Szili-Torok

Radiofrequency lesions can, theoretically, be the substrate for new persistent arrhythmias. As far as we know, this has never previously been encountered after transcatheter ablation of accessory pathways. A child with Wolff–Parkinson–White syndrome was referred for radiofrequency catheter ablation of a left-sided accessory pathway. After successful ablation of the accessory pathway using a retrograde transaortic approach, the child developed an incessant wide QRS complex tachycardia at slow rate that was resistant to pharmacologic interventions. The focus of the tachycardia was identical to the ventricular site of insertion of the eliminated accessory pathway.


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