Acute skin trauma induces hyperemia, but superficial papillary nutritive perfusion remains unchanged

2017 ◽  
Vol 24 (7) ◽  
pp. e12389
Author(s):  
Liv Kristin Sundheim ◽  
Ane Halse Sporastøyl ◽  
Torjus Wester ◽  
Göran Salerud ◽  
Knut Kvernebo
Keyword(s):  
Nutritive Perfusion

Can Transcutaneous Oximetry Detect Nutritive Perfusion Disturbances in Patients with Lower Limb Ischemia?

1995 ◽  
Vol 49 (3) ◽  
pp. 315-324 ◽  
Author(s):  
D.Th. Ubbink ◽  
M.J.H.M. Jacobs ◽  
D.W. Slaaf
Keyword(s):  
Lower Limb ◽  
Limb Ischemia ◽  
Lower Limb Ischemia ◽  
Transcutaneous Oximetry ◽  
Nutritive Perfusion

Can the Green Laser Doppler Measure Skin-Nutritive Perfusion in Patients with Peripheral Vascular Disease?

2000 ◽  
Vol 37 (3) ◽  
pp. 195-201 ◽  
Author(s):  
Dirk T. Ubbink ◽  
Igor I. Tulevski ◽  
Michael J.H.M. Jacobs
Keyword(s):  
Vascular Disease ◽  
Peripheral Vascular Disease ◽  
Green Laser ◽  
Laser Doppler ◽  
Peripheral Vascular ◽  
Nutritive Perfusion

Improvement of nutritive perfusion after free tissue transfer by local heat shock-priming-induced preservation of capillary flowmotion

2005 ◽  
Vol 123 (1) ◽  
pp. 102-108 ◽  
Author(s):  
Martin Rücker ◽  
Bülent Kadirogullari ◽  
Brigitte Vollmar ◽  
Wolfgang J. Spitzer ◽  
Michael D. Menger
Keyword(s):  
Heat Shock ◽  
Free Tissue Transfer ◽  
Local Heat ◽  
Tissue Transfer ◽  
Nutritive Perfusion

Nutritive perfusion at donor site after microvascular fibula transfer

Microsurgery ◽  
2003 ◽  
Vol 23 (4) ◽  
pp. 306-312 ◽  
Author(s):  
Frank Hölzle ◽  
Sami Swaid ◽  
Dirk Nolte ◽  
Klaus-Dietrich Wolff
Keyword(s):  
Donor Site ◽  
Nutritive Perfusion

The time course of restoration of nutritive perfusion, myocardial oxygen consumption, and regional function after coronary thrombolysis

1990 ◽  
Vol 1 (6) ◽  
pp. 687-696 ◽  
Author(s):  
C. Gregory Henes ◽  
Steven R. Bergmann ◽  
Julio E. Perez ◽  
Burton E. Sobel ◽  
Edward M. Geltman
Keyword(s):  
Oxygen Consumption ◽  
Myocardial Oxygen Consumption ◽  
Time Course ◽  
Regional Function ◽  
Coronary Thrombolysis ◽  
Nutritive Perfusion

Visualization of Nutritive Perfusion Following Tourniquet Ischemia in Arterial Pattern Skin Flaps

1994 ◽  
Vol 94 (5) ◽  
pp. 652-660 ◽  
Author(s):  
Rainer K. Saetzler ◽  
Hans-Anton Lehr ◽  
John H. Barker ◽  
Markus Kamler ◽  
Thomas J. Galla ◽  
...  
Keyword(s):  
Skin Flaps ◽  
Nutritive Perfusion ◽  
Tourniquet Ischemia

NUTRITIVE PERFUSION OF PANCREATIC ENDOCRINE TISSUE DURING HEMORRHAGIC HYPOTENSION

Shock ◽  
2008 ◽  
Vol 30 (4) ◽  
pp. 428-433 ◽  
Author(s):  
Brigitte Vollmar ◽  
Knut Franke ◽  
Michael D. Menger
Keyword(s):  
Endocrine Tissue ◽  
Hemorrhagic Hypotension ◽  
Nutritive Perfusion

Leukocyte stasis in hepatic sinusoids

1996 ◽  
Vol 270 (5) ◽  
pp. G798-G803 ◽  
Author(s):  
B. Vollmar ◽  
S. Richter ◽  
M. D. Menger
Keyword(s):  
Flow Resistance ◽  
Ischemia Reperfusion ◽  
Flow Analysis ◽  
Leukocyte Trafficking ◽  
Intravital Fluorescence Microscopy ◽  
Major Fraction ◽  
Ongoing Debate ◽  
No Reflow ◽  
Perfusion Failure ◽  
Nutritive Perfusion

There is ongoing debate on the significance of capillary leukostasis for the manifestation of ischemia-reperfusion (I/R)-induced capillary “no-reflow”. Using intravital fluorescence microscopy, we studied leukocyte trafficking through the hepatic microvasculature and the relevance of sinusoidal leukostasis for nutritive perfusion failure in rats after hepatic I/R (n = 8). Sham-operated animals (n = 8) served as controls. Hepatic reperfusion was characterized by perfusion failure of individual sinusoids and a significant increase of sinusoidal leukostasis. However, in both nonischemic and postischemic livers, the major fraction of sinusoids presenting with stagnant leukocytes were found perfused (97 +/- 1 and 73 +/- 5%, respectively), whereas only 3 +/- 1 and 27 +/- 5% failed to conduct flow. Analysis of leukocyte trafficking in sinusoids with leukostasis revealed a marked reduction of leukocyte velocity and leukocyte flux in nonischemic and postischemic livers compared with sinusoids without leukostasis. Thus stagnant leukocytes retard cellular passage through hepatic sinusoids, probably due to an increase of flow resistance. However, the fact that during postischemic reperfusion > 70% of the sinusoids accommodating stagnant leukocytes are still perfused indicates that sinusoidal leukostasis per se does not necessarily determine perfusion failure ("no-reflow") after I/R of the liver.

POLYETHYLENE-10 NUTRITIVE PERFUSION OF PANCREATIC ENDOCRINE TISSUE DURING HEMORRHAGIC HYPOTENSION

Shock ◽  
2008 ◽  
pp. 1
Author(s):  
Brigitte Vollmar ◽  
Knut Franke ◽  
Michael D. Menger
Keyword(s):  
Endocrine Tissue ◽  
Hemorrhagic Hypotension ◽  
Nutritive Perfusion

Skin microvascular adaptations during maturation and aging of hairless mice

2000 ◽  
Vol 279 (4) ◽  
pp. H1591-H1599 ◽  
Author(s):  
Brigitte Vollmar ◽  
Martin Morgenthaler ◽  
Michaela Amon ◽  
Michael D. Menger
Keyword(s):  
Functional Analysis ◽  
Adult Life ◽  
Capillary Density ◽  
Branching Pattern ◽  
Intravital Fluorescence Microscopy ◽  
Hairless Mice ◽  
Capillary Formation ◽  
Age Related ◽  
Nutritive Perfusion ◽  
Hyperemic Response

Using intravital fluorescence microscopy in the ears of hairless mice, we determined skin microvascular adaptations during the process of aging from juvenile to adult and senescent life (6–78 wk). Despite an increase of ear area within the first 36 wk, the number and branching pattern of both arteriolar and venular microvessels remained constant during the whole life period. Both arterioles and venules exhibited an increase in length, diameter, and intervascular distance up to the age of 36 wk. With the increase of the size of the ears, the observation that cutaneous capillary density remained unchanged implied new capillary formation. During aging to 78 wk, capillary density in the ears was reduced to ∼40%. Functional analysis revealed an appropriate hyperemic response to a 2-min period of ischemia during late juvenile and adult life, which, however, was markedly reduced during senescence. Thus, except for capillaries, there is no indication for age-related new vessel formation. The process of aging from adult to senescent life does not cause any significant remodeling but is associated with a decrease of nutritive perfusion and a functional impairment to respond to stimuli such as ischemia.

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