State of the Surface of Polycrystalline Silver after Exposure to Activated Oxygen

2019 ◽  
Vol 53 (15) ◽  
pp. 1983-1985
Author(s):  
O. G. Ashkhotov ◽  
S. A. Khubezhov ◽  
I. B. Ashkhotova
2018 ◽  
Vol 23 (6) ◽  
pp. 620-624
Author(s):  
O.G. Ashkhotov ◽  
◽  
O.G. S.A. Khubezhov ◽  
I.B. Ashhotova ◽  
◽  
...  

2009 ◽  
Vol 1192 ◽  
Author(s):  
Luke M. Davis ◽  
Tyler S. Stukenbroeker ◽  
Christopher J. Abelt ◽  
Joseph L. Scott ◽  
Evguenia Orlova ◽  
...  

AbstractA straightforward ambient temperature route to the fabrication of surface silver-metallized polyimide films is described. Silver(I) trifluoromethane sulfonate and a polyimide, derived from 2,2-bis(3,4-dicarboxyphenyl)hexafluoropropane dianhydride (6FDA) and an equimolar amount of 4,4'-oxydianiline (ODA) and 3,5-diaminobenzoic acid (DABA), were dissolved together in dimethylacetamide. Silver(I)-doped films were prepared at thicknesses of 25-50 microns and depleted of solvent by evaporation. The silver(I)-containing films were then treated with aqueous reducing agents, which brought forth silvered films exhibiting conductivity on the order of bulk polycrystalline silver and good specular reflectivity.


CrystEngComm ◽  
2020 ◽  
Vol 22 (3) ◽  
pp. 478-486 ◽  
Author(s):  
Lijuan Wang ◽  
Jens-Petter Andreassen ◽  
Seniz Ucar

Mono- and polycrystalline silver particles were formed with morphologies ranging from polyhedral, to hopper, dendritic and spherulitic particles with increasing supersaturation.


2001 ◽  
Vol 183 (3-4) ◽  
pp. 191-204 ◽  
Author(s):  
G.I.N. Waterhouse ◽  
G.A. Bowmaker ◽  
J.B. Metson

2002 ◽  
Vol 149 (11) ◽  
pp. E440 ◽  
Author(s):  
J. I. Millán ◽  
J. J. Ruiz ◽  
L. Camacho ◽  
R. Rodrı́guez-Amaro

2013 ◽  
Vol 114 (9) ◽  
pp. 093512 ◽  
Author(s):  
J. Q. Xu ◽  
S. J. Xiong ◽  
X. L. Wu ◽  
T. H. Li ◽  
J. C. Shen ◽  
...  

Parasitology ◽  
1985 ◽  
Vol 90 (2) ◽  
pp. 241-254 ◽  
Author(s):  
Stephanie M. Millott ◽  
F. E. G. Cox

Swiss mice with chronicTrypanosoma bruceiinfections become refractory to subsequent infection withBabesia microtiandB. rodhaini. Infection withB. microti7 days afterT. bruceiresulted in an obvious inhibition of the babesia parasitaemias and this inhibition became more profound as the time interval between the infections increased, until at 17–20 days the parasitaemias were totally abolished. Even after intravenous injection of large numbers of parasites parasitaemias were inhibited. Similar inhibition was obtained in BALB/c mice but not in C57BL/6 mice. Mice with establishedT. bruceiinfections also showed reduced susceptibility toB. rodhaini. In mice similarly infected withT. bruceiand the malaria parasitesPlasmodium chabaudi chabaudiandP. c. adamithe pre-patent periods were noticeably prolonged but the subsequent parasitaemias were unaffected. Infections withP. yoeliiwere unaffected.Trypanosoma bruceiinfections were not affected by the intracellular parasites. Among the mechanisms investigated to explain these findings were changes in red blood cell populations, cross-reacting antigens, the release of toxic factors and the generation of activated oxygen species. None of these could account for the inhibition observed.


2011 ◽  
Vol 2011 (8) ◽  
pp. 729-733 ◽  
Author(s):  
E. V. Kirillova ◽  
S. I. Dokashenko ◽  
V. P. Stepanov

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