scholarly journals Isolated hepatic artery injury in blunt abdominal trauma presenting as upper gastrointestinal bleeding: treatment with transcatheter embolisation

2012 ◽  
Vol 2012 (nov14 2) ◽  
pp. bcr2012007464-bcr2012007464
Author(s):  
B. Taslakian ◽  
O. Ghaith ◽  
A. Al-Kutoubi
2014 ◽  
Vol 3 (52) ◽  
pp. 12214-12216
Author(s):  
Hariprakash Rajendran ◽  
Baskaran R ◽  
Prema M ◽  
Karthik Raja K

2021 ◽  
Vol 15 (1) ◽  
Author(s):  
Kurniawan Kurniawan ◽  
I Dewa Nyoman Wibawa ◽  
Gde Somayana ◽  
I Ketut Mariadi ◽  
I Made Mulyawan

Abstract Background Hemobilia is a rare cause of upper gastrointestinal bleeding that originates from the biliary tract. It is infrequently considered in diagnosis, especially in the absence of abdominal trauma or history of hepatopancreatobiliary procedure, such as cholecystectomy, which can cause arterial pseudoaneurysm. Prompt diagnosis is crucial because its management strategy is distinct from other types of upper gastrointestinal bleeding. Here, we present a case of massive hemobilia caused by the rupture of a gastroduodenal artery pseudoaneurysm in a patient with a history of laparoscopic cholecystectomy 3 years prior to presentation. Case presentation A 44-year-old Indonesian female presented to the emergency department with complaint of hematemesis and melena accompanied by abdominal pain and icterus. History of an abdominal trauma was denied. However, she reported having undergone a laparoscopic cholecystectomy 3 years prior to presentation. On physical examination, we found anemic conjunctiva and icteric sclera. Nonvariceal bleeding was suspected, but esophagogastroduodenoscopy showed a blood clot at the ampulla of Vater. Angiography showed contrast extravasation from a gastroduodenal artery pseudoaneurysm. The patient underwent pseudoaneurysm ligation and excision surgery to stop the bleeding. After surgery, the patient’s vital signs were stable, and there was no sign of rebleeding. Conclusion Gastroduodenal artery pseudoaneurysm is a rare complication of laparoscopic cholecystectomy. The prolonged time interval, as compared with other postcholecystectomy hemobilia cases, resulted in hemobilia not being considered as an etiology of the gastrointestinal bleeding at presentation. Hemobilia should be considered as a possible etiology of gastrointestinal bleeding in patients with history of cholecystectomy, regardless of the time interval between the invasive procedure and onset of bleeding.


2020 ◽  
Vol 74 ◽  
pp. 230-233
Author(s):  
Laura Alonso-Lamberti Rizo ◽  
Carlos Bustamante Recuenco ◽  
Julián Cuesta Pérez ◽  
José Luis Ramos Rodríguez ◽  
Andrea Salazar Carrasco ◽  
...  

Open Medicine ◽  
2013 ◽  
Vol 8 (5) ◽  
pp. 665-668
Author(s):  
K. Habib ◽  
G. Williams

AbstractA literature trawl reveals a substantial number of reports on true visceral aneurysms, including the hepatic artery, but only a handful of cases of visceral pseudoaneurysms. The ones in relation to the biliary tree are associated with previous gall bladder surgery and can result in significant gastrointestinal bleeding. There are more than 10 reported cases of cystic artery pseudoaneurysms but a thorough search revealed only two cases in English (1,2) and perhaps one in Japanese literature of right hepatic artery pseudoaneurysm secondary to cholecystitis presenting as massive upper gastrointestinal bleed. We present a probable fourth case in a 52 year old woman with classical clinical/biochemical picture, typical radiological appearance and who underwent successful interventional radiological treatment of this condition.


2007 ◽  
Vol 54 (1) ◽  
pp. 41-45 ◽  
Author(s):  
D. Galun ◽  
D. Basaric ◽  
N. Lekic ◽  
Z. Raznatovic ◽  
S. Barovic ◽  
...  

The phenomenon now known as haemobilia was first recorded in XVII century by well known anatomist from Cambridge, Francis Glisson and his description was published in Anatomia Hepatis in 1654. Until today etiology, clinical presentation and management are clearly defined. Haemobilia is a rare clinical condition that has to be considered in differential diagnosis of upper gastrointestinal bleeding. In Western countries, the leading cause of haemobilia is hepatic trauma with bleeding from an intrahepatic branch of the hepatic artery into a biliary duct (mostly iatrogenic in origin, e.g. needle biopsy of the liver or percutaneous cholangiography). Less common causes include hepatic neoplasm; rupture of a hepatic artery aneurysm, hepatic abscess, choledocholithiasis and in the Orient, additional causes include ductal parasitism by Ascaris lumbricoides and Oriental cholangiohepatitis. Clinical presentation of heamobilia includes one symptom and two signs (Quinke triad): a. upper abdominal pain, b. upper gastrointestinal bleeding and c. jaundice. The complications of haemobilia are uncommon and include pancreatitis, cholecystitis and cholangitis. Investigation of haemobilia depends on clinical presentation. For patients with upper gastrointestinal bleeding oesophagogastroduodenoscopy is the first investigation choice. The presence of blood clot at the papilla of Vater clearly indicates the bleeding from biliary tree. Other investigations include CT and angiography. The management of haemobilia isdirected at stopping bleeding and relieving biliary obstruction. Today, transarterial embolization is the golden standard in the management of heamobilia and if it fails further management is surgical.


2016 ◽  
Vol 36 (1) ◽  
pp. 100-102
Author(s):  
Ritu Lamichhane ◽  
Binita G. Joshi ◽  
Nishant Wadhwa ◽  
Arun Gupta

Haemobilia is one rare but potentially life threatening complication of delayed haemorrhage following liver trauma which occurs as a result of pathological communication between bile ducts and intra or extrahepatic vessels. We describe here a case of two year old child who presented with upper gastrointestinal bleeding and developed haemobilia in one month duration after blunt abdominal trauma. CT angiography revealed a right hepatic artery pseudo aneurysm as the cause of haemobilia and was successfully treated with right hepatic artery embolisation.J Nepal Paediatr Soc 2016;36(1):100-102.


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