FACIAL NERVE DECOMPRESSION VIA COMBINED TRANSMASTOID AND TRANS MIDDLE CRANIAL FOSSA ROUTES FOR TRAUMATIC TOTAL FACIAL PARALYSIS

Author(s):  
EIJI YUMOTO ◽  
MASAKO MASUDA ◽  
YOSHIHIKO KUMAI ◽  
MOTOHIRO MORIOKA
1994 ◽  
Vol 111 (1) ◽  
pp. 70-75 ◽  
Author(s):  
Christopher J. Linstrom ◽  
Marek J. Krajewski ◽  
Aaron L. Shapiro ◽  
Salvatore Caruana

We describe a surgical technique in which the middle fossa craniotomy ordinarily used for facial nerve decompression and related surgery is extended superiorly to allow the harvest of a split-thickness calvarial graft. This graft allows the tegmen defect in middle fossa surgery to be repaired without shortening the original craniotomy plate. Avoiding the usual temporoparietal depression after middle cranial fossa surgery is both a structural and cosmetic benefit for the patient.


1996 ◽  
Vol 105 (1) ◽  
pp. 58-62 ◽  
Author(s):  
Clark A. Elliott ◽  
George H. Zalzal ◽  
Wendy R. Gottlieb

We reviewed 10 children who presented with facial paralysis after the onset of acute otitis media. The objective of the study was to examine the outcome of facial paralysis in children with acute otitis media treated without facial nerve decompression. Two groups were identified: 8 patients with incomplete paralysis and 2 with complete paralysis. Seven of the 8 patients with incomplete paralysis had rapid return of function after myringotomy and intravenous antibiotics. The eighth patient had delayed recovery requiring 9 months before complete return of function. The 2 patients with complete paralysis required mastoidectomy to control otorrhea and fever after initial myringotomy and antibiotics. Both patients had a prolonged recovery requiring 3 and 7 months for complete recovery. Patients with incomplete paralysis generally show rapid improvement following wide myringotomy and antibiotic treatment. A more protracted recovery may be expected in patients with complete paralysis; excellent return of function is expected when mastoidectomy without facial nerve decompression is employed.


2005 ◽  
Vol 119 (2) ◽  
pp. 144-147 ◽  
Author(s):  
Tuncay Ulug ◽  
S Arif Ulubil

Bilateral traumatic facial paralysis is a very rare clinical condition. Abducens palsy, associated with bilateral traumatic paralysis, is even rarer and has not been well described in the literature. In this report, a 24-year-old male, who developed immediate bilateral facial and right abducens paralyses following a motor vehicle accident, is presented. The patient was referred for neurotologic evaluation 22 days after the injury. Electroneurography (ENoG) demonstrated 100 per cent degeneration at the first examination and, correspondingly, electromyography showed no regeneration potentials. Using high-resolution computed tomography (HRCT), a longitudinal fracture on the right and a mixed-type fracture on the left were identified. The patient had good cochlear reserve on both sides. The decision for surgery was based not on ENoG, because of the delayed referral of the patient, but on the HRCT, which showed clear fracture lines on both sides. The middle cranial fossa approach for decompression of the right facial nerve was performed on the 55th day following the trauma, and a combined procedure using the middle cranial fossa and transmastoid approaches was applied for decompression of the left facial nerve on the 75th day following the trauma. On the right, there was dense fibrosis surrounding the geniculate ganglion and the proximal tympanic segment whereas, on the left, bone fragments impinging on the geniculate ganglion, dense fibrosis surrounding the geniculate ganglion, and a less extensive fibrotic tissue surrounding the pyramidal segment were encountered. There were no complications or hearing deterioration. At the one-year follow up, the patient had House-Brackmann (HB) grade 1 recovery on the right, and HB grade 2 recovery on the left side, and the abducens palsy regressed spontaneously. The middle cranial fossa approach and its combinations can be performed safely in bilateral temporal bone fractures as labyrinthine sparing procedures if done on separate occasions.


PEDIATRICS ◽  
1972 ◽  
Vol 49 (1) ◽  
pp. 102-109
Author(s):  
John J. Manning ◽  
Kedar K. Adour

In any instance of facial paralysis in a child, an effort should be made to determine immediately whether it is caused by a specific, treatable entity. Of 61 cases of facial paralysis in children seen in a Facial Paralysis Clinic, 38% were not Bell's palsy. Eight of the 61 children had disease amenable to specific therapy available today. Experience with 504 patients of all age groups seen within 4 years has led the authors to abandon facial nerve decompression in the treatment of Bell's palsy.


2013 ◽  
Vol 149 (2_suppl) ◽  
pp. P225-P225
Author(s):  
Ju Hyoung Lee ◽  
Joo Hyun Jung ◽  
Chang-Hyun Cho ◽  
Bokyung Kwak ◽  
Youn Hee Ju

2000 ◽  
Vol 109 (3) ◽  
pp. 255-257 ◽  
Author(s):  
Ho-Ki Lee ◽  
Won-Sang Lee ◽  
Ek-Ho Lee ◽  
Won Sok Kim

Detailed anatomic knowledge of the microsurgical anatomy of the perigeniculate ganglion area is essential to probing adjacent to the facial nerve by a translabyrinthine approach. This study was designed to investigate the surgical anatomy of the perigeniculate ganglion area of the facial nerve from a translabyrinthine point of view. We dissected 15 human temporal bones under a microscope, measured the lengths of the tympanic segment and the labyrinthine segment by a middle cranial fossa approach, and measured the angle between the tympanic and labyrinthine segments by a translabyrinthine approach. The distance of the facial nerve from the cochleariform process to the geniculate ganglion was 3.8 ± 0.7 mm. The length of the labyrinthine segment of the facial nerve was 4 ± 0.8 mm. The angle between the tympanic and labyrinthine segments from a translabyrinthine point of view was 26° ± 5°. Precise knowledge about the microsurgical anatomy of the perigeniculate ganglion area of the facial nerve from a translabyrinthine viewpoint is imperative for facial nerve decompression by a translabyrinthine approach.


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