scholarly journals Neurobehavioral Aspects of the Delayed Encephalopathy of Carbon Monoxide Intoxication: Case Report and Review

1995 ◽  
Vol 8 (1) ◽  
pp. 47-52 ◽  
Author(s):  
M. F. Mendez ◽  
R. C. Doss
Keyword(s):  
Carbon Monoxide ◽  
White Matter ◽  
White Matter Lesions ◽  
Subcortical White Matter ◽  
Clinical Aspects ◽  
Co Poisoning ◽  
Diffuse White Matter ◽  
Co Intoxication ◽  
Frontal Lobe Syndrome ◽  
Delayed Encephalopathy

We report the neurobehavioral aspects of the delayed encephalopathy of carbon monoxide (CO) intoxication in a 29 year old woman and review the literature. Four weeks after CO poisoning, the patient developed a frontal lobe syndrome, visuoperceptual impairment, and diffuse white matter lesions with an otherwise normal neurological examination. In contrast, patients with the classical syndrome also have a parkinsonian state or an akinetic–mute state. The delayed encephalopathy of CO poisoning usually results from demyelination of subcortical white matter, necrosis of the globus pallidus, or both. The clinical aspects, risk factors, neurobiological features, and therapy and prognosis are discussed.

Impact of subcortical white matter lesions on dopamine transporter SPECT

2013 ◽  
Vol 120 (7) ◽  
pp. 1053-1060 ◽  
Author(s):  
Elisabeth Funke ◽  
Andreas Kupsch ◽  
Ralph Buchert ◽  
Winfried Brenner ◽  
Michail Plotkin
Keyword(s):  
White Matter ◽  
Dopamine Transporter ◽  
White Matter Lesions ◽  
Subcortical White Matter

IC-P-083: Automated classification of periventricular and subcortical white matter lesions on MRI

2009 ◽  
Vol 5 (4S_Part_1) ◽  
pp. P38-P38
Author(s):  
Benjamin F.J. Verhaaren ◽  
Fedde van der Lijn ◽  
Stefan Klein ◽  
Aad van der Lugt ◽  
Wiro J. Niessen ◽  
...  
Keyword(s):  
White Matter ◽  
White Matter Lesions ◽  
Subcortical White Matter ◽  
Automated Classification

Diffuse white matter lesions in a case of herpes simplex encephalitis

2003 ◽  
Vol 250 (7) ◽  
pp. 867-868 ◽  
Author(s):  
Naohisa Ueda ◽  
Hidetake Miyasaki ◽  
Yoshiyuki Kuroiwa
Keyword(s):  
Herpes Simplex ◽  
White Matter ◽  
Herpes Simplex Encephalitis ◽  
White Matter Lesions ◽  
Diffuse White Matter

Migraine with aura white matter lesions: preliminary data on clinical aspects

2017 ◽  
Vol 38 (S1) ◽  
pp. 7-10 ◽  
Author(s):  
Alberto Galli ◽  
P. Di Fiore ◽  
G. D’Arrigo ◽  
C. Uggetti ◽  
S. Squarza ◽  
...  
Keyword(s):  
White Matter ◽  
Migraine With Aura ◽  
Preliminary Data ◽  
White Matter Lesions ◽  
Clinical Aspects

Cortical deficits in multiple sclerosis on the basis of subcortical lesions

2000 ◽  
Vol 6 (1) ◽  
pp. 50-55 ◽  
Author(s):  
Douglas R Jeffery ◽  
John Absher ◽  
Frederick E Pfeiffer ◽  
Hagan Jackson
Keyword(s):  
Multiple Sclerosis ◽  
White Matter ◽  
Cognitive Deficits ◽  
High Frequency ◽  
White Matter Lesions ◽  
Subcortical White Matter ◽  
Emission Tomography ◽  
Axonal Loss ◽  
Cortical Function ◽  
Positron Emission

Patients suffering from multiple sclerosis have a high frequency of cognitive deficits usually attributable to demyelination and axonal loss in the subcortical white matter. Neurologic abnormalities referable to cortical function are uncommon but have been described. The present study describes three patients with clinically definite MS with deficits in cognitive function referable to cortical location. Two of the patients underwent positron emission tomography and showed profound cortical hypometabolism adjacent to subcortical white matter lesions seen on MRI. This paper points out that neurologic deficits referable to cortical sites may be caused by subcortical white matter lesions and that cognitive dysfunction in patients with MS may progress rapidly in the absence of motoria deficits or other evidence of clinical deterioration.

scholarly journals Transient Global Amnesia with Reversible White Matter Lesions: A Variant of Posterior Reversible Encephalopathy Syndrome?

2015 ◽  
Vol 2015 ◽  
pp. 1-3
Author(s):  
Tomoki Nakamizo ◽  
Ippei Tsuzuki ◽  
Takashi Koide
Keyword(s):  
White Matter ◽  
Posterior Reversible Encephalopathy Syndrome ◽  
Clinical Course ◽  
Vasogenic Edema ◽  
White Matter Lesions ◽  
Transient Global Amnesia ◽  
Subcortical White Matter ◽  
Posterior Reversible Encephalopathy ◽  
Global Amnesia ◽  
Reversible Encephalopathy

Transient global amnesia (TGA) is a self-limited disease characterized by isolated amnesia, which resolves within 24 h. In contrast, posterior reversible encephalopathy syndrome (PRES) is a potentially life-threatening disease that usually presents with seizures, altered mental status, headache, and visual disturbances. It is characterized by reversible vasogenic edema that predominantly involves the parieto-occipital subcortical white matter as shown by neuroimaging studies. To date, there have been no reported cases of PRES with a clinical course resembling TGA. Here we report the case of a 58-year-old woman who presented with isolated amnesia and headache. On admission, her blood pressure was 187/100 mmHg. She had complete anterograde amnesia and slight retrograde amnesia without other neurological findings. After the treatment of her hypertension, the amnesia resolved within 24 h. Although the initial magnetic resonance image (MRI) was almost normal, the fluid attenuation inversion recovery (FLAIR) images of the MRI on the next day revealed several small foci of high intensity areas in the fronto-parieto-occipital subcortical white matter, presumed to be vasogenic edema in PRES. The lesions disappeared one month later. This case suggests that PRES can mimic the clinical course of TGA. PRES should be considered in the differential diagnosis for TGA.

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