scholarly journals Reply: Apneic Oxygenation Has Not Been Disproven

2016 ◽  
Vol 193 (11) ◽  
pp. 1316-1317
Author(s):  
Matthew W. Semler ◽  
David R. Janz ◽  
Todd W. Rice
Keyword(s):  
Apneic Oxygenation

scholarly journals Blocking the double-lumen orifice versus apneic oxygenation during 1-lung ventilation

2017 ◽  
Vol 154 (6) ◽  
pp. 2122-2123
Author(s):  
Dae Myung Jung ◽  
Hyun Joo Ahn ◽  
Sin-Ho Jung ◽  
Mikyung Yang ◽  
Jie Ae Kim ◽  
...  
Keyword(s):  
Lung Ventilation ◽  
Double Lumen ◽  
Apneic Oxygenation

scholarly journals Apneic Oxygenation As a Quality Improvement Intervention in an Academic PICU

2019 ◽  
pp. 1
Author(s):  
Natalie Napolitano ◽  
Elizabeth K. Laverriere ◽  
Nancy Craig ◽  
Megan Snyder ◽  
Allison Thompson ◽  
...  
Keyword(s):  
Quality Improvement ◽  
Quality Improvement Intervention ◽  
Apneic Oxygenation ◽  
Improvement Intervention

Cerebrospinal fluid pressures during hypercapnia and hypoxia in dogs

1960 ◽  
Vol 198 (4) ◽  
pp. 704-708 ◽  
Author(s):  
Harold S. Small ◽  
Stanley W. Weitzner ◽  
Gabriel G. Nahas
Keyword(s):  
Cerebrospinal Fluid ◽  
Similar Pattern ◽  
Fluid Pressure ◽  
Probable Mechanism ◽  
Cerebrospinal Fluid Pressure ◽  
Apneic Oxygenation ◽  
Cisterna Magna ◽  
Controlled Ventilation ◽  
Cerebral Vasodilatation

Cerebrospinal fluid pressure (CSFP) in the cisterna magna, intra-aortic and intrathoracic venous pressures were measured during apneic oxygenation (diffusion respiration) in dogs under light pentobarbital-succinylcholine anesthesia. CSFP doubled in 2 minutes and reached a peak 375% above control in 10 minutes. During this time there were no consistent changes in arterial or venous pressures. Dogs ventilated with 5, 10 and 15% CO2 showed a similar pattern. Cerebral vasodilatation caused by CO2 is the probable mechanism in both groups. A sustained elevation of CSFP was observed throughout CO2 administration for as long as 90 minutes. In all cases there was a prompt return to control levels on termination of the hypercapnia. Controlled ventilation with 8% O2 caused an average 84% rise in CSFP, with a plateau occurring after 4–5 minutes. This was accompanied by marked increases in arterial and venous pressures. Cerebral vasodilatation probably occurs here also. During 90 seconds of asphyxia the sharpest increase (175%) in CSFP occurred. This was accompanied by marked increases in arterial and venous pressures, and represents the effect of hypoxia and hypercapnia combined.

EFFECTS OF A ???CO2 BUFFER??? ON HYPERCAPNIA OF APNEIC OXYGENATION

1961 ◽  
Vol 5 (3) ◽  
pp. 251
Author(s):  
G. G. NAHAS ◽  
E. C. JORDAN ◽  
J. C. LIGOU
Keyword(s):  
Apneic Oxygenation
Sign in / Sign up

Export Citation Format

Share Document