Middle Ear Ventilation in Secretory Otitis Media

1976 ◽  
Vol 85 (2_suppl) ◽  
pp. 178-181 ◽  
Author(s):  
Jörgen Holmquist ◽  
Ulf Renvall

The Eustachian tube function was determined repeatedly in 42 patients during as well as after the course of secretory otitis media. Air pressure equalization technique and impedance audiometry were used. Also the size of the mastoid air cell system was determined. It was found that poor tubal function and a small mastoid air cell system are significant findings in these ears.

1998 ◽  
Vol 77 (9) ◽  
pp. 744-747 ◽  
Author(s):  
Mirko Tos

Poor eustachian tube function plays a major role in the pathogenesis of chronic secretory otitis media. This is illustrated by epidemiologic studies of secretory otitis media and long-term studies of treated secretory otitis. Evaluation of hearing results demonstrate that effective preoperative and postoperative tubal function is highly important for good surgical outcome in cases of chronic otitis and cholesteatoma.


1984 ◽  
Vol 92 (3) ◽  
pp. 312-318 ◽  
Author(s):  
Bernt Falk ◽  
Bengt Magnuson

Previous research on eustachian tube function has been devoted mainly to the study of the tubal opening ability and pressure equalization. This article summarizes a series of experimental studies focusing on the closing ability of the tube. Results support the belief that the purpose of the tube should be seen primarily as protecting the middle ear from the extensive pressure variations that physiologically take place in the nasopharynx. A number of studies of diseased ears have shown that tubal malfunction was characterized mainly by a reduced ability to withstand negative pressure in the nasopharynx. Sniffing can evacuate the middle ear, causing high negative intratympanic pressure. It seems likely that this mechanism is involved in the development of middle ear effusion and manifest retraction-type middle ear disease.


1984 ◽  
Vol 77 (5) ◽  
pp. 1111-1117 ◽  
Author(s):  
Iwao Honjo ◽  
Koichi Ushiro ◽  
Tokichiro Mitoma ◽  
Kazuyuki Tashima

2005 ◽  
Vol 30 (5) ◽  
pp. 409-413 ◽  
Author(s):  
F. Salvinelli ◽  
M. Casale ◽  
F. Greco ◽  
L. D'Ascanio ◽  
T. Petitti ◽  
...  

1980 ◽  
Vol 89 (3_suppl) ◽  
pp. 65-66 ◽  
Author(s):  
Jörgen Holmquist ◽  
Ulf Renvall ◽  
Pål Svendsen

The medial displacement of the tympanic membrane or the retracted tympanic membrane is a common clinical finding. Also, retractions are looked upon as the precursor of middle ear cholesteatoma. In current otologic literature there seems to be some dispute whether the retractions of the tympanic membrane are caused by negative intratympanic air pressure or by shrinkage of middle ear adhesions pulling the tympanic membrane or part of it medially. In this study only ears with posterior/superior retractions of the tympanic membrane were investigated. Tympanometry revealed that only 33% of the ears did have middle ear pressure within ± 100 mm H2O. After insertion of a polyethylene tube through the tympanic membrane, the eustachian tube function was tested using the air pressure equalization technique. None of the ears could change the negative air pressure upon swallowing. Planometric measurements on the x-ray of the mastoid air cell system of the present material deviate from those of a normal material, indicating a significantly smaller air volume in the present material. It may be concluded that dysfunction of the eustachian tube and a small air volume of the mastoid are characteristic features of an ear with posterior/superior retraction of the tympanic membrane.


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