scholarly journals Cultural, Morphological and Pathogenic Variability of Exserohilum turcicum causing Turcicum Leaf Blight in Maize

Author(s):  
Gee ta ◽  
D.S. Aswathanarayana ◽  
M.K. Naik ◽  
Mallikarjun Kenganal ◽  
Prakash H. Kuchanur
2016 ◽  
Vol 8 (3) ◽  
pp. 1523-1529
Author(s):  
M. Ashraf Ahangar ◽  
Z. A. Bhat ◽  
F. A. Sheikh ◽  
Z. A. Dar ◽  
Ajaz A. Lone ◽  
...  

Turcicum leaf blight of maize incited by Exserohilum turcicum (Pass.) Leonard and Suggs is the major limiting factor of maize production in temperate agro-ecologies. Disease management through host plant resistance is the most effective strategy. In the present study among 26 maize genotypes which were initially screened for resistance against E. turcicum under field conditions, 8 genotypes viz., PS 39, CML 451, CML 470, CML 472, VL 1030, VL 1018140, VL1018527 and SMI178-1 were found resistant when screened against twelve isolates of E. turcicum under artificial epiphytotic conditions. Eight genotypes viz., PS45, CML165, CML459, VL1249, VL0536, SMC-5, SMC-3 and KDL 211 were found moderately resistant with disease grade ranged from 2.1-2.5. These maize genotypes possess resistance to turcicum leaf blight can be used successfully in developing high yielding early maturing varieties for high altitude temperate agro-ecologies. The fungus E. turcicum is highly variable in nature. Variability studies on pathogenicity were conducted on twelve isolates of E. turcicum on eleven putative differential maize lines. During the present study a wide pathogenic variation was observed among the twelve isolates of E. turcicum. Cluster analysis on the basis of similarity or dissimilarity in reaction types exhibited by the differential hosts, clustered the isolates into 6 pathogenic groups. The isolates belonged to higher altitudes (Kti 10, Kti11, Kti5) were found to be more aggressive as compared to the isolates of low altitude areas.


2019 ◽  
Vol 102 (2) ◽  
pp. 387-393
Author(s):  
Emine Burcu Turgay ◽  
Orhan Büyük ◽  
Berna Tunalı ◽  
Özlem Helvacıoğlu ◽  
Şener Kurt

HortScience ◽  
1990 ◽  
Vol 25 (9) ◽  
pp. 1067a-1067 ◽  
Author(s):  
A. C. Meyer ◽  
J. K Pataky ◽  
J.A. Juvik

Approximately 200 sweet corn inbred lines were screened for two years for resistance to northern leaf blight, caused by Exserohilum turcicum, and Stewart's wilt, caused by Erwinia stewartii. Inbreds with the best levels of partial resistance to races 1 and 2 of E. turcicum included IL11d, IL676a, IL677a, IL685d, IL766a, IL767a and IL797a. Inbreds with the best partial resistance to E. stewartii included IL126b, IL676a, IL767a, IL772a, IL774g, IL797a, IL798a and M6011. Several of these resistant and moderately resistant inbreds had common ancestors; however, inspection of pedigrees suggested that resistance was derived from Puerto Rican, Bolivian, and other tropical sources and/or dent corn. Thus, many of the sweet corn inbreds may carry different genes for resistance and can be used for the development of populations with improved resistance.


2018 ◽  
Vol 10 (1) ◽  
pp. 98-101
Author(s):  
T. A. Wani ◽  
G. N. Bhat ◽  
Mushtaq Ahmad ◽  
A. Anwar ◽  
Gul Zaffar

: A study was conducted during Kharif 2012 and 2013 at Sher-e-Kashmir University of Agricultural Sciences and Technology of Kashmir, Shalimar Campus Srinagar with the objective of screening sixty maize genotypes against Turcicum leaf blight caused by Exserohilum turcicum (Pass.) Leonard and Suggs. Field experiment was laid out in a randomized block design with three replications. In order to ensure establishment of infection, artificial inoculation of E. turcicum on test genotypes was made twice at 30 and 40 days after sowing with two different methods of inoculation (spraying on foliage of maize with spore suspension of pathogen @ 5x104 spores/ml at 30 DAS and by whorl drop inoculation of pathogen multiplies on sorghum grains at 40 DAS). Disease severity on test entries was scored at silk drying stage and studies revealed that two inbred lines, viz., NAI-112 and NAI-147, and one hybrid, viz., HQPM-1 were found resistant with pooled disease intensity of 4.12 per cent, 4.04 per cent and 4.38 per cent, respectively. Four inbred lines, viz., KDM 381 A, KDM 918 A, NAI-152 and NAI-167 were found susceptible with pooled disease intensity of 52.82 per cent, 51.02 per cent, 58.58 per cent and 61.33 per cent, respectively. The remaining genotypes were moderately resistant to moderately susceptible.


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