A framework to combine vector-valued metrics into a scalar-metric: Application to data comparison

2021 ◽  
pp. 1-10
Author(s):  
Gemma Piella
Filomat ◽  
2018 ◽  
Vol 32 (9) ◽  
pp. 3237-3243
Author(s):  
In Hwang ◽  
In Kim ◽  
Sumin Kim

In this note we give a connection between the closure of the range of block Hankel operators acting on the vector-valued Hardy space H2Cn and the left coprime factorization of its symbol. Given a subset F ? H2Cn, we also consider the smallest invariant subspace S*F of the backward shift S* that contains F.


2020 ◽  
pp. 1-13
Author(s):  
SEBASTIÁN PAVEZ-MOLINA

Abstract Let $(X,T)$ be a topological dynamical system. Given a continuous vector-valued function $F \in C(X, \mathbb {R}^{d})$ called a potential, we define its rotation set $R(F)$ as the set of integrals of F with respect to all T-invariant probability measures, which is a convex body of $\mathbb {R}^{d}$ . In this paper we study the geometry of rotation sets. We prove that if T is a non-uniquely ergodic topological dynamical system with a dense set of periodic measures, then the map $R(\cdot )$ is open with respect to the uniform topologies. As a consequence, we obtain that the rotation set of a generic potential is strictly convex and has $C^{1}$ boundary. Furthermore, we prove that the map $R(\cdot )$ is surjective, extending a result of Kucherenko and Wolf.


2019 ◽  
Vol 2019 (1) ◽  
Author(s):  
Feng Liu

Abstract In this note we study the rough singular integral $$ T_{\varOmega }f(x)=\mathrm{p.v.} \int _{\mathbb{R}^{n}}f(x-y)\frac{\varOmega (y/ \vert y \vert )}{ \vert y \vert ^{n}}\,dy, $$ T Ω f ( x ) = p . v . ∫ R n f ( x − y ) Ω ( y / | y | ) | y | n d y , where $n\geq 2$ n ≥ 2 and Ω is a function in $L\log L(\mathrm{S} ^{n-1})$ L log L ( S n − 1 ) with vanishing integral. We prove that $T_{\varOmega }$ T Ω is bounded on the mixed radial-angular spaces $L_{|x|}^{p}L_{\theta }^{\tilde{p}}( \mathbb{R}^{n})$ L | x | p L θ p ˜ ( R n ) , on the vector-valued mixed radial-angular spaces $L_{|x|}^{p}L_{\theta }^{\tilde{p}}(\mathbb{R}^{n},\ell ^{\tilde{p}})$ L | x | p L θ p ˜ ( R n , ℓ p ˜ ) and on the vector-valued function spaces $L^{p}(\mathbb{R}^{n}, \ell ^{\tilde{p}})$ L p ( R n , ℓ p ˜ ) if $1<\tilde{p}\leq p<\tilde{p}n/(n-1)$ 1 < p ˜ ≤ p < p ˜ n / ( n − 1 ) or $\tilde{p}n/(\tilde{p}+n-1)< p\leq \tilde{p}<\infty $ p ˜ n / ( p ˜ + n − 1 ) < p ≤ p ˜ < ∞ . The same conclusions hold for the well-known Riesz transforms and directional Hilbert transforms. It should be pointed out that our proof is based on the Calderón–Zygmund’s rotation method.


2021 ◽  
Vol 103 (4) ◽  
Author(s):  
M. G. Aartsen ◽  
R. Abbasi ◽  
M. Ackermann ◽  
J. Adams ◽  
J. A. Aguilar ◽  
...  

2021 ◽  
Vol 22 (12) ◽  
pp. 6642
Author(s):  
Nina Krako Jakovljevic ◽  
Kasja Pavlovic ◽  
Aleksandra Jotic ◽  
Katarina Lalic ◽  
Milica Stoiljkovic ◽  
...  

Type 2 diabetes (T2D), one of the most prevalent noncommunicable diseases, is often preceded by insulin resistance (IR), which underlies the inability of tissues to respond to insulin and leads to disturbed metabolic homeostasis. Mitochondria, as a central player in the cellular energy metabolism, are involved in the mechanisms of IR and T2D. Mitochondrial function is affected by insulin resistance in different tissues, among which skeletal muscle and liver have the highest impact on whole-body glucose homeostasis. This review focuses on human studies that assess mitochondrial function in liver, muscle and blood cells in the context of T2D. Furthermore, different interventions targeting mitochondria in IR and T2D are listed, with a selection of studies using respirometry as a measure of mitochondrial function, for better data comparison. Altogether, mitochondrial respiratory capacity appears to be a metabolic indicator since it decreases as the disease progresses but increases after lifestyle (exercise) and pharmacological interventions, together with the improvement in metabolic health. Finally, novel therapeutics developed to target mitochondria have potential for a more integrative therapeutic approach, treating both causative and secondary defects of diabetes.


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