Postinfectious glomerulonephritis: a cause of pulmonary renal syndrome

Author(s):  
Randa I. Farah ◽  
Nadia Alqurini ◽  
Ayman Wahbeh
2012 ◽  
Vol 19 (4) ◽  
pp. e136-e138 ◽  
Author(s):  
Ankur Kalra ◽  
Naoto Yokogawa ◽  
Haroon Raja ◽  
Chandrasekar Palaniswamy ◽  
Priyank Desai ◽  
...  

2017 ◽  
Vol 66 (6) ◽  
pp. 802-805 ◽  
Author(s):  
Di Wang ◽  
Li Li ◽  
Lei Wei ◽  
Yingying Liu ◽  
Shiren Sun

2015 ◽  
Vol 31 (3) ◽  
pp. 353-356 ◽  
Author(s):  
Emily Elizabeth Bowen ◽  
Robert Hangartner ◽  
Iain Macdougall

1978 ◽  
Vol 130 (6) ◽  
pp. 1141-1148 ◽  
Author(s):  
PG Herman ◽  
JP Balikian ◽  
SE Seltzer ◽  
M Ehrie

1988 ◽  
Vol 8 (5) ◽  
pp. 431-432 ◽  
Author(s):  
Angela Volpi ◽  
Mietta Meroni ◽  
Graziana Battini ◽  
Carola Fabbri ◽  
Gianmichele Ferrario ◽  
...  

2018 ◽  
Vol 90 (6) ◽  
pp. 130-136 ◽  
Author(s):  
M L Bulanova ◽  
D V Potapov ◽  
N M Bulanov ◽  
L V Lysenko(Kozlovskaya)

Goodpasture’s disease (anti-GBM disease) is a rare small vessels vasculitis characterized by the presence of autoantibodies directed against the glomerular basement membrane (GBM) and alveolar basement membrane. Common feature of anti-GBM disease is a combination of rapidly progressive glomerulonephritis and alveolar hemorrhage (pulmonary-renal syndrome). We present a case of atypical disease course in a young male patient who developed alveolar hemorrhage without renal failure. The only symptom of renal involvement was isolated hematuria. Plasmapheresis combined with immunosuppression (cyclophosphamide and corticosteroids) was effective. We present a review of state-of-art data on the pathogenesis and disease course of anti-GBM disease.


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