Physiological implications of arteriovenous anastomoses and venous hemodynamic dysfunction in early gestational uterine circulation: a review

2013 ◽  
Vol 26 (9) ◽  
pp. 841-846 ◽  
Author(s):  
Wilfried Gyselaers ◽  
Louis Peeters
1990 ◽  
Vol 5 (2) ◽  
pp. 77-83 ◽  
Author(s):  
H. J. Scott

The pathogenesis of the development of varicose veins remains obscure, and there have been many theories suggesting the primary cause. Recognized predisposing factors include age, sex, race, weight, height, pregnancy, diet, erect stance, clothes, alcohol, heredity, bowel habit and occupation. However the primary causative factors have centred around vein wall weaknesses, valvular deficiencies, and haemodynamic effects. This review looks at the evidence for and against arteriovenous anastomoses being the initiating factor in the development of varicose veins.


1976 ◽  
Vol 96 (2) ◽  
pp. 285-300 ◽  
Author(s):  
S.F. Amakiri

Physiology ◽  
2009 ◽  
Vol 24 (1) ◽  
pp. 58-71 ◽  
Author(s):  
George Osol ◽  
Maurizio Mandala

Sufficient uteroplacental blood flow is essential for normal pregnancy outcome and is accomplished by the coordinated growth and remodeling of the entire uterine circulation, as well as the creation of a new fetal vascular organ: the placenta. The process of remodeling involves a number of cellular processes, including hyperplasia and hypertrophy, rearrangement of existing elements, and changes in extracellular matrix. In this review, we provide information on uterine blood flow increases during pregnancy, the influence of placentation type on the distribution of uterine vascular resistance, consideration of the patterns, nature, and extent of maternal uterine vascular remodeling during pregnancy, and what is known about the underlying cellular mechanisms.


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